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Hippocampal mutant APP and amyloid beta-induced cognitive decline, dendritic spine loss, defective autophagy, mitophagy and mitochondrial abnormalities in a mouse model of Alzheimer's disease

Human molecular genetics, 2018-04, Vol.27 (8), p.1332-1342

2018

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Hippocampal phosphorylated tau induced cognitive decline, dendritic spine loss and mitochondrial abnormalities in a mouse model of Alzheimer's disease

Human molecular genetics, 2018-01, Vol.27 (1), p.30-40

2018

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Mitochondrial division inhibitor 1 reduces dynamin-related protein 1 and mitochondrial fission activity

Human molecular genetics, 2019-01, Vol.28 (2), p.177-199

2019

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Mutant APP and amyloid beta-induced defective autophagy, mitophagy, mitochondrial structural and functional changes and synaptic damage in hippocampal neurons from Alzheimer's disease

Human molecular genetics, 2018-07, Vol.27 (14), p.2502-2516

2018

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Reduced dynamin-related protein 1 protects against phosphorylated Tau-induced mitochondrial dysfunction and synaptic damage in Alzheimer's disease

Human molecular genetics, 2016-11, Vol.25 (22), p.4881-4897

2016

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Abnormal mitochondrial dynamics and synaptic degeneration as early events in Alzheimer's disease: Implications to mitochondria-targeted antioxidant therapeutics

Biochimica et biophysica acta, 2012-05, Vol.1822 (5), p.639-649

2012

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Reduced VDAC1 protects against Alzheimer's disease, mitochondria, and synaptic deficiencies

Journal of Alzheimer's disease, 2013-01, Vol.37 (4), p.679

2013

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Dynamin-related protein 1 and mitochondrial fragmentation in neurodegenerative diseases

Brain Research Reviews, 2011-06, Vol.67 (1), p.103-118

2011

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Differential loss of synaptic proteins in Alzheimer's disease: implications for synaptic dysfunction

Journal of Alzheimer's disease, 2005-04, Vol.7 (2), p.103

2005

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Gene expression profiles of transcripts in amyloid precursor protein transgenic mice: up-regulation of mitochondrial metabolism and apoptotic genes is an early cellular change in Alzheimer's disease

Human molecular genetics, 2004-06, Vol.13 (12), p.1225-1240

2004

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Mitochondria-Targeted Antioxidant SS31 Prevents Amyloid Beta-Induced Mitochondrial Abnormalities and Synaptic Degeneration in Alzheimer's Disease

Pharmaceuticals, 2012-10, Vol.5 (10), p.1103-1119

2012

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Mitochondrial structural and functional dynamics in Huntington's disease

Brain Research Reviews, 2009-06, Vol.61 (1), p.33-48

2009

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Neutralization of granulocyte macrophage colony-stimulating factor decreases amyloid beta 1-42 and suppresses microglial activity in a transgenic mouse model of Alzheimer's disease

Human molecular genetics, 2009-10, Vol.18 (20), p.3876-3893

2009

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Maternal exercise upregulates mitochondrial gene expression and increases enzyme activity of fetal mouse hearts

Physiological reports, 2017-03, Vol.5 (5), p.np-n/a

2017

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Mitochondria-targeted molecules MitoQ and SS31 reduce mutant huntingtin-induced mitochondrial toxicity and synaptic damage in Huntington's disease

Human molecular genetics, 2016-05, Vol.25 (9), p.1739-1753

2016

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Abnormal interaction of VDAC1 with amyloid beta and phosphorylated tau causes mitochondrial dysfunction in Alzheimer's disease

Human molecular genetics, 2012-12, Vol.21 (23), p.5131-5146

2012

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Abnormal interaction between the mitochondrial fission protein Drp1 and hyperphosphorylated tau in Alzheimer's disease neurons: implications for mitochondrial dysfunction and neuronal damage

Human molecular genetics, 2012-06, Vol.21 (11), p.2538-2547

2012

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Mitochondria-Division Inhibitor 1 Protects Against Amyloid-β induced Mitochondrial Fragmentation and Synaptic Damage in Alzheimer's Disease

Journal of Alzheimer's disease, 2017-01, Vol.58 (1), p.147

2017

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Granulocyte-macrophage colony-stimulating factor antibody suppresses microglial activity: implications for anti-inflammatory effects in Alzheimer's Disease and multiple sclerosis

Journal of neurochemistry, 2009-12, Vol.111 (6), p.1514-1528

2009

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Impaired mitochondrial dynamics and abnormal interaction of amyloid beta with mitochondrial protein Drp1 in neurons from patients with Alzheimer's disease: implications for neuronal damage

Human molecular genetics, 2011-07, Vol.20 (13), p.2495-2509

2011

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Protective effects of reduced dynamin-related protein 1 against amyloid beta-induced mitochondrial dysfunction and synaptic damage in Alzheimer's disease

Human molecular genetics, 2016-12, Vol.25 (23), p.5148-5166

2016

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Impaired mitochondrial biogenesis, defective axonal transport of mitochondria, abnormal mitochondrial dynamics and synaptic degeneration in a mouse model of Alzheimer's disease

Human molecular genetics, 2011-12, Vol.20 (23), p.4515-4529

2011

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A partial reduction of Drp1 improves cognitive behavior and enhances mitophagy, autophagy and dendritic spines in a transgenic Tau mouse model of Alzheimer disease

Human molecular genetics, 2022-06, Vol.31 (11), p.1788-1805

2022

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Mitochondria-targeted small molecule SS31: a potential candidate for the treatment of Alzheimer's disease

Human molecular genetics, 2017-04, Vol.26 (8), p.1483-1496

2017

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Protective effects of a natural product, curcumin, against amyloid β induced mitochondrial and synaptic toxicities in Alzheimer's disease

Journal of investigative medicine, 2016-12, Vol.64 (8), p.1220-1234

2016

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Hippocampal mutant APP and amyloid beta-induced cognitive decline, dendritic spine loss, defective autophagy, mitophagy and mitochondrial abnormalities in a mouse model of Alzheimer's disease

Hippocampal phosphorylated tau induced cognitive decline, dendritic spine loss and mitochondrial abnormalities in a mouse model of Alzheimer's disease

Mitochondrial division inhibitor 1 reduces dynamin-related protein 1 and mitochondrial fission activity

Mutant APP and amyloid beta-induced defective autophagy, mitophagy, mitochondrial structural and functional changes and synaptic damage in hippocampal neurons from Alzheimer's disease

Reduced dynamin-related protein 1 protects against phosphorylated Tau-induced mitochondrial dysfunction and synaptic damage in Alzheimer's disease

Abnormal mitochondrial dynamics and synaptic degeneration as early events in Alzheimer's disease: Implications to mitochondria-targeted antioxidant therapeutics

Reduced VDAC1 protects against Alzheimer's disease, mitochondria, and synaptic deficiencies

Dynamin-related protein 1 and mitochondrial fragmentation in neurodegenerative diseases

Differential loss of synaptic proteins in Alzheimer's disease: implications for synaptic dysfunction

Gene expression profiles of transcripts in amyloid precursor protein transgenic mice: up-regulation of mitochondrial metabolism and apoptotic genes is an early cellular change in Alzheimer's disease

Mitochondria-Targeted Antioxidant SS31 Prevents Amyloid Beta-Induced Mitochondrial Abnormalities and Synaptic Degeneration in Alzheimer's Disease

Mitochondrial structural and functional dynamics in Huntington's disease

Neutralization of granulocyte macrophage colony-stimulating factor decreases amyloid beta 1-42 and suppresses microglial activity in a transgenic mouse model of Alzheimer's disease

Maternal exercise upregulates mitochondrial gene expression and increases enzyme activity of fetal mouse hearts

Mitochondria-targeted molecules MitoQ and SS31 reduce mutant huntingtin-induced mitochondrial toxicity and synaptic damage in Huntington's disease

Abnormal interaction of VDAC1 with amyloid beta and phosphorylated tau causes mitochondrial dysfunction in Alzheimer's disease

Abnormal interaction between the mitochondrial fission protein Drp1 and hyperphosphorylated tau in Alzheimer's disease neurons: implications for mitochondrial dysfunction and neuronal damage

Mitochondria-Division Inhibitor 1 Protects Against Amyloid-β induced Mitochondrial Fragmentation and Synaptic Damage in Alzheimer's Disease

Granulocyte-macrophage colony-stimulating factor antibody suppresses microglial activity: implications for anti-inflammatory effects in Alzheimer's Disease and multiple sclerosis

Impaired mitochondrial dynamics and abnormal interaction of amyloid beta with mitochondrial protein Drp1 in neurons from patients with Alzheimer's disease: implications for neuronal damage

Protective effects of reduced dynamin-related protein 1 against amyloid beta-induced mitochondrial dysfunction and synaptic damage in Alzheimer's disease

Impaired mitochondrial biogenesis, defective axonal transport of mitochondria, abnormal mitochondrial dynamics and synaptic degeneration in a mouse model of Alzheimer's disease

A partial reduction of Drp1 improves cognitive behavior and enhances mitophagy, autophagy and dendritic spines in a transgenic Tau mouse model of Alzheimer disease

Mitochondria-targeted small molecule SS31: a potential candidate for the treatment of Alzheimer's disease

Protective effects of a natural product, curcumin, against amyloid β induced mitochondrial and synaptic toxicities in Alzheimer's disease

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