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A Process of Resection-Dependent Nonhomologous End Joining Involving the Goddess Artemis

Trends in biochemical sciences (Amsterdam. Regular ed.), 2017-09, Vol.42 (9), p.690-701

2017

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One end to rule them all: Non-homologous end-joining and homologous recombination at DNA double-strand breaks

British journal of radiology, 2020-11, Vol.93 (1115), p.20191054-20191054

2020

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ATRX Promotes DNA Repair Synthesis and Sister Chromatid Exchange during Homologous Recombination

Molecular cell, 2018-07, Vol.71 (1), p.11-24.e7

2018

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Homologous Recombination Subpathways: A Tangle to Resolve

Frontiers in genetics, 2021-08, Vol.12, p.723847-723847

2021

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The impact of a negligent G2/M checkpoint on genomic instability and cancer induction

Nature reviews. Cancer, 2007-11, Vol.7 (11), p.861-869

2007

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Evidence for a Lack of DNA Double-Strand Break Repair in Human Cells Exposed to Very Low X-Ray Doses

Proceedings of the National Academy of Sciences - PNAS, 2003-04, Vol.100 (9), p.5057-5062

2003

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Hazards of human spaceflight

Science (American Association for the Advancement of Science), 2019-04, Vol.364 (6436), p.127-128

2019

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How cancer cells hijack DNA double-strand break repair pathways to gain genomic instability

Biochemical journal, 2015-10, Vol.471 (1), p.1-11

2015

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Reparaturwege in Krebszellen gezielt ausschalten

Biospektrum, 2022-03, Vol.28 (2), p.159-161

2022

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Factors determining DNA double-strand break repair pathway choice in G2 phase

The EMBO journal, 2011-03, Vol.30 (6), p.1079-1092

2011

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ATM and DNA-PK function redundantly to phosphorylate H2AX after exposure to ionizing radiation

Cancer research (Chicago, Ill.), 2004-04, Vol.64 (7), p.2390-2396

2004

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DNA Double-Strand Break Resection Occurs during Non-homologous End Joining in G1 but Is Distinct from Resection during Homologous Recombination

Molecular cell, 2017-02, Vol.65 (4), p.671-684.e5

2017

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Understanding the limitations of radiation-induced cell cycle checkpoints

Critical reviews in biochemistry and molecular biology, 2011-08, Vol.46 (4), p.271-283

2011

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DNA double-strand breaks in heterochromatin elicit fast repair protein recruitment, histone H2AX phosphorylation and relocation to euchromatin

Nucleic acids research, 2011-08, Vol.39 (15), p.6489-6499

2011

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POLθ-mediated end joining is restricted by RAD52 and BRCA2 until the onset of mitosis

Nature cell biology, 2021-10, Vol.23 (10), p.1095-1104

2021

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γH2AX foci analysis for monitoring DNA double-strand break repair: Strengths, limitations and optimization

Cell cycle (Georgetown, Tex.), 2010-02, Vol.9 (4), p.662-669

2010

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ATM release at resected double-strand breaks provides heterochromatin reconstitution to facilitate homologous recombination

PLoS genetics, 2013-08, Vol.9 (8), p.e1003667-e1003667

2013

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The role of homologous recombination in radiation-induced double-strand break repair

Radiotherapy and oncology, 2011-10, Vol.101 (1), p.7-12

2011

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Nek1 Regulates Rad54 to Orchestrate Homologous Recombination and Replication Fork Stability

Molecular cell, 2016-06, Vol.62 (6), p.903-917

2016

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53BP1-dependent robust localized KAP-1 phosphorylation is essential for heterochromatic DNA double-strand break repair

Nature cell biology, 2010-02, Vol.12 (2), p.177-184

2010

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CtIP and MRN promote non-homologous end-joining of etoposide-induced DNA double-strand breaks in G1

Nucleic acids research, 2011-03, Vol.39 (6), p.2144-2152

2011

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DNA Double-Strand Break Repair of Blood Lymphocytes and Normal Tissues Analysed in a Preclinical Mouse Model: Implications for Radiosensitivity Testing

Clinical cancer research, 2008-10, Vol.14 (20), p.6546-6555

2008

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Role of ATM and the Damage Response Mediator Proteins 53BP1 and MDC1 in the Maintenance of G2/M Checkpoint Arrest

Molecular and Cellular Biology, 2010-07, Vol.30 (13), p.3371-3383

2010

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Chromosome Breakage after G2 Checkpoint Release

The Journal of cell biology, 2007-03, Vol.176 (6), p.749-755

2007

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Polo-like kinase 3 regulates CtIP during DNA double-strand break repair in G1

The Journal of cell biology, 2014-09, Vol.206 (7), p.877-894

2014

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A Process of Resection-Dependent Nonhomologous End Joining Involving the Goddess Artemis

One end to rule them all: Non-homologous end-joining and homologous recombination at DNA double-strand breaks

ATRX Promotes DNA Repair Synthesis and Sister Chromatid Exchange during Homologous Recombination

Homologous Recombination Subpathways: A Tangle to Resolve

The impact of a negligent G2/M checkpoint on genomic instability and cancer induction

Evidence for a Lack of DNA Double-Strand Break Repair in Human Cells Exposed to Very Low X-Ray Doses

Hazards of human spaceflight

How cancer cells hijack DNA double-strand break repair pathways to gain genomic instability

Reparaturwege in Krebszellen gezielt ausschalten

Factors determining DNA double-strand break repair pathway choice in G2 phase

ATM and DNA-PK function redundantly to phosphorylate H2AX after exposure to ionizing radiation

DNA Double-Strand Break Resection Occurs during Non-homologous End Joining in G1 but Is Distinct from Resection during Homologous Recombination

Understanding the limitations of radiation-induced cell cycle checkpoints

DNA double-strand breaks in heterochromatin elicit fast repair protein recruitment, histone H2AX phosphorylation and relocation to euchromatin

POLθ-mediated end joining is restricted by RAD52 and BRCA2 until the onset of mitosis

γH2AX foci analysis for monitoring DNA double-strand break repair: Strengths, limitations and optimization

ATM release at resected double-strand breaks provides heterochromatin reconstitution to facilitate homologous recombination

The role of homologous recombination in radiation-induced double-strand break repair

Nek1 Regulates Rad54 to Orchestrate Homologous Recombination and Replication Fork Stability

53BP1-dependent robust localized KAP-1 phosphorylation is essential for heterochromatic DNA double-strand break repair

CtIP and MRN promote non-homologous end-joining of etoposide-induced DNA double-strand breaks in G1

DNA Double-Strand Break Repair of Blood Lymphocytes and Normal Tissues Analysed in a Preclinical Mouse Model: Implications for Radiosensitivity Testing

Role of ATM and the Damage Response Mediator Proteins 53BP1 and MDC1 in the Maintenance of G2/M Checkpoint Arrest

Chromosome Breakage after G2 Checkpoint Release

Polo-like kinase 3 regulates CtIP during DNA double-strand break repair in G1

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