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Down‐regulation of MEIS1 promotes the maturation of oxidative phosphorylation in perinatal cardiomyocytes

The FASEB journal, 2019-06, Vol.33 (6), p.7417-7426

2019

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The strong selective sweep candidate gene ADRA2C does not explain domestication related changes in the stress response of chickens

PloS one, 2014-08, Vol.9 (8), p.e103218-e103218

2014

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Chronic prenatal hypoxia sensitizes beta-adrenoceptors in the embryonic heart but causes postnatal desensitization

American journal of physiology. Regulatory, integrative and comparative physiology, 2009-08, Vol.297 (2), p.R258-R264

2009

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Sex-biased microRNA expression in mammals and birds reveals underlying regulatory mechanisms and a role in dosage compensation

Genome research, 2017-12, Vol.27 (12), p.1961

2017

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Prenatal hypoxia triggers maladaptive alterations in the heart of very late but not in late chicken embryos

The FASEB journal, 2011-04, Vol.25 (S1), p.1112.2-1112.2

2011

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Prenatal hypoxia programs changes in β-adrenergic signaling and postnatal cardiac contractile dysfunction

American journal of physiology. Regulatory, integrative and comparative physiology, 2013-11, Vol.305 (10), p.R1093-R1101

2013

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Thyroid hormone receptor function in maturing ovine cardiomyocytes

The Journal of physiology, 2019-04, Vol.597 (8), p.2163-2176

2019

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Abstract 573: Mechanical Stress Alters Cell Signaling and Extracellular Matrix Genes in Fetal Cardiac Valves

Circulation research, 2018-08, Vol.123 (Suppl_1)

2018

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Aerobic performance in tinamous is limited by their small heart. A novel hypothesis in the evolution of avian flight

Scientific reports, 2017-11, Vol.7 (1), p.15964-15, Article 15964

2017

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Chronic hypoxia during development does not trigger pathologic remodeling of the chicken embryonic heart but reduces cardiomyocyte number

American journal of physiology. Regulatory, integrative and comparative physiology, 2015-11, Vol.309 (10), p.R1204-R1214

2015

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Postnatal β‐adrenergic desensitization caused by chronic prenatal hypoxia is linked to an increase in Gs and decreased β1AR/β2AR ratio

The FASEB journal, 2011-04, Vol.25, p.858.15-858.15

2011

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Chronic prenatal hypoxia sensitizes {beta}-adrenoceptors in the embryonic heart but causes postnatal desensitization

American journal of physiology. Regulatory, integrative and comparative physiology, 2009-08, Vol.297 (2), p.R258

2009

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Role for Kisspeptin and Neurokinin B in Regulation of Luteinizing Hormone and Testosterone Secretion in the Fetal Sheep

Endocrinology (Philadelphia), 2020-04, Vol.161 (4), p.1

2020

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Chronic prenatal hypoxia sensitizes β-adrenoceptors in the embryonic heart but causes postnatal desensitization

American journal of physiology. Regulatory, integrative and comparative physiology, 2009, Vol.66 (2)

2009

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Pressure-induced maturation of endothelial cells on newborn mouse carotid arteries

American journal of physiology. Heart and circulatory physiology, 2013-08, Vol.305 (3), p.H321-H329

2013

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Prenatal hypoxia programs adult cardiac contractile dysfunction through changes in beta‐adrenergic signaling

The FASEB journal, 2013-04, Vol.27 (S1)

2013

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Mathematical modeling improves EC50 estimations from classical dose–response curves

The FEBS journal, 2015-03, Vol.282 (5), p.951-962

2015

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Hypotension in the chronically hypoxic chicken embryo is related to the β-adrenergic response of chorioallantoic and femoral arteries and not to bradycardia

American journal of physiology. Regulatory, integrative and comparative physiology, 2011-10, Vol.301 (4), p.R1161-R1168

2011

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Sensitivity of organ growth to chronically low oxygen levels during incubation in Red Junglefowl and domesticated chicken breeds

Poultry science, 2011, Vol.90 (1), p.126-135

2011

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Effect of chronic hypoxia on beta‐adrenoceptor density and cardiac contractile response to agonist during the last week of incubation in chicken fetuses

The FASEB journal, 2007, Vol.21 (6), p.A1208-A1208

2007

Volltextzugriff (PDF)

Mathematical modeling improves EC 50 estimations from classical dose–response curves

The FEBS journal, 2015-03, Vol.282 (5), p.951-962

2015

Volltextzugriff (PDF)

Reactivity of chicken chorioallantoic arteries, avian homologue of human fetoplacental arteries

Journal of physiology and pharmacology : an official journal of the Polish Physiological Society, 2010-10, Vol.61 (5), p.619

2010

Volltextzugriff (PDF)

Hypotension in the chronically hypoxic chicken embryo is related to the [Beta]-adrenergic response of chorioallantoic and femoral arteries and not to bradycardia

American journal of physiology. Regulatory, integrative and comparative physiology, 2011-10, Vol.301 (4), p.R1161

2011

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IGF‐1 induces proliferation and T 3 causes maturation of cultured embryonic chicken cardiomyocytes

The FASEB journal, 2013-04, Vol.27 (S1)

2013

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Prenatal hypoxia programs an altered β 1 /β 2 adrenoceptor subtype ratio in the heart of adult broiler chickens

The FASEB journal, 2009-04, Vol.23 (S1)

2009

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Down‐regulation of MEIS1 promotes the maturation of oxidative phosphorylation in perinatal cardiomyocytes

The strong selective sweep candidate gene ADRA2C does not explain domestication related changes in the stress response of chickens

Chronic prenatal hypoxia sensitizes beta-adrenoceptors in the embryonic heart but causes postnatal desensitization

Sex-biased microRNA expression in mammals and birds reveals underlying regulatory mechanisms and a role in dosage compensation

Prenatal hypoxia triggers maladaptive alterations in the heart of very late but not in late chicken embryos

Prenatal hypoxia programs changes in β-adrenergic signaling and postnatal cardiac contractile dysfunction

Thyroid hormone receptor function in maturing ovine cardiomyocytes

Abstract 573: Mechanical Stress Alters Cell Signaling and Extracellular Matrix Genes in Fetal Cardiac Valves

Aerobic performance in tinamous is limited by their small heart. A novel hypothesis in the evolution of avian flight

Chronic hypoxia during development does not trigger pathologic remodeling of the chicken embryonic heart but reduces cardiomyocyte number

Postnatal β‐adrenergic desensitization caused by chronic prenatal hypoxia is linked to an increase in Gs and decreased β1AR/β2AR ratio

Chronic prenatal hypoxia sensitizes {beta}-adrenoceptors in the embryonic heart but causes postnatal desensitization

Role for Kisspeptin and Neurokinin B in Regulation of Luteinizing Hormone and Testosterone Secretion in the Fetal Sheep

Chronic prenatal hypoxia sensitizes β-adrenoceptors in the embryonic heart but causes postnatal desensitization

Pressure-induced maturation of endothelial cells on newborn mouse carotid arteries

Prenatal hypoxia programs adult cardiac contractile dysfunction through changes in beta‐adrenergic signaling

Mathematical modeling improves EC50 estimations from classical dose–response curves

Hypotension in the chronically hypoxic chicken embryo is related to the β-adrenergic response of chorioallantoic and femoral arteries and not to bradycardia

Sensitivity of organ growth to chronically low oxygen levels during incubation in Red Junglefowl and domesticated chicken breeds

Effect of chronic hypoxia on beta‐adrenoceptor density and cardiac contractile response to agonist during the last week of incubation in chicken fetuses

Mathematical modeling improves EC 50 estimations from classical dose–response curves

Reactivity of chicken chorioallantoic arteries, avian homologue of human fetoplacental arteries

Hypotension in the chronically hypoxic chicken embryo is related to the [Beta]-adrenergic response of chorioallantoic and femoral arteries and not to bradycardia

IGF‐1 induces proliferation and T 3 causes maturation of cultured embryonic chicken cardiomyocytes

Prenatal hypoxia programs an altered β 1 /β 2 adrenoceptor subtype ratio in the heart of adult broiler chickens

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