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Prognostic Risk Assessment and Prediction of Radiotherapy Benefit for Women with Ductal Carcinoma In Situ (DCIS) of the Breast, in a Randomized Clinical Trial (SweDCIS)

Cancers, 2021-12, Vol.13 (23), p.6103

2021

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Nitric Oxide-Induced Cellular Stress and p53 Activation in Chronic Inflammation

Proceedings of the National Academy of Sciences - PNAS, 2003-01, Vol.100 (1), p.143-148

2003

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BLM helicase-dependent transport of p53 to sites of stalled DNA replication forks modulates homologous recombination

The EMBO journal, 2003-03, Vol.22 (5), p.1210-1222

2003

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Genetic interactions between tumor suppressors Brca1 and p53 in apoptosis, cell cycle and tumorigenesis

Nature genetics, 2001-07, Vol.28 (3), p.266-271

2001

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The p53 network in lung carcinogenesis

Oncogene, 2002-10, Vol.21 (45), p.6898-6907

2002

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Involvement of Crm1 in Hepatitis B Virus X Protein-Induced Aberrant Centriole Replication and Abnormal Mitotic Spindles

Molecular and Cellular Biology, 2003-08, Vol.23 (15), p.5282-5292

2003

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p53 interacts with hRAD51 and hRAD54, and directly modulates homologous recombination

Cancer research (Chicago, Ill.), 2003-05, Vol.63 (10), p.2596-2605

2003

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Functional Interaction between BLM Helicase and 53BP1 in a Chk1-Mediated Pathway during S-Phase Arrest

The Journal of cell biology, 2004-09, Vol.166 (6), p.801-813

2004

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A multimarker model to predict outcome in tamoxifen-treated breast cancer patients

Clinical cancer research, 2006-02, Vol.12 (4), p.1175-1183

2006

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DNA rereplication in the presence of mitotic spindle inhibitors in human and mouse fibroblasts lacking either p53 or pRb function

Cancer research (Chicago, Ill.), 1997-03, Vol.57 (6), p.1013-1019

1997

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P2-12-25: Blinded Multi-Site Validation of a Pathology-Based Prognostic Marker Profile for Operable Hormone Receptor-Positive Breast Cancer

Cancer research (Chicago, Ill.), 2011-12, Vol.71 (24_Supplement), p.P2-P2-12-25

2011

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P4-18-01: Prognostic Role of Tumor and Stromal p16/INK4A in Ductal Carcinoma In Situ (DCIS)

Cancer research (Chicago, Ill.), 2011-12, Vol.71 (24_Supplement), p.P4-P4-18-01

2011

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The Processing of Holliday Junctions by BLM and WRN Helicases Is Regulated by p53

The Journal of biological chemistry, 2002-08, Vol.277 (35), p.31980-31987

2002

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Functional Interaction of p53 and BLM DNA Helicase in Apoptosis

The Journal of biological chemistry, 2001-08, Vol.276 (35), p.32948-32955

2001

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The mismatch DNA repair heterodimer, hMSH2 6, regulates BLM helicase

Oncogene, 2004-05, Vol.23 (21), p.3749-3756

2004

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Abstract P3-10-42: Stromal Caveolin-1 Is a Powerful Marker That Further Enhances a Multi-Marker Prognostic Profile

Cancer research (Chicago, Ill.), 2010-12, Vol.70 (24_Supplement), p.P3-P3-10-42

2010

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BLM helicase facilitates Mus81 endonuclease activity in human cells

Cancer research (Chicago, Ill.), 2005-04, Vol.65 (7), p.2526-2531

2005

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p53 mediates permanent arrest over multiple cell cycles in response to γ-irradiation

Cancer research (Chicago, Ill.), 1997-03, Vol.57 (6), p.1171-1179

1997

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UV-C-induced DNA damage leads to p53-dependent nuclear trafficking of PML

Oncogene, 2003-03, Vol.22 (11), p.1620-1628

2003

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Immunohistochemistry-based marker profile accurately predicts risk in operable, hormone receptor-positive patients

Cancer research (Chicago, Ill.), 2009-01, Vol.69 (2_Supplement), p.1062

2009

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Human malignant glioma cell lines are sensitive to low radiation doses

International journal of cancer, 2003-05, Vol.105 (1), p.33-40

2003

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The Tumorigenic Potential and Cell Growth Characteristics of p53-Deficient Cells are Equivalent in the Presence or Absence of Mdm2

Proceedings of the National Academy of Sciences - PNAS, 1996-11, Vol.93 (24), p.14106-14111

1996

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Cancer Has the smart bomb been defused?

Nature (London), 1998-09, Vol.395 (6697), p.13-15

Linke, Steven P

1998

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Genetic instability as a consequence of inappropriate entry into and progression through S-phase

Cancer and metastasis reviews, 1995-03, Vol.14 (1), p.59-73

1995

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Maintaining genetic stability through TP53 mediated checkpoint control

Cancer surveys, 1997, Vol.29, p.183

1997

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Prognostic Risk Assessment and Prediction of Radiotherapy Benefit for Women with Ductal Carcinoma In Situ (DCIS) of the Breast, in a Randomized Clinical Trial (SweDCIS)

Nitric Oxide-Induced Cellular Stress and p53 Activation in Chronic Inflammation

BLM helicase-dependent transport of p53 to sites of stalled DNA replication forks modulates homologous recombination

Genetic interactions between tumor suppressors Brca1 and p53 in apoptosis, cell cycle and tumorigenesis

The p53 network in lung carcinogenesis

Involvement of Crm1 in Hepatitis B Virus X Protein-Induced Aberrant Centriole Replication and Abnormal Mitotic Spindles

p53 interacts with hRAD51 and hRAD54, and directly modulates homologous recombination

Functional Interaction between BLM Helicase and 53BP1 in a Chk1-Mediated Pathway during S-Phase Arrest

A multimarker model to predict outcome in tamoxifen-treated breast cancer patients

DNA rereplication in the presence of mitotic spindle inhibitors in human and mouse fibroblasts lacking either p53 or pRb function

P2-12-25: Blinded Multi-Site Validation of a Pathology-Based Prognostic Marker Profile for Operable Hormone Receptor-Positive Breast Cancer

P4-18-01: Prognostic Role of Tumor and Stromal p16/INK4A in Ductal Carcinoma In Situ (DCIS)

The Processing of Holliday Junctions by BLM and WRN Helicases Is Regulated by p53

Functional Interaction of p53 and BLM DNA Helicase in Apoptosis

The mismatch DNA repair heterodimer, hMSH2 6, regulates BLM helicase

Abstract P3-10-42: Stromal Caveolin-1 Is a Powerful Marker That Further Enhances a Multi-Marker Prognostic Profile

BLM helicase facilitates Mus81 endonuclease activity in human cells

p53 mediates permanent arrest over multiple cell cycles in response to γ-irradiation

UV-C-induced DNA damage leads to p53-dependent nuclear trafficking of PML

Immunohistochemistry-based marker profile accurately predicts risk in operable, hormone receptor-positive patients

Human malignant glioma cell lines are sensitive to low radiation doses

The Tumorigenic Potential and Cell Growth Characteristics of p53-Deficient Cells are Equivalent in the Presence or Absence of Mdm2

Cancer Has the smart bomb been defused?

Genetic instability as a consequence of inappropriate entry into and progression through S-phase

Maintaining genetic stability through TP53 mediated checkpoint control

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