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AMP-activated protein kinase mediates effects of oxidative stress on embryo gene expression in a mouse model of diabetic embryopathy

Diabetologia, 2012-01, Vol.55 (1), p.245-254

2012

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Oxidant regulation of gene expression and neural tube development: Insights gained from diabetic pregnancy on molecular causes of neural tube defects

Diabetologia, 2003-04, Vol.46 (4), p.538-545

2003

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A core outcome set for the treatment of pregnant women with pregestational diabetes: an international consensus study

BJOG : an international journal of obstetrics and gynaecology, 2021-10, Vol.128 (11), p.1855-1868

2021

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Expression of the gene encoding the high-Km glucose transporter 2 by the early postimplantation mouse embryo is essential for neural tube defects associated with diabetic embryopathy

Diabetologia, 2007-03, Vol.50 (3), p.682-689

2007

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Evidence that elevated glucose causes altered gene expression, apoptosis, and neural tube defects in a mouse model of diabetic pregnancy

Diabetes (New York, N.Y.), 1999-12, Vol.48 (12), p.2454-2462

1999

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Neural tube defects in embryos of diabetic mice: role of the Pax-3 gene and apoptosis

Diabetes (New York, N.Y.), 1997-07, Vol.46 (7), p.1189-1197

1997

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Rescue of neural tube defects in Pax-3-deficient embryos by p53 loss of function: implications for Pax-3- dependent development and tumorigenesis

Genes & development, 2002-03, Vol.16 (6), p.676-680

2002

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Expression of the gene encoding the high-K m glucose transporter 2 by the early postimplantation mouse embryo is essential for neural tube defects associated with diabetic embryopathy

Diabetologia, 2007-03, Vol.50 (3), p.682-689

2007

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Stimulation of endothelin-1 gene expression by insulin in endothelial cells

The Journal of biological chemistry, 1991-12, Vol.266 (34), p.23251-23256

1991

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Free radicals and birth defects

The journal of maternal-fetal & neonatal medicine, 2004, Vol.15 (1), p.6-14

Loeken, M. R.

2004

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Neural tube defects in embryos of diabetic mice: role of the Pax-3 gene and apoptosis

Diabetes (New York, N.Y.), 1997-07, Vol.46 (7), p.1189-1197

1997

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Activation of the hexosamine pathway causes oxidative stress and abnormal embryo gene expression: Involvement in diabetic teratogenesis

Birth defects research. A Clinical and molecular teratology, 2004-08, Vol.70 (8), p.519-527

2004

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The adenovirus EIIA enhancer. Analysis of regulatory sequences and changes in binding activity of ATF and EIIF following adenovirus infection

The Journal of biological chemistry, 1989-04, Vol.264 (11), p.6572-6579

1989

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Expression of the gene encoding glycogen phosphorylase is elevated in diabetic rat skeletal muscle and is regulated by insulin and cyclic AMP

Diabetologia, 1996-02, Vol.39 (2), p.183-189

1996

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Identification of Dep-1, a new gene regulated by the transcription factor Pax-3, as a marker for altered embryonic gene expression during diabetic pregnancy

Diabetes (New York, N.Y.), 1998-11, Vol.47 (11), p.1803-1805

1998

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Hyperglycemia-induced oxidative stress in diabetic complications

Histochemistry and cell biology, 2004-10, Vol.122 (4), p.333-338

2004

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Multiple, distinct trans-activation functions are encoded by the simian virus 40 large T and small t antigens, only some of which require the 82-residue amino-terminal common domain

Journal of Virology, 1993-12, Vol.67 (12), p.7684-7689

LOEKEN, M. R

1993

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Reduced expression of pax-3 is associated with overexpression of cdc46 in the mouse embryo

Development genes and evolution, 1998-05, Vol.208 (3), p.128-134

1998

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Simian virus 40 small t antigen trans activates the adenovirus E2A promoter by using mechanisms distinct from those used by adenovirus E1A

Journal of Virology, 1992-04, Vol.66 (4), p.2551-2555

Loeken, M R

1992

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Involvement of simian virus 40 (SV40) small t antigen in trans activation of SV40 early and late promoters

Journal of Virology, 1992-03, Vol.66 (3), p.1489-1494

1992

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Current perspectives on the causes of neural tube defects resulting from diabetic pregnancy

American journal of medical genetics. Part C, Seminars in medical genetics, 2005-05, Vol.135C (1), p.77-87

Loeken, Mary R.

2005

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Identification of a New Binding Motif for the Paired Domain of Pax-3 and Unusual Characteristics of Spacing of Bipartite Recognition Elements on Binding and Transcription Activation

The Journal of biological chemistry, 1998-07, Vol.273 (30), p.19153-19159

1998

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Hypoxic stress in diabetic pregnancy contributes to impaired embryo gene expression and defective development by inducing oxidative stress

American journal of physiology: endocrinology and metabolism, 2005-10, Vol.289 (4), p.E591-E599

2005

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Stimulation of the adenovirus E2 promoter by simian virus 40 T antigen or E1A occurs by different mechanisms

Molecular and Cellular Biology, 1986-06, Vol.6 (6), p.2020-2026

1986

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Genotoxicity and diabetic embryopathy: impaired expression of developmental control genes as a cause of defective morphogenesis

Seminars in reproductive endocrinology, 1999-01, Vol.17 (2), p.153-165

1999

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AMP-activated protein kinase mediates effects of oxidative stress on embryo gene expression in a mouse model of diabetic embryopathy

Oxidant regulation of gene expression and neural tube development: Insights gained from diabetic pregnancy on molecular causes of neural tube defects

A core outcome set for the treatment of pregnant women with pregestational diabetes: an international consensus study

Expression of the gene encoding the high-Km glucose transporter 2 by the early postimplantation mouse embryo is essential for neural tube defects associated with diabetic embryopathy

Evidence that elevated glucose causes altered gene expression, apoptosis, and neural tube defects in a mouse model of diabetic pregnancy

Neural tube defects in embryos of diabetic mice: role of the Pax-3 gene and apoptosis

Rescue of neural tube defects in Pax-3-deficient embryos by p53 loss of function: implications for Pax-3- dependent development and tumorigenesis

Expression of the gene encoding the high-K m glucose transporter 2 by the early postimplantation mouse embryo is essential for neural tube defects associated with diabetic embryopathy

Stimulation of endothelin-1 gene expression by insulin in endothelial cells

Free radicals and birth defects

Neural tube defects in embryos of diabetic mice: role of the Pax-3 gene and apoptosis

Activation of the hexosamine pathway causes oxidative stress and abnormal embryo gene expression: Involvement in diabetic teratogenesis

The adenovirus EIIA enhancer. Analysis of regulatory sequences and changes in binding activity of ATF and EIIF following adenovirus infection

Expression of the gene encoding glycogen phosphorylase is elevated in diabetic rat skeletal muscle and is regulated by insulin and cyclic AMP

Identification of Dep-1, a new gene regulated by the transcription factor Pax-3, as a marker for altered embryonic gene expression during diabetic pregnancy

Hyperglycemia-induced oxidative stress in diabetic complications

Multiple, distinct trans-activation functions are encoded by the simian virus 40 large T and small t antigens, only some of which require the 82-residue amino-terminal common domain

Reduced expression of pax-3 is associated with overexpression of cdc46 in the mouse embryo

Simian virus 40 small t antigen trans activates the adenovirus E2A promoter by using mechanisms distinct from those used by adenovirus E1A

Involvement of simian virus 40 (SV40) small t antigen in trans activation of SV40 early and late promoters

Current perspectives on the causes of neural tube defects resulting from diabetic pregnancy

Identification of a New Binding Motif for the Paired Domain of Pax-3 and Unusual Characteristics of Spacing of Bipartite Recognition Elements on Binding and Transcription Activation

Hypoxic stress in diabetic pregnancy contributes to impaired embryo gene expression and defective development by inducing oxidative stress

Stimulation of the adenovirus E2 promoter by simian virus 40 T antigen or E1A occurs by different mechanisms

Genotoxicity and diabetic embryopathy: impaired expression of developmental control genes as a cause of defective morphogenesis

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