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Three Epstein-Barr virus latency proteins independently promote genomic instability by inducing DNA damage, inhibiting DNA repair and inactivating cell cycle checkpoints

Oncogene, 2009-11, Vol.28 (45), p.3997-4008

2009

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The Epstein-Barr virus nuclear antigen-1 promotes telomere dysfunction via induction of oxidative stress

Leukemia, 2011-06, Vol.25 (6), p.1017-1025

2011

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Oxidative stress enables Epstein-Barr virus-induced B-cell transformation by posttranscriptional regulation of viral and cellular growth-promoting factors

Oncogene, 2016-07, Vol.35 (29), p.3807-3816

2016

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Telomere dysfunction and activation of alternative lengthening of telomeres in B-lymphocytes infected by Epstein-Barr virus

Oncogene, 2013-12, Vol.32 (49), p.5522-5530

2013

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Epstein-Barr virus promotes genomic instability in Burkitt's lymphoma

Oncogene, 2007-08, Vol.26 (35), p.5115-5123

2007

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A transgenic mouse model of the ubiquitin/proteasome system

Nature biotechnology, 2003-08, Vol.21 (8), p.897-902

2003

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Epstein-Barr virus (EBV) load in bone marrow transplant recipients at risk to develop posttransplant lymphoproliferative disease: prophylactic infusion of EBV-specific cytotoxic T cells

Blood, 2000-02, Vol.95 (3), p.807-814

2000

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Inhibition of antigen processing by the internal repeat region of the Epstein-Barr virus nuclear antigen-1

Nature (London), 1995-06, Vol.375 (6533), p.685-688

1995

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The ubiquitin-specific protease USP25 interacts with three sarcomeric proteins

Cellular and molecular life sciences : CMLS, 2006-03, Vol.63 (6), p.723-734

2006

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Short-lived green fluorescent proteins for quantifying ubiquitin/proteasome-dependent proteolysis in living cells

Nature biotechnology, 2000-05, Vol.18 (5), p.538-543

2000

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Differential Regulation of MHC Class‐I‐Restricted and Unrestricted Cytotoxicity by the Us3 Protein Kinase of Herpes Simplex Virus‐1

Scandinavian journal of immunology, 2004-12, Vol.60 (6), p.592-599

2004

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Activity-Based Ubiquitin-Specific Protease (USP) Profiling of Virus-Infected and Malignant Human Cells

Proceedings of the National Academy of Sciences - PNAS, 2004-02, Vol.101 (8), p.2253-2258

2004

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Inhibition of Ubiquitin/Proteasome-Dependent Protein Degradation by the Gly-Ala Repeat Domain of the Epstein-Barr Virus Nuclear Antigen 1

Proceedings of the National Academy of Sciences - PNAS, 1997-11, Vol.94 (23), p.12616-12621

1997

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The UBA2 Domain Functions as an Intrinsic Stabilization Signal that Protects Rad23 from Proteasomal Degradation

Molecular cell, 2005-04, Vol.18 (2), p.225-235

2005

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Patterns of Epstein-Barr Virus Infection in Non-Neoplastic Lymphoid Tissue

Blood, 1992-05, Vol.79 (10), p.2520-2526

1992

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Mutant Ubiquitin Found in Neurodegenerative Disorders Is a Ubiquitin Fusion Degradation Substrate That Blocks Proteasomal Degradation

The Journal of cell biology, 2002-04, Vol.157 (3), p.417-427

2002

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HLA-A11 Epitope Loss Isolates of Epstein-Barr Virus from a Highly A11$^+$ Population

Science (American Association for the Advancement of Science), 1993-04, Vol.260 (5104), p.98-100

1993

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Local Suppression of Epstein-Barr Virus (EBV)-Specific Cytotoxicity in Biopsies of EBV-Positive Hodgkin's Disease

Blood, 1995-08, Vol.86 (4), p.1493-1501

1995

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Epstein-Barr virus oncogenesis and the ubiquitin-proteasome system

Oncogene, 2004-03, Vol.23 (11), p.2107-2115

Masucci, Maria G

2004

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The epstein‐barr virus latent membrane protein‐1 (LMP1) induces interleukin‐10 production in burkitt lymphoma lines

International journal of cancer, 1994-04, Vol.57 (2), p.240-244

1994

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Multiple HLA A11-restricted cytotoxic T-lymphocyte epitopes of different immunogenicities in the Epstein-Barr virus-encoded nuclear antigen 4

Journal of Virology, 1993-03, Vol.67 (3), p.1572-1578

1993

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Avoiding proteasomal processing: the case of EBNA1

Current topics in microbiology and immunology, 2002, Vol.269, p.23

2002

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Pharmacological Disintegration of Lipid Rafts Decreases Specific Tetramer Binding and Disrupts the CD3 Complex and CD8 Heterodimer in Human Cytotoxic T Lymphocytes

Scandinavian journal of immunology, 2003-02, Vol.57 (2), p.99-106

2003

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Recognition of the Epstein-Barr Virus-Encoded Nuclear Antigens EBNA-4 and EBNA-6 by HLA-A11-Restricted Cytotoxic T Lymphocytes: Implications for Down-Regulation of HLA-A11 in Burkitt Lymphoma

Proceedings of the National Academy of Sciences - PNAS, 1992-07, Vol.89 (13), p.5862-5866

1992

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5-Azacytidine up regulates the expression of Epstein-Barr virus nuclear antigen 2 (EBNA-2) through EBNA-6 and latent membrane protein in the Burkitt's lymphoma line rael

Journal of Virology, 1989-07, Vol.63 (7), p.3135-3141

1989

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Three Epstein-Barr virus latency proteins independently promote genomic instability by inducing DNA damage, inhibiting DNA repair and inactivating cell cycle checkpoints

The Epstein-Barr virus nuclear antigen-1 promotes telomere dysfunction via induction of oxidative stress

Oxidative stress enables Epstein-Barr virus-induced B-cell transformation by posttranscriptional regulation of viral and cellular growth-promoting factors

Telomere dysfunction and activation of alternative lengthening of telomeres in B-lymphocytes infected by Epstein-Barr virus

Epstein-Barr virus promotes genomic instability in Burkitt's lymphoma

A transgenic mouse model of the ubiquitin/proteasome system

Epstein-Barr virus (EBV) load in bone marrow transplant recipients at risk to develop posttransplant lymphoproliferative disease: prophylactic infusion of EBV-specific cytotoxic T cells

Inhibition of antigen processing by the internal repeat region of the Epstein-Barr virus nuclear antigen-1

The ubiquitin-specific protease USP25 interacts with three sarcomeric proteins

Short-lived green fluorescent proteins for quantifying ubiquitin/proteasome-dependent proteolysis in living cells

Differential Regulation of MHC Class‐I‐Restricted and Unrestricted Cytotoxicity by the Us3 Protein Kinase of Herpes Simplex Virus‐1

Activity-Based Ubiquitin-Specific Protease (USP) Profiling of Virus-Infected and Malignant Human Cells

Inhibition of Ubiquitin/Proteasome-Dependent Protein Degradation by the Gly-Ala Repeat Domain of the Epstein-Barr Virus Nuclear Antigen 1

The UBA2 Domain Functions as an Intrinsic Stabilization Signal that Protects Rad23 from Proteasomal Degradation

Patterns of Epstein-Barr Virus Infection in Non-Neoplastic Lymphoid Tissue

Mutant Ubiquitin Found in Neurodegenerative Disorders Is a Ubiquitin Fusion Degradation Substrate That Blocks Proteasomal Degradation

HLA-A11 Epitope Loss Isolates of Epstein-Barr Virus from a Highly A11$^+$ Population

Local Suppression of Epstein-Barr Virus (EBV)-Specific Cytotoxicity in Biopsies of EBV-Positive Hodgkin's Disease

Epstein-Barr virus oncogenesis and the ubiquitin-proteasome system

The epstein‐barr virus latent membrane protein‐1 (LMP1) induces interleukin‐10 production in burkitt lymphoma lines

Multiple HLA A11-restricted cytotoxic T-lymphocyte epitopes of different immunogenicities in the Epstein-Barr virus-encoded nuclear antigen 4

Avoiding proteasomal processing: the case of EBNA1

Pharmacological Disintegration of Lipid Rafts Decreases Specific Tetramer Binding and Disrupts the CD3 Complex and CD8 Heterodimer in Human Cytotoxic T Lymphocytes

Recognition of the Epstein-Barr Virus-Encoded Nuclear Antigens EBNA-4 and EBNA-6 by HLA-A11-Restricted Cytotoxic T Lymphocytes: Implications for Down-Regulation of HLA-A11 in Burkitt Lymphoma

5-Azacytidine up regulates the expression of Epstein-Barr virus nuclear antigen 2 (EBNA-2) through EBNA-6 and latent membrane protein in the Burkitt's lymphoma line rael

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