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Hyperglycemia-induced oxidative stress in diabetic complications

Histochemistry and cell biology, 2004-10, Vol.122 (4), p.333-338

2004

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Rescue of neural tube defects in Pax-3-deficient embryos by p53 loss of function: implications for Pax-3- dependent development and tumorigenesis

Genes & development, 2002-03, Vol.16 (6), p.676-680

2002

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A core outcome set for the treatment of pregnant women with pregestational diabetes: an international consensus study

BJOG : an international journal of obstetrics and gynaecology, 2021-10, Vol.128 (11), p.1855-1868

2021

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Current perspectives on the causes of neural tube defects resulting from diabetic pregnancy

American journal of medical genetics. Part C, Seminars in medical genetics, 2005-05, Vol.135C (1), p.77-87

Loeken, Mary R.

2005

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Oxidant regulation of gene expression and neural tube development: Insights gained from diabetic pregnancy on molecular causes of neural tube defects

Diabetologia, 2003-04, Vol.46 (4), p.538-545

2003

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Hypoxic stress in diabetic pregnancy contributes to impaired embryo gene expression and defective development by inducing oxidative stress

American journal of physiology: endocrinology and metabolism, 2005-10, Vol.289 (4), p.E591-E599

2005

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Evidence that elevated glucose causes altered gene expression, apoptosis, and neural tube defects in a mouse model of diabetic pregnancy

Diabetes (New York, N.Y.), 1999-12, Vol.48 (12), p.2454

1999

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Activation of the hexosamine pathway causes oxidative stress and abnormal embryo gene expression: Involvement in diabetic teratogenesis

Birth defects research. A Clinical and molecular teratology, 2004-08, Vol.70 (8), p.519-527

2004

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Polymorphic Susceptibility to the Molecular Causes of Neural Tube Defects During Diabetic Embryopathy

Diabetes (New York, N.Y.), 2002-09, Vol.51 (9), p.2871-2874

2002

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Diacylglycerol Production and Protein Kinase C Activity Are Increased in a Mouse Model of Diabetic Embryopathy

Diabetes (New York, N.Y.), 2002-09, Vol.51 (9), p.2804-2810

2002

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Maternal Diabetes Increases the Risk of Caudal Regression Caused by Retinoic Acid

Diabetes (New York, N.Y.), 2002-09, Vol.51 (9), p.2811-2816

2002

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Stimulation of endothelin-1 gene expression by insulin in endothelial cells

The Journal of biological chemistry, 1991-12, Vol.266 (34), p.23251-23256

1991

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Identification of a New Binding Motif for the Paired Domain of Pax-3 and Unusual Characteristics of Spacing of Bipartite Recognition Elements on Binding and Transcription Activation

The Journal of biological chemistry, 1998-07, Vol.273 (30), p.19153-19159

1998

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Identification of Dep-1, a new gene regulated by the transcription factor Pax-3, as a marker for altered embryonic gene expression during diabetic pregnancy

Diabetes (New York, N.Y.), 1998-11, Vol.47 (11), p.1803

1998

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Genotoxicity and diabetic embryopathy: impaired expression of developmental control genes as a cause of defective morphogenesis

Seminars in reproductive endocrinology, 1999-01, Vol.17 (2), p.153-165

1999

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The adenovirus EIIA enhancer. Analysis of regulatory sequences and changes in binding activity of ATF and EIIF following adenovirus infection

The Journal of biological chemistry, 1989-04, Vol.264 (11), p.6572-6579

1989

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Advances in Understanding the Molecular Causes of Diabetes-Induced Birth Defects

Journal of the Society for Gynecologic Investigation, 2006-01, Vol.13 (1), p.2-10

Loeken, Mary R.

2006

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Reduced expression of pax-3 is associated with overexpression of cdc46 in the mouse embryo

Development genes and evolution, 1998-05, Vol.208 (3), p.128-134

1998

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Expression of the gene encoding glycogen phosphorylase is elevated in diabetic rat skeletal muscle and is regulated by insulin and cyclic AMP

Diabetologia, 1996-02, Vol.39 (2), p.183-189

1996

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Simian virus 40 small t antigen trans activates the adenovirus E2A promoter by using mechanisms distinct from those used by adenovirus E1A

Journal of Virology, 1992-04, Vol.66 (4), p.2551-2555

Loeken, M R

1992

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Analysis of maternal IgG subpopulations which are transported into the chicken oocyte

Immunology, 1983-05, Vol.49 (1), p.21-28

1983

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Graft-versus-leukemia effect in acute lymphoblastic leukemia: the importance of tumor burden and early detection

Leukemia, 2004-06, Vol.18 (6), p.1156-1158

2004

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Heterogeneity of expression and secretion of native and mutant [AspB10]insulin in AtT20 cells

Molecular endocrinology (Baltimore, Md.), 1991-03, Vol.5 (3), p.319-352

1991

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Stimulatory effect of luteinizing hormone upon relaxin secretion by cultured porcine preovulatory granulosa cells

Endocrinology (Philadelphia), 1983-02, Vol.112 (2), p.769-771

1983

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Intraovarian control of granulosa cell luteinization

Endocrinologia experimentalis, 1982-11, Vol.16 (3-4), p.301

1982

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Hyperglycemia-induced oxidative stress in diabetic complications

Rescue of neural tube defects in Pax-3-deficient embryos by p53 loss of function: implications for Pax-3- dependent development and tumorigenesis

A core outcome set for the treatment of pregnant women with pregestational diabetes: an international consensus study

Current perspectives on the causes of neural tube defects resulting from diabetic pregnancy

Oxidant regulation of gene expression and neural tube development: Insights gained from diabetic pregnancy on molecular causes of neural tube defects

Hypoxic stress in diabetic pregnancy contributes to impaired embryo gene expression and defective development by inducing oxidative stress

Evidence that elevated glucose causes altered gene expression, apoptosis, and neural tube defects in a mouse model of diabetic pregnancy

Activation of the hexosamine pathway causes oxidative stress and abnormal embryo gene expression: Involvement in diabetic teratogenesis

Polymorphic Susceptibility to the Molecular Causes of Neural Tube Defects During Diabetic Embryopathy

Diacylglycerol Production and Protein Kinase C Activity Are Increased in a Mouse Model of Diabetic Embryopathy

Maternal Diabetes Increases the Risk of Caudal Regression Caused by Retinoic Acid

Stimulation of endothelin-1 gene expression by insulin in endothelial cells

Identification of a New Binding Motif for the Paired Domain of Pax-3 and Unusual Characteristics of Spacing of Bipartite Recognition Elements on Binding and Transcription Activation

Identification of Dep-1, a new gene regulated by the transcription factor Pax-3, as a marker for altered embryonic gene expression during diabetic pregnancy

Genotoxicity and diabetic embryopathy: impaired expression of developmental control genes as a cause of defective morphogenesis

The adenovirus EIIA enhancer. Analysis of regulatory sequences and changes in binding activity of ATF and EIIF following adenovirus infection

Advances in Understanding the Molecular Causes of Diabetes-Induced Birth Defects

Reduced expression of pax-3 is associated with overexpression of cdc46 in the mouse embryo

Expression of the gene encoding glycogen phosphorylase is elevated in diabetic rat skeletal muscle and is regulated by insulin and cyclic AMP

Simian virus 40 small t antigen trans activates the adenovirus E2A promoter by using mechanisms distinct from those used by adenovirus E1A

Analysis of maternal IgG subpopulations which are transported into the chicken oocyte

Graft-versus-leukemia effect in acute lymphoblastic leukemia: the importance of tumor burden and early detection

Heterogeneity of expression and secretion of native and mutant [AspB10]insulin in AtT20 cells

Stimulatory effect of luteinizing hormone upon relaxin secretion by cultured porcine preovulatory granulosa cells

Intraovarian control of granulosa cell luteinization

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