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Roles for KRAS in Pancreatic Tumor Development and Progression

Gastroenterology (New York, N.Y. 1943), 2013-06, Vol.144 (6), p.1220-1229

2013

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Activation of Nuclear Factor-κB in Acinar Cells Increases the Severity of Pancreatitis in Mice

Gastroenterology (New York, N.Y. 1943), 2013-01, Vol.144 (1), p.202-210

2013

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A Novel FoxM1-Caveolin Signaling Pathway Promotes Pancreatic Cancer Invasion and Metastasis

Cancer research (Chicago, Ill.), 2012-02, Vol.72 (3), p.655-665

2012

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The role of protein synthesis and digestive enzymes in acinar cell injury

Nature reviews. Gastroenterology & hepatology, 2013-06, Vol.10 (6), p.362

2013

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Cell surface lactate receptor GPR81 is crucial for cancer cell survival

Cancer research (Chicago, Ill.), 2014-09, Vol.74 (18), p.5301-5310

2014

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Dynamic Mast Cell-Stromal Cell Interactions Promote Growth of Pancreatic Cancer

Cancer research (Chicago, Ill.), 2013-07, Vol.73 (13), p.3927-3937

2013

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Small interfering RNAs (siRNAs) in cancer therapy: a nano-based approach

International journal of nanomedicine, 2019-05, Vol.14, p.3111-3128

2019

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Galectin-3 Mediates Tumor Cell–Stroma Interactions by Activating Pancreatic Stellate Cells to Produce Cytokines via Integrin Signaling

Gastroenterology (New York, N.Y. 1943), 2018-04, Vol.154 (5), p.1524-1537.e6

2018

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Epithelial to Mesenchymal Transition Contributes to Drug Resistance in Pancreatic Cancer

Cancer research (Chicago, Ill.), 2009-07, Vol.69 (14), p.5820-5828

2009

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S100P: a novel therapeutic target for cancer

Amino acids, 2011-10, Vol.41 (4), p.893-899

2011

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Mast Cells in Tumor Microenvironment Promotes the In Vivo Growth of Pancreatic Ductal Adenocarcinoma

Clinical cancer research, 2011-11, Vol.17 (22), p.7015-7023

2011

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Cancer-Associated Stromal Fibroblasts Promote Pancreatic Tumor Progression

Cancer research (Chicago, Ill.), 2008-02, Vol.68 (3), p.918-926

2008

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TM4SF1 Promotes Gemcitabine Resistance of Pancreatic Cancer In Vitro and In Vivo

PloS one, 2015-12, Vol.10 (12), p.e0144969

2015

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The Significance of Ras Activity in Pancreatic Cancer Initiation

International journal of biological sciences, 2016-01, Vol.12 (3), p.338-346

2016

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An NF-κB pathway-mediated positive feedback loop amplifies Ras activity to pathological levels in mice

The Journal of clinical investigation, 2012-04, Vol.122 (4), p.1519-1528

2012

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A High-Fat Diet Activates Oncogenic Kras and COX2 to Induce Development of Pancreatic Ductal Adenocarcinoma in Mice

Gastroenterology (New York, N.Y. 1943), 2013-12, Vol.145 (6), p.1449-1458

2013

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S100P-Derived RAGE Antagonistic Peptide Reduces Tumor Growth and Metastasis

Clinical cancer research, 2012-08, Vol.18 (16), p.4356-4364

2012

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The immune system in cancer metastasis: friend or foe?

Journal for Immunotherapy of Cancer, 2017-10, Vol.5 (1), p.79-79, Article 79

2017

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RAGE and RAGE ligands in cancer

Current molecular medicine, 2007-12, Vol.7 (8), p.777

2007

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Lipocalin-2 Promotes Pancreatic Ductal Adenocarcinoma by Regulating Inflammation in the Tumor Microenvironment

Cancer research (Chicago, Ill.), 2017-05, Vol.77 (10), p.2647-2660

2017

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Anterior Gradient 2 Is Expressed and Secreted during the Development of Pancreatic Cancer and Promotes Cancer Cell Survival

Cancer research (Chicago, Ill.), 2008-10, Vol.68 (19), p.7811-7818

2008

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RAGE maintains high levels of NFκB and oncogenic Kras activity in pancreatic cancer

Biochemical and biophysical research communications, 2017-11, Vol.493 (1), p.592-597

2017

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Kras mutation rate precisely orchestrates ductal derived pancreatic intraepithelial neoplasia and pancreatic cancer

Laboratory investigation, 2021-02, Vol.101 (2), p.177-192

2021

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Effect of Cromolyn on S100P Interactions With RAGE and Pancreatic Cancer Growth and Invasion in Mouse Models

JNCI : Journal of the National Cancer Institute, 2006-12, Vol.98 (24), p.1806-1818

2006

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Early elevations of RAS protein level and activity are critical for the development of PDAC in the context of inflammation

Cancer letters, 2024-04, Vol.586, p.216694-216694, Article 216694

2024

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Roles for KRAS in Pancreatic Tumor Development and Progression

Activation of Nuclear Factor-κB in Acinar Cells Increases the Severity of Pancreatitis in Mice

A Novel FoxM1-Caveolin Signaling Pathway Promotes Pancreatic Cancer Invasion and Metastasis

The role of protein synthesis and digestive enzymes in acinar cell injury

Cell surface lactate receptor GPR81 is crucial for cancer cell survival

Dynamic Mast Cell-Stromal Cell Interactions Promote Growth of Pancreatic Cancer

Small interfering RNAs (siRNAs) in cancer therapy: a nano-based approach

Galectin-3 Mediates Tumor Cell–Stroma Interactions by Activating Pancreatic Stellate Cells to Produce Cytokines via Integrin Signaling

Epithelial to Mesenchymal Transition Contributes to Drug Resistance in Pancreatic Cancer

S100P: a novel therapeutic target for cancer

Mast Cells in Tumor Microenvironment Promotes the In Vivo Growth of Pancreatic Ductal Adenocarcinoma

Cancer-Associated Stromal Fibroblasts Promote Pancreatic Tumor Progression

TM4SF1 Promotes Gemcitabine Resistance of Pancreatic Cancer In Vitro and In Vivo

The Significance of Ras Activity in Pancreatic Cancer Initiation

An NF-κB pathway-mediated positive feedback loop amplifies Ras activity to pathological levels in mice

A High-Fat Diet Activates Oncogenic Kras and COX2 to Induce Development of Pancreatic Ductal Adenocarcinoma in Mice

S100P-Derived RAGE Antagonistic Peptide Reduces Tumor Growth and Metastasis

The immune system in cancer metastasis: friend or foe?

RAGE and RAGE ligands in cancer

Lipocalin-2 Promotes Pancreatic Ductal Adenocarcinoma by Regulating Inflammation in the Tumor Microenvironment

Anterior Gradient 2 Is Expressed and Secreted during the Development of Pancreatic Cancer and Promotes Cancer Cell Survival

RAGE maintains high levels of NFκB and oncogenic Kras activity in pancreatic cancer

Kras mutation rate precisely orchestrates ductal derived pancreatic intraepithelial neoplasia and pancreatic cancer

Effect of Cromolyn on S100P Interactions With RAGE and Pancreatic Cancer Growth and Invasion in Mouse Models

Early elevations of RAS protein level and activity are critical for the development of PDAC in the context of inflammation

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