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Massive palmitoylation-dependent endocytosis during reoxygenation of anoxic cardiac muscle

eLife, 2013-11, Vol.2, p.e01295-e01295

2013

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Intrathecal enzyme replacement therapy improves motor function and survival in a preclinical mouse model of infantile neuronal ceroid lipofuscinosis

Molecular genetics and metabolism, 2015-09, Vol.116 (1-2), p.98-105

2015

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Incidence and Risk Factors for Extremity Osteoradionecrosis after Limb-Sparing Surgery and Adjuvant Radiotherapy

Cancers, 2023-04, Vol.15 (8), p.2339

2023

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A mutation in canine PPT1 causes early onset neuronal ceroid lipofuscinosis in a Dachshund

Molecular genetics and metabolism, 2010-08, Vol.100 (4), p.349-356

2010

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Cross-Linked Enzyme Aggregates as Versatile Tool for Enzyme Delivery: Application to Polymeric Nanoparticles

Bioconjugate chemistry, 2018-07, Vol.29 (7), p.2225-2231

2018

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Perfusion Dynamics during Secondary Flap Debulking with Liposuction

Journal of reconstructive microsurgery, 2024-07, Vol.40 (6), p.423

2024

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The Crystal Structure of Palmitoyl Protein Thioesterase 1 and the Molecular Basis of Infantile Neuronal Ceroid Lipofuscinosis

Proceedings of the National Academy of Sciences - PNAS, 2000-04, Vol.97 (9), p.4573-4578

2000

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A 10-year experience in microsurgical reconstruction of the nose with a lower extremity flap

Journal of plastic, reconstructive & aesthetic surgery, 2023-10, Vol.85, p.10-17

2023

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Gene expression profiling in a mouse model of infantile neuronal ceroid lipofuscinosis reveals upregulation of immediate early genes and mediators of the inflammatory response

BMC neuroscience, 2007-11, Vol.8 (1), p.95-95, Article 95

2007

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Cross-species efficacy of enzyme replacement therapy for CLN1 disease in mice and sheep

The Journal of clinical investigation, 2022-10, Vol.132 (20), p.1-11

2022

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Systematic siRNA Screen Unmasks NSCLC Growth Dependence by Palmitoyltransferase DHHC5

Molecular cancer research, 2015-04, Vol.13 (4), p.784-794

2015

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Human recombinant palmitoyl-protein thioesterase-1 (PPT1) for preclinical evaluation of enzyme replacement therapy for infantile neuronal ceroid lipofuscinosis

Molecular genetics and metabolism, 2010-04, Vol.99 (4), p.374-378

2010

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Intravenous high-dose enzyme replacement therapy with recombinant palmitoyl–protein thioesterase reduces visceral lysosomal storage and modestly prolongs survival in a preclinical mouse model of infantile neuronal ceroid lipofuscinosis

Molecular genetics and metabolism, 2012-09, Vol.107 (1-2), p.213-221

2012

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Neuronal ceroid lipofuscinoses caused by defects in soluble lysosomal enzymes (CLN1 and CLN2)

Current molecular medicine, 2002-08, Vol.2 (5), p.423-437

2002

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Molecular genetics of palmitoyl-protein thioesterase deficiency in the U.S

The Journal of clinical investigation, 1998-07, Vol.102 (2), p.361-370

1998

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Thematic review series: Lipid Posttranslational Modifications. Lysosomal metabolism of lipid-modified proteins

Journal of lipid research, 2006-07, Vol.47 (7), p.1352-1357

2006

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Lipid Thioesters Derived from Acylated Proteins Accumulate in Infantile Neuronal Ceroid Lipofuscinosis: Correction of the Defect in Lymphoblasts by Recombinant Palmitoyl-Protein Thioesterase

Proceedings of the National Academy of Sciences - PNAS, 1996-09, Vol.93 (19), p.10046-10050

1996

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The effects of lysosomotropic agents on normal and INCL cells provide further evidence for the lysosomal nature of palmitoyl-protein thioesterase function

Biochimica et biophysica acta, 2002-06, Vol.1583 (1), p.35-44

2002

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Molecular basis of infantile Batten disease: impact of disordered palmitoylated protein degradation on the nervous system (466.1)

The FASEB journal, 2014-04, Vol.28 (S1), p.n/a

2014

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Biochemical analysis of mutations in palmitoyl-protein thioesterase causing infantile and late-onset forms of neuronal ceroid lipofuscinosis

Human molecular genetics, 2001-06, Vol.10 (13), p.1431-1439

2001

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Genotype–Phenotype Correlations in Neuronal Ceroid Lipofuscinosis Due to Palmitoyl-Protein Thioesterase Deficiency

Molecular genetics and metabolism, 1999-04, Vol.66 (4), p.234-239

1999

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Depalmitoylation of CAAX motif proteins. Protein structural determinants of palmitate turnover rate

The Journal of biological chemistry, 1995-03, Vol.270 (13), p.7251-7256

1995

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3 Positional candidate gene cloning of CLN1

Advances in Genetics, 2001, Vol.45, p.69-92

2001

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Palmitoyl-protein thioesterase and the molecular pathogenesis of infantile neuronal ceroid lipofuscinosis

Neuropediatrics, 1997-02, Vol.28 (1), p.27-30

1997

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Crystallization and preliminary x-ray diffraction analysis of P450terp and the hemoprotein domain of P450BM-3, enzymes belonging to two distinct classes of the cytochrome P450 superfamily

Proceedings of the National Academy of Sciences - PNAS, 1992-06, Vol.89 (12), p.5567-5571

1992

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Massive palmitoylation-dependent endocytosis during reoxygenation of anoxic cardiac muscle

Intrathecal enzyme replacement therapy improves motor function and survival in a preclinical mouse model of infantile neuronal ceroid lipofuscinosis

Incidence and Risk Factors for Extremity Osteoradionecrosis after Limb-Sparing Surgery and Adjuvant Radiotherapy

A mutation in canine PPT1 causes early onset neuronal ceroid lipofuscinosis in a Dachshund

Cross-Linked Enzyme Aggregates as Versatile Tool for Enzyme Delivery: Application to Polymeric Nanoparticles

Perfusion Dynamics during Secondary Flap Debulking with Liposuction

The Crystal Structure of Palmitoyl Protein Thioesterase 1 and the Molecular Basis of Infantile Neuronal Ceroid Lipofuscinosis

A 10-year experience in microsurgical reconstruction of the nose with a lower extremity flap

Gene expression profiling in a mouse model of infantile neuronal ceroid lipofuscinosis reveals upregulation of immediate early genes and mediators of the inflammatory response

Cross-species efficacy of enzyme replacement therapy for CLN1 disease in mice and sheep

Systematic siRNA Screen Unmasks NSCLC Growth Dependence by Palmitoyltransferase DHHC5

Human recombinant palmitoyl-protein thioesterase-1 (PPT1) for preclinical evaluation of enzyme replacement therapy for infantile neuronal ceroid lipofuscinosis

Intravenous high-dose enzyme replacement therapy with recombinant palmitoyl–protein thioesterase reduces visceral lysosomal storage and modestly prolongs survival in a preclinical mouse model of infantile neuronal ceroid lipofuscinosis

Neuronal ceroid lipofuscinoses caused by defects in soluble lysosomal enzymes (CLN1 and CLN2)

Molecular genetics of palmitoyl-protein thioesterase deficiency in the U.S

Thematic review series: Lipid Posttranslational Modifications. Lysosomal metabolism of lipid-modified proteins

Lipid Thioesters Derived from Acylated Proteins Accumulate in Infantile Neuronal Ceroid Lipofuscinosis: Correction of the Defect in Lymphoblasts by Recombinant Palmitoyl-Protein Thioesterase

The effects of lysosomotropic agents on normal and INCL cells provide further evidence for the lysosomal nature of palmitoyl-protein thioesterase function

Molecular basis of infantile Batten disease: impact of disordered palmitoylated protein degradation on the nervous system (466.1)

Biochemical analysis of mutations in palmitoyl-protein thioesterase causing infantile and late-onset forms of neuronal ceroid lipofuscinosis

Genotype–Phenotype Correlations in Neuronal Ceroid Lipofuscinosis Due to Palmitoyl-Protein Thioesterase Deficiency

Depalmitoylation of CAAX motif proteins. Protein structural determinants of palmitate turnover rate

3 Positional candidate gene cloning of CLN1

Palmitoyl-protein thioesterase and the molecular pathogenesis of infantile neuronal ceroid lipofuscinosis

Crystallization and preliminary x-ray diffraction analysis of P450terp and the hemoprotein domain of P450BM-3, enzymes belonging to two distinct classes of the cytochrome P450 superfamily

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