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SOD1A4V aggregation alters ubiquitin homeostasis in a cell model of ALS

Journal of cell science, 2018-06, Vol.131 (11), p.jcs209122-jcs209122

2018

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The microglial NLRP3 inflammasome is activated by amyotrophic lateral sclerosis proteins

Glia, 2020-02, Vol.68 (2), p.407-421

2020

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Prion-Like Propagation of Protein Misfolding and Aggregation in Amyotrophic Lateral Sclerosis

Frontiers in molecular neuroscience, 2019-11, Vol.12, p.262-262

2019

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The cysteine-reactive small molecule ebselen facilitates effective SOD1 maturation

Nature communications, 2018-04, Vol.9 (1), p.1693-9, Article 1693

2018

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High-content analysis of proteostasis capacity in cellular models of amyotrophic lateral sclerosis (ALS)

Scientific reports, 2024-06, Vol.14 (1), p.13844-11

2024

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Amyotrophic Lateral Sclerosis: Proteins, Proteostasis, Prions, and Promises

Frontiers in cellular neuroscience, 2020-11, Vol.14, p.581907-581907

2020

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Amyloidogenic regions in beta-strands II and III modulate the aggregation and toxicity of SOD1 in living cells

Open biology, 2024-06, Vol.14 (6), p.230418

2024

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CuATSM improves motor function and extends survival but is not tolerated at a high dose in SOD1G93A mice with a C57BL/6 background

Scientific reports, 2021-09, Vol.11 (1), Article 19392

2021

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Protocol for the generation and automated confocal imaging of whole multi-cellular tumor spheroids

STAR protocols, 2023-06, Vol.4 (2), p.102331-102331, Article 102331

2023

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Strategies to promote the maturation of ALS-associated SOD1 mutants: small molecules return to the fold

Neural regeneration research, 2019-09, Vol.14 (9), p.1511-1512

2019

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Cerebrovascular amyloid Angiopathy in bioengineered vessels is reduced by high-density lipoprotein particles enriched in Apolipoprotein E

Molecular neurodegeneration, 2020-03, Vol.15 (1), p.23-23, Article 23

2020

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Ubiquitin Homeostasis Is Disrupted in TDP-43 and FUS Cell Models of ALS

iScience, 2020-11, Vol.23 (11), p.101700-101700, Article 101700

2020

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Assessment of protein inclusions in cultured cells using automated image analysis

STAR protocols, 2022-12, Vol.3 (4), p.101748-101748, Article 101748

2022

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Corrigendum: Prion-Like Propagation of Protein Misfolding and Aggregation in Amyotrophic Lateral Sclerosis

Frontiers in molecular neuroscience, 2020-01, Vol.12, p.311-311

2020

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Flow cytometric measurement of the cellular propagation of TDP-43 aggregation

Prion, 2017-05, Vol.11 (3), p.195-204

2017

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Proteome Homeostasis Dysfunction: A Unifying Principle in ALS Pathogenesis

Trends in neurosciences (Regular ed.), 2020-05, Vol.43 (5), p.274-284

2020

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Susceptibility of Mutant SOD1 to Form a Destabilized Monomer Predicts Cellular Aggregation and Toxicity but Not In vitro Aggregation Propensity

Frontiers in neuroscience, 2016-11, Vol.10, p.499-499

2016

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Emerging Developments in Targeting Proteotoxicity in Neurodegenerative Diseases

CNS drugs, 2019-09, Vol.33 (9), p.883-904

2019

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Tryptophan 32-mediated SOD1 aggregation is attenuated by pyrimidine-like compounds in living cells

Scientific reports, 2018-10, Vol.8 (1), p.15590-12, Article 15590

2018

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Neurodegenerative disease-associated protein aggregates are poor inducers of the heat shock response in neuronal cells

Journal of cell science, 2020-08, Vol.133 (15)

2020

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Rapid classification of a novel ALS-causing I149S variant in superoxide dismutase-1

Amyotrophic lateral sclerosis and frontotemporal degeneration, 2024-05, Vol.25 (5-6), p.1-614

2024

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RNA-binding properties orchestrate TDP-43 homeostasis through condensate formation in vivo

Nucleic acids research, 2024-05, Vol.52 (9), p.5301

2024

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Tryptophan residues in TDP-43 and SOD1 modulate the cross-seeding and toxicity of SOD1

The Journal of biological chemistry, 2024-05, Vol.300 (5), p.107207-107207, Article 107207

2024

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ALS-linked CCNF variant disrupts motor neuron ubiquitin homeostasis

Human molecular genetics, 2023-07, Vol.32 (14), p.2386-2398

2023

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Glutathionylation potentiates benign superoxide dismutase 1 variants to the toxic forms associated with amyotrophic lateral sclerosis

Scientific reports, 2013-11, Vol.3 (1), p.3275, Article 3275

2013

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SOD1A4V aggregation alters ubiquitin homeostasis in a cell model of ALS

The microglial NLRP3 inflammasome is activated by amyotrophic lateral sclerosis proteins

Prion-Like Propagation of Protein Misfolding and Aggregation in Amyotrophic Lateral Sclerosis

The cysteine-reactive small molecule ebselen facilitates effective SOD1 maturation

High-content analysis of proteostasis capacity in cellular models of amyotrophic lateral sclerosis (ALS)

Amyotrophic Lateral Sclerosis: Proteins, Proteostasis, Prions, and Promises

Amyloidogenic regions in beta-strands II and III modulate the aggregation and toxicity of SOD1 in living cells

CuATSM improves motor function and extends survival but is not tolerated at a high dose in SOD1G93A mice with a C57BL/6 background

Protocol for the generation and automated confocal imaging of whole multi-cellular tumor spheroids

Strategies to promote the maturation of ALS-associated SOD1 mutants: small molecules return to the fold

Cerebrovascular amyloid Angiopathy in bioengineered vessels is reduced by high-density lipoprotein particles enriched in Apolipoprotein E

Ubiquitin Homeostasis Is Disrupted in TDP-43 and FUS Cell Models of ALS

Assessment of protein inclusions in cultured cells using automated image analysis

Corrigendum: Prion-Like Propagation of Protein Misfolding and Aggregation in Amyotrophic Lateral Sclerosis

Flow cytometric measurement of the cellular propagation of TDP-43 aggregation

Proteome Homeostasis Dysfunction: A Unifying Principle in ALS Pathogenesis

Susceptibility of Mutant SOD1 to Form a Destabilized Monomer Predicts Cellular Aggregation and Toxicity but Not In vitro Aggregation Propensity

Emerging Developments in Targeting Proteotoxicity in Neurodegenerative Diseases

Tryptophan 32-mediated SOD1 aggregation is attenuated by pyrimidine-like compounds in living cells

Neurodegenerative disease-associated protein aggregates are poor inducers of the heat shock response in neuronal cells

Rapid classification of a novel ALS-causing I149S variant in superoxide dismutase-1

RNA-binding properties orchestrate TDP-43 homeostasis through condensate formation in vivo

Tryptophan residues in TDP-43 and SOD1 modulate the cross-seeding and toxicity of SOD1

ALS-linked CCNF variant disrupts motor neuron ubiquitin homeostasis

Glutathionylation potentiates benign superoxide dismutase 1 variants to the toxic forms associated with amyotrophic lateral sclerosis

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