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Impact of loss of SOAT2 function on disease progression in the lysosomal acid lipase-deficient mouse

Steroids, 2018-02, Vol.130, p.7-14

2018

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Ontogenesis and Modulation of Intestinal Unesterified Cholesterol Sequestration in a Mouse Model of Niemann–Pick C1 Disease

Digestive diseases and sciences, 2020-01, Vol.65 (1), p.158-167

2020

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Deletion of sterol O-acyltransferase 2 (SOAT2) function in mice deficient in lysosomal acid lipase (LAL) dramatically reduces esterified cholesterol sequestration in the small intestine and liver

Biochemical and biophysical research communications, 2014-11, Vol.454 (1), p.162-166

2014

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Hepatic entrapment of esterified cholesterol drives continual expansion of whole body sterol pool in lysosomal acid lipase-deficient mice

American journal of physiology: Gastrointestinal and liver physiology, 2014-10, Vol.307 (8), p.G836-G847

2014

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Ezetimibe markedly attenuates hepatic cholesterol accumulation and improves liver function in the lysosomal acid lipase-deficient mouse, a model for cholesteryl ester storage disease

Biochemical and biophysical research communications, 2014-01, Vol.443 (3), p.1073-1077

2014

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Impact of physiological levels of chenodeoxycholic acid supplementation on intestinal and hepatic bile acid and cholesterol metabolism in Cyp7a1-deficient mice

Steroids, 2015-01, Vol.93, p.87-95

2015

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Lysosomal Acid Lipase Activity: A Tool for the Detection and Management of Fatty Liver Disease?

EBioMedicine, 2015-07, Vol.2 (7), p.638-639

2015

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Suppression of brain cholesterol synthesis in male Mecp2-deficient mice is age dependent and not accompanied by a concurrent change in the rate of fatty acid synthesis

Brain research, 2017-01, Vol.1654 (Pt A), p.77-84

2017

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Molecular markers of brain cholesterol homeostasis are unchanged despite a smaller brain mass in a mouse model of cholesteryl ester storage disease

Lipids, 2022-01, Vol.57 (1), p.3-16

2022

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Quantitation of the rates of hepatic and intestinal cholesterol synthesis in lysosomal acid lipase-deficient mice before and during treatment with ezetimibe

Biochemical pharmacology, 2017-07, Vol.135, p.116-125

2017

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Niemann-Pick C1-deficient mice lacking sterol O-acyltransferase 2 have less hepatic cholesterol entrapment and improved liver function

American journal of physiology: Gastrointestinal and liver physiology, 2018-10, Vol.315 (4), p.G454-G463

2018

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Delineation of metabolic responses of Npc1−/−nih mice lacking the cholesterol-esterifying enzyme SOAT2 to acute treatment with 2-hydroxypropyl-β-cyclodextrin

Steroids, 2020-12, Vol.164, p.108725-108725, Article 108725

2020

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Measurement of Rates of Cholesterol and Fatty Acid Synthesis In Vivo Using Tritiated Water

Methods in molecular biology (Clifton, N.J.), 2017-01, Vol.1583, p.241

2017

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Systemic administration of 2-hydroxypropyl-β-cyclodextrin to symptomatic Npc1-deficient mice slows cholesterol sequestration in the major organs and improves liver function

Clinical and experimental pharmacology & physiology, 2014-10, Vol.41 (10), p.780-787

2014

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Identification of Correlative Shifts in Indices of Brain Cholesterol Metabolism in the C57BL6/Mecp2 tm1.1Bird Mouse, a Model for Rett Syndrome

Lipids, 2018-04, Vol.53 (4), p.363-373

2018

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Identification of Correlative Shifts in Indices of Brain Cholesterol Metabolism in the C57BL6/Mecp2tm1.1Bird Mouse, a Model for Rett Syndrome

Lipids, 2018-04, Vol.53 (4), p.363-373

2018

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Corrigenda to “Suppression of brain cholesterol synthesis in male Mecp2-deficient mice is age dependent and not accompanied by a concurrent change in the rate of fatty acid synthesis” [Brain Res. 1654 (2017) 77–84]

Brain research, 2017-02, Vol.1657, p.383-383

2017

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PRD125, a potent and selective inhibitor of sterol O-acyltransferase 2 markedly reduces hepatic cholesteryl ester accumulation and improves liver function in lysosomal acid lipase-deficient mice

The Journal of pharmacology and experimental therapeutics, 2015-11, Vol.355 (2), p.159

2015

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Ontogenic changes in lung cholesterol metabolism, lipid content, and histology in mice with Niemann–Pick type C disease

Biochimica et biophysica acta, 2014-01, Vol.1841 (1), p.54-61

2014

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Sustained and selective suppression of intestinal cholesterol synthesis by Ro 48-8071, an inhibitor of 2,3-oxidosqualene:lanosterol cyclase, in the BALB/c mouse

Biochemical pharmacology, 2014-04, Vol.88 (3), p.351-363

2014

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Impact of the loss of caveolin-1 on lung mass and cholesterol metabolism in mice with and without the lysosomal cholesterol transporter, Niemann–Pick type C1

Biochimica et biophysica acta, 2014-07, Vol.1841 (7), p.995-1002

2014

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Delineation of metabolic responses of Npc1 mice lacking the cholesterol-esterifying enzyme SOAT2 to acute treatment with 2-hydroxypropyl-β-cyclodextrin

Steroids, 2020-12, Vol.164, p.108725, Article 108725

2020

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434: MICROBIOME-PROMOTED HYPERPHAGIA AND OBESITY ARE CONTROLLED BY ENDOCRINE PATHWAYS IN THE COLON

Gastroenterology (New York, N.Y. 1943), 2022-05, Vol.162 (7), p.S-95-S-95

2022

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688 COLONIC ENTEROENDOCRINE CELLS CONTROL HOST METABOLISM THROUGH MICROBIAL REGULATION

Gastroenterology (New York, N.Y. 1943), 2024-05, Vol.166 (5), p.S-159-S-159

2024

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Systemic administration of 2-hydroxypropyl-[beta]-cyclodextrin to symptomatic Npc1-deficient mice slows cholesterol sequestration in the major organs and improves liver function

Clinical and experimental pharmacology & physiology, 2014-10, Vol.41 (10), p.780

2014

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Impact of loss of SOAT2 function on disease progression in the lysosomal acid lipase-deficient mouse

Ontogenesis and Modulation of Intestinal Unesterified Cholesterol Sequestration in a Mouse Model of Niemann–Pick C1 Disease

Deletion of sterol O-acyltransferase 2 (SOAT2) function in mice deficient in lysosomal acid lipase (LAL) dramatically reduces esterified cholesterol sequestration in the small intestine and liver

Hepatic entrapment of esterified cholesterol drives continual expansion of whole body sterol pool in lysosomal acid lipase-deficient mice

Ezetimibe markedly attenuates hepatic cholesterol accumulation and improves liver function in the lysosomal acid lipase-deficient mouse, a model for cholesteryl ester storage disease

Impact of physiological levels of chenodeoxycholic acid supplementation on intestinal and hepatic bile acid and cholesterol metabolism in Cyp7a1-deficient mice

Lysosomal Acid Lipase Activity: A Tool for the Detection and Management of Fatty Liver Disease?

Suppression of brain cholesterol synthesis in male Mecp2-deficient mice is age dependent and not accompanied by a concurrent change in the rate of fatty acid synthesis

Molecular markers of brain cholesterol homeostasis are unchanged despite a smaller brain mass in a mouse model of cholesteryl ester storage disease

Quantitation of the rates of hepatic and intestinal cholesterol synthesis in lysosomal acid lipase-deficient mice before and during treatment with ezetimibe

Niemann-Pick C1-deficient mice lacking sterol O-acyltransferase 2 have less hepatic cholesterol entrapment and improved liver function

Delineation of metabolic responses of Npc1−/−nih mice lacking the cholesterol-esterifying enzyme SOAT2 to acute treatment with 2-hydroxypropyl-β-cyclodextrin

Measurement of Rates of Cholesterol and Fatty Acid Synthesis In Vivo Using Tritiated Water

Systemic administration of 2-hydroxypropyl-β-cyclodextrin to symptomatic Npc1-deficient mice slows cholesterol sequestration in the major organs and improves liver function

Identification of Correlative Shifts in Indices of Brain Cholesterol Metabolism in the C57BL6/Mecp2 tm1.1Bird Mouse, a Model for Rett Syndrome

Identification of Correlative Shifts in Indices of Brain Cholesterol Metabolism in the C57BL6/Mecp2tm1.1Bird Mouse, a Model for Rett Syndrome

Corrigenda to “Suppression of brain cholesterol synthesis in male Mecp2-deficient mice is age dependent and not accompanied by a concurrent change in the rate of fatty acid synthesis” [Brain Res. 1654 (2017) 77–84]

PRD125, a potent and selective inhibitor of sterol O-acyltransferase 2 markedly reduces hepatic cholesteryl ester accumulation and improves liver function in lysosomal acid lipase-deficient mice

Ontogenic changes in lung cholesterol metabolism, lipid content, and histology in mice with Niemann–Pick type C disease

Sustained and selective suppression of intestinal cholesterol synthesis by Ro 48-8071, an inhibitor of 2,3-oxidosqualene:lanosterol cyclase, in the BALB/c mouse

Impact of the loss of caveolin-1 on lung mass and cholesterol metabolism in mice with and without the lysosomal cholesterol transporter, Niemann–Pick type C1

Delineation of metabolic responses of Npc1 mice lacking the cholesterol-esterifying enzyme SOAT2 to acute treatment with 2-hydroxypropyl-β-cyclodextrin

434: MICROBIOME-PROMOTED HYPERPHAGIA AND OBESITY ARE CONTROLLED BY ENDOCRINE PATHWAYS IN THE COLON

688 COLONIC ENTEROENDOCRINE CELLS CONTROL HOST METABOLISM THROUGH MICROBIAL REGULATION

Systemic administration of 2-hydroxypropyl-[beta]-cyclodextrin to symptomatic Npc1-deficient mice slows cholesterol sequestration in the major organs and improves liver function

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