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Epigenetics in Alternative Pre-mRNA Splicing

Cell, 2011-01, Vol.144 (1), p.16-26

2011

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A lncRNA regulates alternative splicing via establishment of a splicing-specific chromatin signature

Nature structural & molecular biology, 2015-05, Vol.22 (5), p.370-376

2015

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Regulation of Alternative Splicing by Histone Modifications

Science (American Association for the Advancement of Science), 2010-02, Vol.327 (5968), p.996-1000

2010

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IRFinder-S: a comprehensive suite to discover and explore intron retention

Genome Biology, 2021-11, Vol.22 (1), p.307-307, Article 307

2021

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More than a splicing code: integrating the role of RNA, chromatin and non-coding RNA in alternative splicing regulation

Current opinion in genetics & development, 2011-08, Vol.21 (4), p.366-372

2011

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A CRISPR-dCas13 RNA-editing tool to study alternative splicing

Nucleic acids research, 2024-08

2024

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A cell-to-patient machine learning transfer approach uncovers novel basal-like breast cancer prognostic markers amongst alternative splice variants

BMC biology, 2021-04, Vol.19 (1), p.70-70, Article 70

2021

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SETDB1-dependent heterochromatin stimulates alternative lengthening of telomeres

Science advances, 2019-05, Vol.5 (5), p.eaav3673-eaav3673

2019

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Retrotransposons jump into alternative-splicing regulation via a long noncoding RNA

Nature structural & molecular biology, 2016-11, Vol.23 (11), p.952-954

Luco, Reini F

2016

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Histone marks regulate the epithelial-to-mesenchymal transition via alternative splicing

Cell reports (Cambridge), 2022-02, Vol.38 (7), p.110357-110357, Article 110357

2022

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A complex epigenome-splicing crosstalk governs epithelial-to-mesenchymal transition in metastasis and brain development

Nature cell biology, 2022-08, Vol.24 (8), p.1265-1277

2022

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IA1 is NGN3-dependent and essential for differentiation of the endocrine pancreas

The EMBO journal, 2006-03, Vol.25 (6), p.1344-1352

2006

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A Conditional Model Reveals That Induction of Hepatocyte Nuclear Factor-1α in Hnf1α-Null Mutant β-Cells Can Activate Silenced Genes Postnatally, Whereas Overexpression Is Deleterious

Diabetes (New York, N.Y.), 2006-08, Vol.55 (8), p.2202-2211

2006

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non-coding genome: a universe in expansion for fine-tuning the coding world

Genome biology, 2013-11, Vol.14 (11), p.314-314

Luco, Reini F

2013

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Hnf6 and Tcf2 (MODY5) are linked in a gene network operating in a precursor cell domain of the embryonic pancreas

Human molecular genetics, 2003-12, Vol.12 (24), p.3307-3314

2003

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Targeted Deficiency of the Transcriptional Activator Hnf1α Alters Subnuclear Positioning of Its Genomic Targets

PLoS genetics, 2008-05, Vol.4 (5), p.e1000079

2008

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Targeted deficiency of the transcriptional activator Hnf1alpha alters subnuclear positioning of its genomic targets

PLoS genetics, 2008-05, Vol.4 (5), p.e1000079-e1000079

2008

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Chromatin immunoprecipitation using isolated islets of Langerhans

Methods in molecular medicine, 2003, Vol.83, p.61

2003

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Distinct Roles of HNF1 Β , HNF1 α , and HNF4 α in Regulating Pancreas Development, Β -Cell Function and Growth

2007

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Targeted Deficiency of the Transcriptional Activator Hnf1[alpha] Alters Subnuclear Positioning of Its Genomic Targets: e1000079

PLoS genetics, 2008-05, Vol.4 (5)

2008

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A Conditional Model Reveals That Induction of Hepatocyte Nuclear Factor-1[alpha] in Hnf1[alpha]-Null Mutant [beta]-Cells Can Activate Silenced Genes Postnatally, Whereas Overexpression Is Deleterious

Diabetes (New York, N.Y.), 2006-08, Vol.55 (8), p.2202

2006

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A conditional model reveals that induction of hepatocyte nuclear factor-1alpha in Hnf1alpha-null mutant beta-cells can activate silenced genes postnatally, whereas overexpression is deleterious

Diabetes (New York, N.Y.), 2006-08, Vol.55 (8), p.2202-2211

2006

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A Conditional Model Reveals That Induction of Hepatocyte Nuclear Factor-1 alpha in Hnf1 alpha -Null Mutant {szligbeta}-Cells Can Activate Silenced Genes Postnatally, Whereas Overexpression Is Deleterious

Diabetes (New York, N.Y.), 2006-08, Vol.55 (8), p.2202-2211

2006

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Distinct roles of HNF1beta, HNF1alpha, and HNF4alpha in regulating pancreas development, beta-cell function and growth

Endocrine development, 2007, Vol.12, p.33-45

2007

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Histone Marks Regulate the Epithelial-to-Mesenchymal Transition via Alternative Splicing

SSRN Electronic Journal

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Epigenetics in Alternative Pre-mRNA Splicing

A lncRNA regulates alternative splicing via establishment of a splicing-specific chromatin signature

Regulation of Alternative Splicing by Histone Modifications

IRFinder-S: a comprehensive suite to discover and explore intron retention

More than a splicing code: integrating the role of RNA, chromatin and non-coding RNA in alternative splicing regulation

A CRISPR-dCas13 RNA-editing tool to study alternative splicing

A cell-to-patient machine learning transfer approach uncovers novel basal-like breast cancer prognostic markers amongst alternative splice variants

SETDB1-dependent heterochromatin stimulates alternative lengthening of telomeres

Retrotransposons jump into alternative-splicing regulation via a long noncoding RNA

Histone marks regulate the epithelial-to-mesenchymal transition via alternative splicing

A complex epigenome-splicing crosstalk governs epithelial-to-mesenchymal transition in metastasis and brain development

IA1 is NGN3-dependent and essential for differentiation of the endocrine pancreas

A Conditional Model Reveals That Induction of Hepatocyte Nuclear Factor-1α in Hnf1α-Null Mutant β-Cells Can Activate Silenced Genes Postnatally, Whereas Overexpression Is Deleterious

non-coding genome: a universe in expansion for fine-tuning the coding world

Hnf6 and Tcf2 (MODY5) are linked in a gene network operating in a precursor cell domain of the embryonic pancreas

Targeted Deficiency of the Transcriptional Activator Hnf1α Alters Subnuclear Positioning of Its Genomic Targets

Targeted deficiency of the transcriptional activator Hnf1alpha alters subnuclear positioning of its genomic targets

Chromatin immunoprecipitation using isolated islets of Langerhans

Distinct Roles of HNF1 Β , HNF1 α , and HNF4 α in Regulating Pancreas Development, Β -Cell Function and Growth

Targeted Deficiency of the Transcriptional Activator Hnf1[alpha] Alters Subnuclear Positioning of Its Genomic Targets: e1000079

A Conditional Model Reveals That Induction of Hepatocyte Nuclear Factor-1[alpha] in Hnf1[alpha]-Null Mutant [beta]-Cells Can Activate Silenced Genes Postnatally, Whereas Overexpression Is Deleterious

A conditional model reveals that induction of hepatocyte nuclear factor-1alpha in Hnf1alpha-null mutant beta-cells can activate silenced genes postnatally, whereas overexpression is deleterious

A Conditional Model Reveals That Induction of Hepatocyte Nuclear Factor-1 alpha in Hnf1 alpha -Null Mutant {szligbeta}-Cells Can Activate Silenced Genes Postnatally, Whereas Overexpression Is Deleterious

Distinct roles of HNF1beta, HNF1alpha, and HNF4alpha in regulating pancreas development, beta-cell function and growth

Histone Marks Regulate the Epithelial-to-Mesenchymal Transition via Alternative Splicing

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