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Extended optimal preventive replacement policies with random working cycle

Reliability engineering & system safety, 2019-08, Vol.188, p.398-415

2019

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Increased urine and serum nerve growth factor levels in interstitial cystitis suggest chronic inflammation is involved in the pathogenesis of disease

PloS one, 2012-09, Vol.7 (9), p.e44687-e44687

2012

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The philosophical views of national innovation system: The LED industry in Taiwan

Asia Pacific management review, 2019-12, Vol.24 (4), p.291-297

Liu, Tzu-Hsin

2019

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Increased pro-inflammatory cytokines, C-reactive protein and nerve growth factor expressions in serum of patients with interstitial cystitis/bladder pain syndrome

PloS one, 2013-10, Vol.8 (10), p.e76779-e76779

2013

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Increased Cell Apoptosis of Urothelium Mediated by Inflammation in Interstitial Cystitis/Painful Bladder Syndrome

Urology (Ridgewood, N.J.), 2012-02, Vol.79 (2), p.484.e7-484.e13

2012

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Urinary nerve growth factor but not prostaglandin E2 increases in patients with interstitial cystitis/bladder pain syndrome and detrusor overactivity

BJU international, 2010-12, Vol.106 (11), p.1681-1685

2010

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Isoaaptamine increases ROS levels causing autophagy and mitochondria-mediated apoptosis in glioblastoma multiforme cells

Biomedicine & pharmacotherapy, 2023-04, Vol.160, p.114359-114359, Article 114359

2023

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Urinary nerve growth factor level is increased in patients with interstitial cystitis/bladder pain syndrome and decreased in responders to treatment

BJU international, 2009-11, Vol.104 (10), p.1476-1481

2009

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Intra-Articular Lactate Dehydrogenase A Inhibitor Oxamate Reduces Experimental Osteoarthritis and Nociception in Rats via Possible Alteration of Glycolysis-Related Protein Expression in Cartilage Tissue

International journal of molecular sciences, 2023-06, Vol.24 (13), p.10770

2023

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Intravesical Botulinum Toxin A Injections Plus Hydrodistension Can Reduce Nerve Growth Factor Production and Control Bladder Pain in Interstitial Cystitis

Urology (Ridgewood, N.J.), 2007-09, Vol.70 (3), p.463-468

2007

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Increased serum nerve growth factor levels in patients with overactive bladder syndrome refractory to antimuscarinic therapy

Neurourology and urodynamics, 2011-11, Vol.30 (8), p.1525-1529

2011

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Decrease of urinary nerve growth factor but not brain-derived neurotrophic factor in patients with interstitial cystitis/bladder pain syndrome treated with hyaluronic acid

PloS one, 2014-03, Vol.9 (3), p.e91609

2014

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Immunohistochemical evidence suggests repeated intravesical application of botulinum toxin A injections may improve treatment efficacy of interstitial cystitis/bladder pain syndrome

BJU international, 2013-04, Vol.111 (4), p.638-646

2013

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Role of PVAT in obesity-related cardiovascular disease through the buffering activity of ATF3

iScience, 2022-12, Vol.25 (12), p.105631-105631, Article 105631

2022

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Baicalein Exerts Therapeutic Effects against Endotoxin-Induced Depression-like Behavior in Mice by Decreasing Inflammatory Cytokines and Increasing Brain-Derived Neurotrophic Factor Levels

Antioxidants, 2022-05, Vol.11 (5), p.947

2022

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Decrease of urinary nerve growth factor levels after antimuscarinic therapy in patients with overactive bladder

BJU international, 2009-06, Vol.103 (12), p.1668-1672

2009

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Increased serum adipokines implicate chronic inflammation in the pathogenesis of overactive bladder syndrome refractory to antimuscarinic therapy

PloS one, 2013-10, Vol.8 (10), p.e76706

2013

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Expressions of urothelial functional proteins in idiopathic detrusor overactivity patients refractory to antimuscarinic therapy with different urodynamic characteristics

Neurourology and urodynamics, 2017-06, Vol.36 (5), p.1313-1319

2017

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Urothelial dysfunction and chronic inflammation in patients with spinal cord injuries at different levels and correlation with urodynamic findings

Neurourology and urodynamics, 2015-11, Vol.34 (8), p.757-762

2015

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Multiserver Retrial Queue with Two-Way Communication and Synchronous Working Vacation

Mathematics (Basel), 2024-04, Vol.12 (8), p.1163

2024

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Elevation of serum c-reactive protein in patients with OAB and IC/BPS implies chronic inflammation in the urinary bladder

Neurourology and urodynamics, 2011-03, Vol.30 (3), p.417-420

2011

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How Trust Pursuing Businesses Play in an Asymmetric Power Network?

IEEE transactions on engineering management, 2020-02, Vol.67 (1), p.18-29

Liu, Tzu-Hsin

2020

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Retrial queues with starting failure and service interruption

IET communications, 2018-07, Vol.12 (12), p.1431-1437

2018

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Urinary nerve growth factor in women with overactive bladder syndrome

BJU international, 2011-03, Vol.107 (5), p.799-803

2011

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Chondroprotective Effects of a Histone Deacetylase Inhibitor, Panobinostat, on Pain Behavior and Cartilage Degradation in Anterior Cruciate Ligament Transection-Induced Experimental Osteoarthritic Rats

International journal of molecular sciences, 2021-07, Vol.22 (14), p.7290

2021

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Extended optimal preventive replacement policies with random working cycle

Increased urine and serum nerve growth factor levels in interstitial cystitis suggest chronic inflammation is involved in the pathogenesis of disease

The philosophical views of national innovation system: The LED industry in Taiwan

Increased pro-inflammatory cytokines, C-reactive protein and nerve growth factor expressions in serum of patients with interstitial cystitis/bladder pain syndrome

Increased Cell Apoptosis of Urothelium Mediated by Inflammation in Interstitial Cystitis/Painful Bladder Syndrome

Urinary nerve growth factor but not prostaglandin E2 increases in patients with interstitial cystitis/bladder pain syndrome and detrusor overactivity

Isoaaptamine increases ROS levels causing autophagy and mitochondria-mediated apoptosis in glioblastoma multiforme cells

Urinary nerve growth factor level is increased in patients with interstitial cystitis/bladder pain syndrome and decreased in responders to treatment

Intra-Articular Lactate Dehydrogenase A Inhibitor Oxamate Reduces Experimental Osteoarthritis and Nociception in Rats via Possible Alteration of Glycolysis-Related Protein Expression in Cartilage Tissue

Intravesical Botulinum Toxin A Injections Plus Hydrodistension Can Reduce Nerve Growth Factor Production and Control Bladder Pain in Interstitial Cystitis

Increased serum nerve growth factor levels in patients with overactive bladder syndrome refractory to antimuscarinic therapy

Decrease of urinary nerve growth factor but not brain-derived neurotrophic factor in patients with interstitial cystitis/bladder pain syndrome treated with hyaluronic acid

Immunohistochemical evidence suggests repeated intravesical application of botulinum toxin A injections may improve treatment efficacy of interstitial cystitis/bladder pain syndrome

Role of PVAT in obesity-related cardiovascular disease through the buffering activity of ATF3

Baicalein Exerts Therapeutic Effects against Endotoxin-Induced Depression-like Behavior in Mice by Decreasing Inflammatory Cytokines and Increasing Brain-Derived Neurotrophic Factor Levels

Decrease of urinary nerve growth factor levels after antimuscarinic therapy in patients with overactive bladder

Increased serum adipokines implicate chronic inflammation in the pathogenesis of overactive bladder syndrome refractory to antimuscarinic therapy

Expressions of urothelial functional proteins in idiopathic detrusor overactivity patients refractory to antimuscarinic therapy with different urodynamic characteristics

Urothelial dysfunction and chronic inflammation in patients with spinal cord injuries at different levels and correlation with urodynamic findings

Multiserver Retrial Queue with Two-Way Communication and Synchronous Working Vacation

Elevation of serum c-reactive protein in patients with OAB and IC/BPS implies chronic inflammation in the urinary bladder

How Trust Pursuing Businesses Play in an Asymmetric Power Network?

Retrial queues with starting failure and service interruption

Urinary nerve growth factor in women with overactive bladder syndrome

Chondroprotective Effects of a Histone Deacetylase Inhibitor, Panobinostat, on Pain Behavior and Cartilage Degradation in Anterior Cruciate Ligament Transection-Induced Experimental Osteoarthritic Rats

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