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A remarkable adaptive paradigm of heart performance and protection emerges in response to the constitutive challenge of marked cardiac-specific overexpression of adenylyl cyclase type 8

Biophysical journal, 2023-02, Vol.122 (3), p.537a-537a

2023

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Ca(2+)/calmodulin-activated phosphodiesterase 1A is highly expressed in rabbit cardiac sinoatrial nodal cells and regulates pacemaker function

Journal of molecular and cellular cardiology, 2016-09, Vol.98, p.73

2016

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Ca²⁺-dependent phosphorylation of Ca²⁺ cycling proteins generates robust rhythmic local Ca²⁺ releases in cardiac pacemaker cells

Science signaling, 2013-01, Vol.6 (260), p.ra6

2013

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Basal Spontaneous Firing of Rabbit Sinoatrial Node Cells Is Regulated by Dual Activation of PDEs (Phosphodiesterases) 3 and 4

Circulation. Arrhythmia and electrophysiology, 2018-06, Vol.11 (6), p.e005896-e005896

2018

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Overexpression of a Neuronal Type Adenylyl Cyclase (Type 8) in Sinoatrial Node Markedly Impacts Heart Rate and Rhythm

Frontiers in neuroscience, 2019-06, Vol.13, p.615-615

2019

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Ca2+-stimulated Basal Adenylyl Cyclase Activity Localization in Membrane Lipid Microdomains of Cardiac Sinoatrial Nodal Pacemaker Cells

The Journal of biological chemistry, 2008-05, Vol.283 (21), p.14461-14468

2008

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Phosphoprotein Phosphatase 1 but Not 2A Activity Modulates Coupled-Clock Mechanisms to Impact on Intrinsic Automaticity of Sinoatrial Nodal Pacemaker Cells

Cells (Basel, Switzerland), 2021-11, Vol.10 (11), p.3106

2021

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Changes in cAMP signaling are associated with age-related downregulation of spontaneously beating atrial tissue energetic indices

GeroScience, 2023-02, Vol.45 (1), p.209-219

2023

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Ca(2+) -stimulated basal adenylyl cyclase activity localization in membrane lipid microdomains of cardiac sinoatrial nodal pacemaker cells

The Journal of biological chemistry, 2008-05, Vol.283 (21), p.14461-14468

2008

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cAMP signaling affects age-associated deterioration of pacemaker beating interval dynamics

GeroScience, 2023-08, Vol.45 (4), p.2589-2600

2023

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Sarcoplasmic reticulum Ca2+ cycling protein phosphorylation in a physiologic Ca2+ milieu unleashes a high-power, rhythmic Ca2+ clock in ventricular myocytes: Relevance to arrhythmias and bio-pacemaker design

Journal of molecular and cellular cardiology, 2014-01, Vol.66, p.106-115

2014

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Ca2+/calmodulin-activated phosphodiesterase 1A is highly expressed in rabbit cardiac sinoatrial nodal cells and regulates pacemaker function

Journal of molecular and cellular cardiology, 2016-09, Vol.98, p.73-82

2016

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A bioluminescence method for direct measurement of phosphodiesterase activity

Analytical biochemistry, 2011-10, Vol.417 (1), p.36-40

2011

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cAMP/PKA signaling affects aged-deteriorated pacemaker beat interval dynamic: Antiaging approach

Journal of molecular and cellular cardiology, 2022-12, Vol.173, p.32-32

2022

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25-Hydroxycholesterol Is Produced by Testicular Macrophages During the Early Postnatal Period and Influences Differentiation of Leydig Cells In Vitro

Biology of reproduction, 2002-05, Vol.66 (5), p.1336-1341

2002

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Transcriptome and Proteome Alterations of Mice that Overexpress Adenylate Cyclase Type 8 Underlie a Chronic and Marked Increase in Sinoatrial Node (SAN) and Left Ventricle (LV) Performance While Ensuring Heart Survival

Biophysical journal, 2019-02, Vol.116 (3), p.131a-131a

2019

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Metabolic Enzyme Acetylation is Elicited in Response to Stress Induced by Cardiac Specific Overexpression of Human ADCY8

The FASEB journal, 2020-04, Vol.34 (S1), p.1-1

2020

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Cardiac Specific Adenyl Cyclase Type 8 Overexpression Induces Chronic Cardiac Exercise and Leads to a Unique Form of Cardiac Growth.

The FASEB journal, 2020-04, Vol.34 (S1), p.1-1

2020

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Testosterone Regulates 25-Hydroxycholesterol Production in Testicular Macrophages

Biology of reproduction, 2002-11, Vol.67 (5), p.1435-1438

2002

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Cardiac Overexpression of Human Adenylyl Cyclase Type 8 in Mice Elicits Phosphorylation- Dependent Mechanisms that Permit Perpetual Heart Exercise while Conferring Protection Against Excessive Camp-PKA Signaling

Biophysical journal, 2019-02, Vol.116 (3), p.117a-117a

2019

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Protein Phosphatase-1 Modulates Basal Spontaneous Beating Rate of Sinoatrial Node Cells (SANC)

Biophysical journal, 2018-02, Vol.114 (3), p.290a-290a

2018

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Concurrent PDE3 and PDE4 Activation Suppresses Local Ca2+ Releases (LCR) to Regulate Normal Spontaneous Firing of Sinoatrial Node Cells (SANC)

Biophysical journal, 2016-02, Vol.110 (3), p.273a-274a

2016

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Basal Spontaneous Firing of Rabbit Sinoatrial Node Cells is Regulated by Dual Phosphodiesterase 3 and 4 Activation

Circulation. Arrhythmia and electrophysiology, 2018-06, Vol.11 (6), p.e005896-e005896

2018

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Oxysterols in heart failure

Future cardiology, 2009-07, Vol.5 (4), p.343-354

2009

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A remarkable adaptive paradigm of heart performance and protection emerges in response to the constitutive challenge of marked cardiac-specific overexpression of adenylyl cyclase type 8

Ca(2+)/calmodulin-activated phosphodiesterase 1A is highly expressed in rabbit cardiac sinoatrial nodal cells and regulates pacemaker function

Ca²⁺-dependent phosphorylation of Ca²⁺ cycling proteins generates robust rhythmic local Ca²⁺ releases in cardiac pacemaker cells

Basal Spontaneous Firing of Rabbit Sinoatrial Node Cells Is Regulated by Dual Activation of PDEs (Phosphodiesterases) 3 and 4

Overexpression of a Neuronal Type Adenylyl Cyclase (Type 8) in Sinoatrial Node Markedly Impacts Heart Rate and Rhythm

Ca2+-stimulated Basal Adenylyl Cyclase Activity Localization in Membrane Lipid Microdomains of Cardiac Sinoatrial Nodal Pacemaker Cells

Phosphoprotein Phosphatase 1 but Not 2A Activity Modulates Coupled-Clock Mechanisms to Impact on Intrinsic Automaticity of Sinoatrial Nodal Pacemaker Cells

Changes in cAMP signaling are associated with age-related downregulation of spontaneously beating atrial tissue energetic indices

Ca(2+) -stimulated basal adenylyl cyclase activity localization in membrane lipid microdomains of cardiac sinoatrial nodal pacemaker cells

cAMP signaling affects age-associated deterioration of pacemaker beating interval dynamics

Sarcoplasmic reticulum Ca2+ cycling protein phosphorylation in a physiologic Ca2+ milieu unleashes a high-power, rhythmic Ca2+ clock in ventricular myocytes: Relevance to arrhythmias and bio-pacemaker design

Ca2+/calmodulin-activated phosphodiesterase 1A is highly expressed in rabbit cardiac sinoatrial nodal cells and regulates pacemaker function

A bioluminescence method for direct measurement of phosphodiesterase activity

cAMP/PKA signaling affects aged-deteriorated pacemaker beat interval dynamic: Antiaging approach

25-Hydroxycholesterol Is Produced by Testicular Macrophages During the Early Postnatal Period and Influences Differentiation of Leydig Cells In Vitro

Transcriptome and Proteome Alterations of Mice that Overexpress Adenylate Cyclase Type 8 Underlie a Chronic and Marked Increase in Sinoatrial Node (SAN) and Left Ventricle (LV) Performance While Ensuring Heart Survival

Metabolic Enzyme Acetylation is Elicited in Response to Stress Induced by Cardiac Specific Overexpression of Human ADCY8

Cardiac Specific Adenyl Cyclase Type 8 Overexpression Induces Chronic Cardiac Exercise and Leads to a Unique Form of Cardiac Growth.

Testosterone Regulates 25-Hydroxycholesterol Production in Testicular Macrophages

Cardiac Overexpression of Human Adenylyl Cyclase Type 8 in Mice Elicits Phosphorylation- Dependent Mechanisms that Permit Perpetual Heart Exercise while Conferring Protection Against Excessive Camp-PKA Signaling

Proteomes of primary skin fibroblasts from healthy individuals reveal altered cell responses across the life span

Protein Phosphatase-1 Modulates Basal Spontaneous Beating Rate of Sinoatrial Node Cells (SANC)

Concurrent PDE3 and PDE4 Activation Suppresses Local Ca2+ Releases (LCR) to Regulate Normal Spontaneous Firing of Sinoatrial Node Cells (SANC)

Basal Spontaneous Firing of Rabbit Sinoatrial Node Cells is Regulated by Dual Phosphodiesterase 3 and 4 Activation

Oxysterols in heart failure

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