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The Mechanisms of Calcium Cycling and Action Potential Dynamics in Cardiac Alternans

Circulation research, 2015-02, Vol.116 (5), p.846-856

2015

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The intricacies of atrial calcium cycling during excitation-contraction coupling

The Journal of general physiology, 2017-09, Vol.149 (9), p.857-865

Blatter, Lothar A

2017

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Distinct mPTP activation mechanisms in ischaemia-reperfusion: contributions of Ca2+, ROS, pH, and inorganic polyphosphate

Cardiovascular research, 2015-05, Vol.106 (2), p.237-248

2015

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Role of β-hydroxybutyrate, its polymer poly-β-hydroxybutyrate and inorganic polyphosphate in mammalian health and disease

Frontiers in physiology, 2014, Vol.5, p.260-260

2014

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Cardiac alternans and intracellular calcium cycling

Clinical and experimental pharmacology & physiology, 2014-07, Vol.41 (7), p.524-532

2014

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A novel mechanism of tandem activation of ryanodine receptors by cytosolic and SR luminal Ca2+ during excitation–contraction coupling in atrial myocytes

The Journal of physiology, 2017-06, Vol.595 (12), p.3835-3845

2017

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Measuring mitochondrial function in intact cardiac myocytes

Journal of molecular and cellular cardiology, 2012-01, Vol.52 (1), p.48-61

2012

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Mechanism of carvedilol induced action potential and calcium alternans

Channels (Austin, Tex.), 2022-12, Vol.16 (1), p.97-112

2022

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Increased Risk for Atrial Alternans in Rabbit Heart Failure: The Role of Ca2+/Calmodulin-Dependent Kinase II and Inositol-1,4,5-trisphosphate Signaling

Biomolecules (Basel, Switzerland), 2024-01, Vol.14 (1), p.53

2024

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The 'Reverse FDUF' Mechanism of Atrial Excitation-Contraction Coupling Sustains Calcium Alternans-A Hypothesis

Biomolecules (Basel, Switzerland), 2022-12, Vol.13 (1), p.7

2022

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Dantrolene prevents arrhythmogenic Ca2+ release in heart failure

American journal of physiology. Heart and circulatory physiology, 2012-02, Vol.302 (4), p.H953-H963

2012

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Role of Mitochondrial ROS for Calcium Alternans in Atrial Myocytes

Biomolecules (Basel, Switzerland), 2024-01, Vol.14 (2), p.144

2024

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Inositol‐1,4,5‐trisphosphate induced Ca2+ release and excitation–contraction coupling in atrial myocytes from normal and failing hearts

The Journal of physiology, 2015-03, Vol.593 (6), p.1459-1477

2015

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Refractoriness of sarcoplasmic reticulum Ca2+ release determines Ca2+ alternans in atrial myocytes

American journal of physiology. Heart and circulatory physiology, 2012-06, Vol.302 (11), p.H2310-H2320

2012

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Characteristics and function of cardiac mitochondrial nitric oxide synthase

The Journal of physiology, 2009-02, Vol.587 (4), p.851-872

2009

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Alternans in atria: Mechanisms and clinical relevance

Medicina (Kaunas, Lithuania), 2017, Vol.53 (3), p.139-149

2017

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Emerging roles of inositol 1,4,5-trisphosphate signaling in cardiac myocytes

Journal of molecular and cellular cardiology, 2008-08, Vol.45 (2), p.128-147

2008

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Dynamic Calcium Movement Inside Cardiac Sarcoplasmic Reticulum During Release

Circulation research, 2011-04, Vol.108 (7), p.847-856

2011

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Alteration of sarcoplasmic reticulum Ca2+ release termination by ryanodine receptor sensitization and in heart failure

The Journal of physiology, 2009-11, Vol.587 (21), p.5197-5209

2009

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L‐type Ca2+ channel recovery from inactivation in rabbit atrial myocytes

Physiological reports, 2022-03, Vol.10 (5), p.e15222-n/a

2022

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Activation of small conductance Ca2+‐activated K+ channels suppresses Ca2+ transient and action potential alternans in ventricular myocytes

The Journal of physiology, 2023-01, Vol.601 (1), p.51-67

2023

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Activation of small conductance Ca 2+ -activated K + channels suppresses Ca 2+ transient and action potential alternans in ventricular myocytes

The Journal of physiology, 2023-01, Vol.601 (1), p.51-67

2023

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Activation of NFATc1 is directly mediated by IP3 in adult cardiac myocytes

American journal of physiology. Heart and circulatory physiology, 2010-11, Vol.299 (5), p.H1701-H1707

2010

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Calcium and IP3 dynamics in cardiac myocytes: experimental and computational perspectives and approaches

Frontiers in pharmacology, 2014-03, Vol.5, p.35-35

2014

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Ca(2+) release events in cardiac myocytes up close: insights from fast confocal imaging

PloS one, 2013, Vol.8 (4), p.e61525-e61525

2013

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The Mechanisms of Calcium Cycling and Action Potential Dynamics in Cardiac Alternans

The intricacies of atrial calcium cycling during excitation-contraction coupling

Distinct mPTP activation mechanisms in ischaemia-reperfusion: contributions of Ca2+, ROS, pH, and inorganic polyphosphate

Role of β-hydroxybutyrate, its polymer poly-β-hydroxybutyrate and inorganic polyphosphate in mammalian health and disease

Cardiac alternans and intracellular calcium cycling

A novel mechanism of tandem activation of ryanodine receptors by cytosolic and SR luminal Ca2+ during excitation–contraction coupling in atrial myocytes

Measuring mitochondrial function in intact cardiac myocytes

Mechanism of carvedilol induced action potential and calcium alternans

Increased Risk for Atrial Alternans in Rabbit Heart Failure: The Role of Ca2+/Calmodulin-Dependent Kinase II and Inositol-1,4,5-trisphosphate Signaling

The 'Reverse FDUF' Mechanism of Atrial Excitation-Contraction Coupling Sustains Calcium Alternans-A Hypothesis

Dantrolene prevents arrhythmogenic Ca2+ release in heart failure

Role of Mitochondrial ROS for Calcium Alternans in Atrial Myocytes

Inositol‐1,4,5‐trisphosphate induced Ca2+ release and excitation–contraction coupling in atrial myocytes from normal and failing hearts

Refractoriness of sarcoplasmic reticulum Ca2+ release determines Ca2+ alternans in atrial myocytes

Characteristics and function of cardiac mitochondrial nitric oxide synthase

Alternans in atria: Mechanisms and clinical relevance

Emerging roles of inositol 1,4,5-trisphosphate signaling in cardiac myocytes

Dynamic Calcium Movement Inside Cardiac Sarcoplasmic Reticulum During Release

Alteration of sarcoplasmic reticulum Ca2+ release termination by ryanodine receptor sensitization and in heart failure

L‐type Ca2+ channel recovery from inactivation in rabbit atrial myocytes

Activation of small conductance Ca2+‐activated K+ channels suppresses Ca2+ transient and action potential alternans in ventricular myocytes

Activation of small conductance Ca 2+ -activated K + channels suppresses Ca 2+ transient and action potential alternans in ventricular myocytes

Activation of NFATc1 is directly mediated by IP3 in adult cardiac myocytes

Calcium and IP3 dynamics in cardiac myocytes: experimental and computational perspectives and approaches

Ca(2+) release events in cardiac myocytes up close: insights from fast confocal imaging

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