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S100a9 deficiency accelerates MDS-associated tumor escape via PD-1/PD-L1 overexpression

Acta biochimica et biophysica Sinica, 2023-02, Vol.55 (2), p.194-201

2023

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Integrated traditional Chinese and conventional medicine in the treatment of anemia due to lower-risk myelodysplastic syndrome: study protocol for a randomized placebo-controlled trial

Trials, 2021-10, Vol.22 (1), p.712-712, Article 712

2021

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Decitabine of reduced dosage in Chinese patients with myelodysplastic syndrome: a retrospective analysis

PloS one, 2014-04, Vol.9 (4), p.e95473

2014

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Combination Therapy of Eltrombopag and Cyclosporine a Improves Hematopoiesis in Patients with Lower-Risk Myelodysplastic Syndrome

Blood, 2022-11, Vol.140 (Supplement 1), p.4084-4085

2022

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The use of selective immunosuppressive therapy on myelodysplastic syndromes in targeted populations results in good response rates and avoids treatment-related disease progression

American journal of hematology, 2012-01, Vol.87 (1), p.26-31

2012

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Diagnosis of unknown nonhematological tumors by bone marrow biopsy: a retrospective analysis of 10,112 samples

Journal of cancer research and clinical oncology, 2009-05, Vol.135 (5), p.687-693

2009

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Eltrombopag restores proliferative capacity and adipose-osteogenic balance of mesenchymal stromal cells in low-risk myelodysplastic syndromes

European journal of pharmacology, 2024-12, Vol.985, p.177086, Article 177086

2024

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Experimental and clinical characteristics in myelodysplastic syndrome patients with or without HLA-DR15 allele

Hematological oncology, 2010-06, Vol.28 (2), p.98-103

2010

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NPM1 mutation with DNMT3A wild type defines a subgroup of MDS with particularly favourable outcomes after decitabine therapy

British journal of haematology, 2020-06, Vol.189 (5), p.982-984

2020

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Bone Marrow Fibrosis at Diagnosis and during the Course of Disease Is Associated with TP53 Mutations and Adverse Prognosis in Primary Myelodysplastic Syndrome

Cancers, 2022-06, Vol.14 (12), p.2984

2022

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S100a9 deficiency accelerates MDS-associated tumor escape via PD-1/PD-L1 overexpression: S100a9 deficiency accelerates tumor escape

Acta biochimica et biophysica Sinica, 2023-02, Vol.55 (2), p.194-201

2023

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The clinical characteristics, gene mutations and outcomes of myelodysplastic syndromes with diabetes mellitus

Journal of cancer research and clinical oncology, 2024-02, Vol.150 (2), p.71-71, Article 71

2024

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Energy efficient design for multiuser downlink energy and uplink information transfer in 5G

Science China. Information sciences, 2016-02, Vol.59 (2), p.70-77

2016

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DNMT3Awt NPM1-Mutation Defines a Subgroup of MDS with Special Favorable Outcomes Towards Decitabine Therapy

Blood, 2019-11, Vol.134 (Supplement_1), p.1724-1724

2019

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Genetic landscape of recurrent ASXL1, U2AF1, SF3B1, SRSF2, and EZH2 mutations in 304 Chinese patients with myelodysplastic syndromes

Tumor biology, 2016-04, Vol.37 (4), p.4633-4640

2016

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Interleukin-17-producing CD4+ T lymphocytes are increased in patients with primary immune thrombocytopenia

Blood coagulation & fibrinolysis, 2016-04, Vol.27 (3), p.301-307

2016

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The efficacy and toxicity of the CHG priming regimen (low-dose cytarabine, homoharringtonine, and G-CSF) in higher risk MDS patients relapsed or refractory to decitabine

Journal of cancer research and clinical oncology, 2019-12, Vol.145 (12), p.3089-3097

2019

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High expression of the human equilibrative nucleoside transporter 1 gene predicts a good response to decitabine in patients with myelodysplastic syndrome

Journal of translational medicine, 2016-03, Vol.14 (65), p.66-66, Article 66

2016

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Efficacy and toxicity of decitabine versus CHG regimen (low-dose cytarabine, homoharringtonine and granulocyte colony-stimulating factor) in patients with higher risk myelodysplastic syndrome: a retrospective study

Leukemia & lymphoma, 2016-06, Vol.57 (6), p.1367-1374

2016

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Efficacy and Toxicity Of Decitabine Versus CHG Priming Regimen In Patients With Higher Risk Myelodysplastic Syndrome

Blood, 2013-11, Vol.122 (21), p.2789-2789

2013

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Cytogenetic response based on revised IPSS cytogenetic risk stratification and minimal residual disease monitoring by FISH in MDS patients treated with low-dose decitabine

Leukemia research, 2013-11, Vol.37 (11), p.1516-1521

2013

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A 15 Mg/M2/d Dose of Decitabine Confers Comparable Responses and Better Tolerance Than the Standard Regimen in MDS patients—results of a Multicenter Prospective Cohort Study

Blood, 2012-11, Vol.120 (21), p.3832-3832

2012

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MRI for assessment of iron overload in patients with hematologic disease

Zhōnghuá xuèyèxué zázhì, 2015-12, Vol.36 (12), p.1039

2015

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Effect of low-dose cytarabine, homoharringtonine and granulocyte colony-stimulating factor priming regimen on patients with advanced myelodysplastic syndrome or acute myeloid leukemia transformed from myelodysplastic syndrome

Leukemia & lymphoma, 2009-01, Vol.50 (9), p.1461-1467

2009

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The Incidence of Corrected Hypercalcemia and Its Relationship with Prognosis for Chinese Multiply Myeloma

Blood, 2009-11, Vol.114 (22), p.4903-4903

2009

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S100a9 deficiency accelerates MDS-associated tumor escape via PD-1/PD-L1 overexpression

Integrated traditional Chinese and conventional medicine in the treatment of anemia due to lower-risk myelodysplastic syndrome: study protocol for a randomized placebo-controlled trial

Decitabine of reduced dosage in Chinese patients with myelodysplastic syndrome: a retrospective analysis

Combination Therapy of Eltrombopag and Cyclosporine a Improves Hematopoiesis in Patients with Lower-Risk Myelodysplastic Syndrome

The use of selective immunosuppressive therapy on myelodysplastic syndromes in targeted populations results in good response rates and avoids treatment-related disease progression

Diagnosis of unknown nonhematological tumors by bone marrow biopsy: a retrospective analysis of 10,112 samples

Eltrombopag restores proliferative capacity and adipose-osteogenic balance of mesenchymal stromal cells in low-risk myelodysplastic syndromes

Experimental and clinical characteristics in myelodysplastic syndrome patients with or without HLA-DR15 allele

NPM1 mutation with DNMT3A wild type defines a subgroup of MDS with particularly favourable outcomes after decitabine therapy

Bone Marrow Fibrosis at Diagnosis and during the Course of Disease Is Associated with TP53 Mutations and Adverse Prognosis in Primary Myelodysplastic Syndrome

S100a9 deficiency accelerates MDS-associated tumor escape via PD-1/PD-L1 overexpression: S100a9 deficiency accelerates tumor escape

The clinical characteristics, gene mutations and outcomes of myelodysplastic syndromes with diabetes mellitus

Energy efficient design for multiuser downlink energy and uplink information transfer in 5G

DNMT3Awt NPM1-Mutation Defines a Subgroup of MDS with Special Favorable Outcomes Towards Decitabine Therapy

Genetic landscape of recurrent ASXL1, U2AF1, SF3B1, SRSF2, and EZH2 mutations in 304 Chinese patients with myelodysplastic syndromes

Interleukin-17-producing CD4+ T lymphocytes are increased in patients with primary immune thrombocytopenia

The efficacy and toxicity of the CHG priming regimen (low-dose cytarabine, homoharringtonine, and G-CSF) in higher risk MDS patients relapsed or refractory to decitabine

High expression of the human equilibrative nucleoside transporter 1 gene predicts a good response to decitabine in patients with myelodysplastic syndrome

Efficacy and toxicity of decitabine versus CHG regimen (low-dose cytarabine, homoharringtonine and granulocyte colony-stimulating factor) in patients with higher risk myelodysplastic syndrome: a retrospective study

Efficacy and Toxicity Of Decitabine Versus CHG Priming Regimen In Patients With Higher Risk Myelodysplastic Syndrome

Cytogenetic response based on revised IPSS cytogenetic risk stratification and minimal residual disease monitoring by FISH in MDS patients treated with low-dose decitabine

A 15 Mg/M2/d Dose of Decitabine Confers Comparable Responses and Better Tolerance Than the Standard Regimen in MDS patients—results of a Multicenter Prospective Cohort Study

MRI for assessment of iron overload in patients with hematologic disease

Effect of low-dose cytarabine, homoharringtonine and granulocyte colony-stimulating factor priming regimen on patients with advanced myelodysplastic syndrome or acute myeloid leukemia transformed from myelodysplastic syndrome

The Incidence of Corrected Hypercalcemia and Its Relationship with Prognosis for Chinese Multiply Myeloma

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