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Microglia in the TBI brain: The good, the bad, and the dysregulated

Experimental neurology, 2016-01, Vol.275 (3), p.316-327

2016

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Neuroinflammation after traumatic brain injury: Opportunities for therapeutic intervention

Brain, behavior, and immunity, 2012-11, Vol.26 (8), p.1191-1201

2012

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Neuroprotection for traumatic brain injury: translational challenges and emerging therapeutic strategies

Trends in pharmacological sciences (Regular ed.), 2010-12, Vol.31 (12), p.596-604

2010

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The far-reaching scope of neuroinflammation after traumatic brain injury

Nature reviews. Neurology, 2017-03, Vol.13 (3), p.171

2017

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Progressive inflammation‐mediated neurodegeneration after traumatic brain or spinal cord injury

British journal of pharmacology, 2016-02, Vol.173 (4), p.681-691

2016

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Understanding microglial responses in large animal models of traumatic brain injury: an underutilized resource for preclinical and translational research

Journal of neuroinflammation, 2023-03, Vol.20 (1), p.67-67, Article 67

2023

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Inhibition of autophagy in microglia and macrophages exacerbates innate immune responses and worsens brain injury outcomes

Autophagy, 2023-07, Vol.19 (7), p.2026-2044

2023

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NOX2 drives M1-like microglial/macrophage activation and neurodegeneration following experimental traumatic brain injury

Brain, behavior, and immunity, 2016-11, Vol.58, p.291-309

2016

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Acute drivers of neuroinflammation in traumatic brain injury

Neural regeneration research, 2019-09, Vol.14 (9), p.1481-1489

2019

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Delayed mGluR5 activation limits neuroinflammation and neurodegeneration after traumatic brain injury

Journal of neuroinflammation, 2012-02, Vol.9 (1), p.43-43, Article 43

2012

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Acute colitis during chronic experimental traumatic brain injury in mice induces dysautonomia and persistent extraintestinal, systemic, and CNS inflammation with exacerbated neurological deficits

Journal of neuroinflammation, 2021-01, Vol.18 (1), p.24-31, Article 24

2021

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Traumatic Brain Injury Induces cGAS Activation and Type I Interferon Signaling in Aged Mice

Frontiers in immunology, 2021-08, Vol.12, p.710608-710608

2021

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Inflammatory response of microglia to prions is controlled by sialylation of PrPSc

Scientific reports, 2018-07, Vol.8 (1), p.1-15, Article 11326

2018

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Late exercise reduces neuroinflammation and cognitive dysfunction after traumatic brain injury

Neurobiology of disease, 2013-06, Vol.54, p.252-263

2013

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Targeting chronic and evolving neuroinflammation following traumatic brain injury to improve long-term outcomes: insights from microglial-depletion models

Neural regeneration research, 2021-05, Vol.16 (5), p.976-977

2021

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Amyloid precursor protein secretases as therapeutic targets for traumatic brain injury

Nature medicine, 2009-04, Vol.15 (4), p.377-379

2009

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Brain-gut axis dysfunction in the pathogenesis of traumatic brain injury

The Journal of clinical investigation, 2021-06, Vol.131 (12)

2021

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Sexual dimorphism in the inflammatory response to traumatic brain injury

Glia, 2017-09, Vol.65 (9), p.1423-1438

2017

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Activation of mGluR5 and inhibition of NADPH oxidase improves functional recovery after traumatic brain injury

Journal of neurotrauma, 2013-03, Vol.30 (5), p.403

2013

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Bi-directional neuro-immune dysfunction after chronic experimental brain injury

Journal of neuroinflammation, 2024-04, Vol.21 (1), p.83-20, Article 83

2024

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MAnGLed astrocytes in traumatic brain injury: astrocytic 2-AG metabolism as a new therapeutic target

Brain (London, England : 1878), 2022-03, Vol.145 (1), p.7-10

Loane, David J

2022

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S100B Inhibition Reduces Behavioral and Pathologic Changes in Experimental Traumatic Brain Injury

Journal of cerebral blood flow and metabolism, 2015-12, Vol.35 (12), p.2010-2020

2015

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Neuroinflammation is a player in coma, but in which role?

Brain (London, England : 1878), 2024-04, Vol.147 (4), p.1121-1123

2024

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Metabotropic glutamate receptor 5 activation inhibits microglial associated inflammation and neurotoxicity

Glia, 2009-04, Vol.57 (5), p.550-560

2009

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Metabotropic Glutamate Receptors as Targets for Multipotential Treatment of Neurological Disorders

Neurotherapeutics, 2009, Vol.6 (1), p.94-107

2009

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Microglia in the TBI brain: The good, the bad, and the dysregulated

Neuroinflammation after traumatic brain injury: Opportunities for therapeutic intervention

Neuroprotection for traumatic brain injury: translational challenges and emerging therapeutic strategies

The far-reaching scope of neuroinflammation after traumatic brain injury

Progressive inflammation‐mediated neurodegeneration after traumatic brain or spinal cord injury

Understanding microglial responses in large animal models of traumatic brain injury: an underutilized resource for preclinical and translational research

Inhibition of autophagy in microglia and macrophages exacerbates innate immune responses and worsens brain injury outcomes

NOX2 drives M1-like microglial/macrophage activation and neurodegeneration following experimental traumatic brain injury

Acute drivers of neuroinflammation in traumatic brain injury

Delayed mGluR5 activation limits neuroinflammation and neurodegeneration after traumatic brain injury

Acute colitis during chronic experimental traumatic brain injury in mice induces dysautonomia and persistent extraintestinal, systemic, and CNS inflammation with exacerbated neurological deficits

Traumatic Brain Injury Induces cGAS Activation and Type I Interferon Signaling in Aged Mice

Inflammatory response of microglia to prions is controlled by sialylation of PrPSc

Late exercise reduces neuroinflammation and cognitive dysfunction after traumatic brain injury

Targeting chronic and evolving neuroinflammation following traumatic brain injury to improve long-term outcomes: insights from microglial-depletion models

Amyloid precursor protein secretases as therapeutic targets for traumatic brain injury

Brain-gut axis dysfunction in the pathogenesis of traumatic brain injury

Sexual dimorphism in the inflammatory response to traumatic brain injury

Activation of mGluR5 and inhibition of NADPH oxidase improves functional recovery after traumatic brain injury

Bi-directional neuro-immune dysfunction after chronic experimental brain injury

MAnGLed astrocytes in traumatic brain injury: astrocytic 2-AG metabolism as a new therapeutic target

S100B Inhibition Reduces Behavioral and Pathologic Changes in Experimental Traumatic Brain Injury

Neuroinflammation is a player in coma, but in which role?

Metabotropic glutamate receptor 5 activation inhibits microglial associated inflammation and neurotoxicity

Metabotropic Glutamate Receptors as Targets for Multipotential Treatment of Neurological Disorders

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