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PINK1 autophosphorylation is required for ubiquitin recognition

EMBO reports, 2018-04, Vol.19 (4), p.n/a

2018

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From CFTR biology toward combinatorial pharmacotherapy: expanded classification of cystic fibrosis mutations

Molecular biology of the cell, 2016-02, Vol.27 (3), p.424-433

2016

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Cooperative assembly and misfolding of CFTR domains in vivo

Molecular biology of the cell, 2009-04, Vol.20 (7), p.1903-1915

2009

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Allosteric folding correction of F508del and rare CFTR mutants by elexacaftor-tezacaftor-ivacaftor (Trikafta) combination

JCI insight, 2020-09, Vol.5 (18)

2020

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Peripheral Protein Quality Control Removes Unfolded CFTR from the Plasma Membrane

Science (American Association for the Advancement of Science), 2010-08, Vol.329 (5993), p.805-810

2010

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Correction of Both NBD1 Energetics and Domain Interface Is Required to Restore ΔF508 CFTR Folding and Function

Cell, 2012-01, Vol.148 (1-2), p.150-163

2012

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Fixing cystic fibrosis by correcting CFTR domain assembly

The Journal of cell biology, 2012-10, Vol.199 (2), p.199-204

2012

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Selective Binding of HSC70 and its Co-Chaperones to Structural Hotspots on CFTR

Scientific reports, 2020-03, Vol.10 (1), p.4176-4176, Article 4176

2020

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Alcohol Disrupts Levels and Function of the Cystic Fibrosis Transmembrane Conductance Regulator to Promote Development of Pancreatitis

Gastroenterology (New York, N.Y. 1943), 2015-02, Vol.148 (2), p.427-439.e16

2015

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Mutation-specific downregulation of CFTR2 variants by gating potentiators

Human molecular genetics, 2017-12, Vol.26 (24), p.4873-4885

2017

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Synergy-based small-molecule screen using a human lung epithelial cell line yields ΔF508-CFTR correctors that augment VX-809 maximal efficacy

Molecular pharmacology, 2014-07, Vol.86 (1), p.42-51

2014

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Folding correctors can restore CFTR posttranslational folding landscape by allosteric domain-domain coupling

Nature communications, 2023-10, Vol.14 (1), p.6868-6868, Article 6868

2023

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Chaperones rescue the energetic landscape of mutant CFTR at single molecule and in cell

Nature communications, 2017-08, Vol.8 (1), p.398-15, Article 398

2017

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Protein homeostasis at the plasma membrane

Physiology (Bethesda, Md.), 2014-07, Vol.29 (4), p.265-277

2014

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Interplay of Endosomal pH and Ligand Occupancy in Integrin α5β1 Ubiquitination, Endocytic Sorting, and Cell Migration

Cell reports (Cambridge), 2015-10, Vol.13 (3), p.599-609

2015

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Proinflammatory cytokine secretion is suppressed by TMEM16A or CFTR channel activity in human cystic fibrosis bronchial epithelia

Molecular biology of the cell, 2012-11, Vol.23 (21), p.4188-4202

2012

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Protein quality control at the plasma membrane

Current opinion in cell biology, 2011-08, Vol.23 (4), p.483-491

2011

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Mutation-specific peripheral and ER quality control of hERG channel cell-surface expression

Scientific reports, 2019-04, Vol.9 (1), p.6066, Article 6066

2019

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Readthrough-induced misincorporated amino acid ratios guide mutant-specific therapeutic approaches for two CFTR nonsense mutations

Frontiers in pharmacology, 2024-04, Vol.15, p.1389586-1389586

2024

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Small heat shock proteins target mutant cystic fibrosis transmembrane conductance regulator for degradation via a small ubiquitin-like modifier-dependent pathway

Molecular biology of the cell, 2013-01, Vol.24 (2), p.74-84

2013

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Control of membrane protein homeostasis by a chaperone-like glial cell adhesion molecule at multiple subcellular locations

Scientific reports, 2021-09, Vol.11 (1), p.18435-18435, Article 18435

2021

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Extracellular oxidation in cystic fibrosis airway epithelium causes enhanced EGFR/ADAM17 activity

American journal of physiology. Lung cellular and molecular physiology, 2018-04, Vol.314 (4), p.L555-L568

2018

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Non-native Conformers of Cystic Fibrosis Transmembrane Conductance Regulator NBD1 Are Recognized by Hsp27 and Conjugated to SUMO-2 for Degradation

The Journal of biological chemistry, 2016-01, Vol.291 (4), p.2004-2017

2016

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Single-particle electron microscopy structure of UDP-glucose:glycoprotein glucosyltransferase suggests a selectivity mechanism for misfolded proteins

The Journal of biological chemistry, 2017-07, Vol.292 (27), p.11499-11507

2017

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N-glycans are direct determinants of CFTR folding and stability in secretory and endocytic membrane traffic

The Journal of cell biology, 2009-03, Vol.184 (6), p.847-862

2009

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PINK1 autophosphorylation is required for ubiquitin recognition

From CFTR biology toward combinatorial pharmacotherapy: expanded classification of cystic fibrosis mutations

Cooperative assembly and misfolding of CFTR domains in vivo

Allosteric folding correction of F508del and rare CFTR mutants by elexacaftor-tezacaftor-ivacaftor (Trikafta) combination

Peripheral Protein Quality Control Removes Unfolded CFTR from the Plasma Membrane

Correction of Both NBD1 Energetics and Domain Interface Is Required to Restore ΔF508 CFTR Folding and Function

Fixing cystic fibrosis by correcting CFTR domain assembly

Selective Binding of HSC70 and its Co-Chaperones to Structural Hotspots on CFTR

Alcohol Disrupts Levels and Function of the Cystic Fibrosis Transmembrane Conductance Regulator to Promote Development of Pancreatitis

Mutation-specific downregulation of CFTR2 variants by gating potentiators

Synergy-based small-molecule screen using a human lung epithelial cell line yields ΔF508-CFTR correctors that augment VX-809 maximal efficacy

Folding correctors can restore CFTR posttranslational folding landscape by allosteric domain-domain coupling

Chaperones rescue the energetic landscape of mutant CFTR at single molecule and in cell

Protein homeostasis at the plasma membrane

Interplay of Endosomal pH and Ligand Occupancy in Integrin α5β1 Ubiquitination, Endocytic Sorting, and Cell Migration

Proinflammatory cytokine secretion is suppressed by TMEM16A or CFTR channel activity in human cystic fibrosis bronchial epithelia

Protein quality control at the plasma membrane

Mutation-specific peripheral and ER quality control of hERG channel cell-surface expression

Readthrough-induced misincorporated amino acid ratios guide mutant-specific therapeutic approaches for two CFTR nonsense mutations

Small heat shock proteins target mutant cystic fibrosis transmembrane conductance regulator for degradation via a small ubiquitin-like modifier-dependent pathway

Control of membrane protein homeostasis by a chaperone-like glial cell adhesion molecule at multiple subcellular locations

Extracellular oxidation in cystic fibrosis airway epithelium causes enhanced EGFR/ADAM17 activity

Non-native Conformers of Cystic Fibrosis Transmembrane Conductance Regulator NBD1 Are Recognized by Hsp27 and Conjugated to SUMO-2 for Degradation

Single-particle electron microscopy structure of UDP-glucose:glycoprotein glucosyltransferase suggests a selectivity mechanism for misfolded proteins

N-glycans are direct determinants of CFTR folding and stability in secretory and endocytic membrane traffic

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