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Dietary Antioxidants Significantly Attenuate Hyperoxia-Induced Acute Inflammatory Lung Injury by Enhancing Macrophage Function via Reducing the Accumulation of Airway HMGB1

International journal of molecular sciences, 2020-02, Vol.21 (3), p.977

2020

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The α7 nicotinic acetylcholine receptor agonist, GTS-21, attenuates hyperoxia-induced acute inflammatory lung injury by alleviating the accumulation of HMGB1 in the airways and the circulation

Molecular medicine (Cambridge, Mass.), 2020-06, Vol.26 (1), p.63-63, Article 63

2020

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A Standardized Botanical Composition Mitigated Acute Inflammatory Lung Injury and Reduced Mortality through Extracellular HMGB1 Reduction

Molecules (Basel, Switzerland), 2023-09, Vol.28 (18), p.6560

2023

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The α7 nicotinic acetylcholine receptor agonist GTS-21 improves bacterial clearance in mice by restoring hyperoxia-compromised macrophage function

Molecular medicine (Cambridge, Mass.), 2020-12, Vol.26 (1), p.1-98

2020

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Correction to: GTS-21, an α7nAChR agonist, increases pulmonary bacterial clearance in mice by restoring hyperoxia-compromised macrophage function

Molecular medicine (Cambridge, Mass.), 2021-08, Vol.27 (1), p.93-93, Article 93

2021

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From nicotine to the cholinergic anti-inflammatory reflex - Can nicotine alleviate the dysregulated inflammation in COVID-19?

Journal of immunotoxicology, 2021-01, Vol.18 (1), p.23-29

2021

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2-O, 3-O desulfated heparin (ODSH) increases bacterial clearance and attenuates lung injury in cystic fibrosis by restoring HMGB1-compromised macrophage function

Molecular medicine (Cambridge, Mass.), 2021-07, Vol.27 (1), p.79-79, Article 79

2021

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Retraction Note: The α7 nicotinic acetylcholine receptor agonist GTS-21 improves bacterial clearance in mice by restoring hyperoxia-compromised macrophage function

Molecular medicine (Cambridge, Mass.), 2020-12, Vol.26 (1), p.132-132, Article 132

2020

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Correction to: 2‑O, 3‑O desulfated heparin (ODSH) increases bacterial clearance and attenuates lung injury in cystic fibrosis by restoring HMGB1‑compromised macrophage function

Molecular medicine (Cambridge, Mass.), 2021-09, Vol.27 (1), p.98-98, Article 98

2021

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Hyperoxia sensing: From molecular mechanisms to significance in disease

Journal of immunotoxicology, 2010-12, Vol.7 (4), p.239-254

2010

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Inhibition of extracellular HMGB1 attenuates hyperoxia-induced inflammatory acute lung injury

Redox biology, 2014-01, Vol.2 (C), p.314-322

2014

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Expression of Concern to: The α7 nicotine acetylcholine receptor agonist GTS-21 improves bacterial clearance in mice by restoring hyperoxia-compromised macrophage function

Molecular medicine (Cambridge, Mass.), 2020-02, Vol.26 (1), p.16-16, Article 16

2020

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The α7 nicotinic acetylcholine receptor agonist GTS-21 improves bacterial clearance in mice by restoring hyperoxia-compromised macrophage function

Molecular medicine (Cambridge, Mass.), 2014-06, Vol.20 (1), p.238-247

2014

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HMGB-1 as a therapeutic target for infectious and inflammatory disorders

Shock (Augusta, Ga.), 2006, Vol.25 (1), p.4-11

2006

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The Role of HMGB1, a Nuclear Damage-Associated Molecular Pattern Molecule, in the Pathogenesis of Lung Diseases

Antioxidants & redox signaling, 2019-11, Vol.31 (13), p.954-993

2019

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Carbon monoxide provides protection against hyperoxic lung injury

American journal of physiology. Lung cellular and molecular physiology, 1999-04, Vol.276 (4), p.688-L694

1999

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Dual effects of supplemental oxygen on pulmonary infection, inflammatory lung injury, and neuromodulation in aging and COVID-19

Free radical biology & medicine, 2022-09, Vol.190, p.247-263

2022

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Carbon Nanomaterials Stimulate HMGB1 Release From Macrophages and Induce Cell Migration and Invasion

Toxicological sciences, 2019-12, Vol.172 (2), p.398-410

2019

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Interleukin-6-Induced Protection in Hyperoxic Acute Lung Injury

American journal of respiratory cell and molecular biology, 2000-05, Vol.22 (5), p.535-542

2000

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The nitric oxide donor, (Z)-1-[N-(2-aminoethyl)-N-(2-ammonioethyl)amino]diazen-1-ium-1,2-diolate (DETA-NONOate/D-NO), increases survival by attenuating hyperoxia-compromised innate immunity in bacterial clearance in a mouse model of ventilator-associated pneumonia

Biochemical pharmacology, 2020-06, Vol.176, p.113817-113817, Article 113817

2020

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Nitric Oxide Regulates Gene Expression in Cancers by Controlling Histone Posttranslational Modifications

Cancer research (Chicago, Ill.), 2015-12, Vol.75 (24), p.5299-5308

2015

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GAT107-mediated α7 nicotinic acetylcholine receptor signaling attenuates inflammatory lung injury and mortality in a mouse model of ventilator-associated pneumonia by alleviating macrophage mitochondrial oxidative stress via reducing MnSOD-S-glutathionylation

Redox biology, 2023-04, Vol.60, p.102614-102614, Article 102614

2023

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Inhibition of high-mobility group box 1 protein (HMGB1) enhances bacterial clearance and protects against Pseudomonas Aeruginosa pneumonia in cystic fibrosis

Molecular medicine (Cambridge, Mass.), 2012-05, Vol.18 (3), p.477-485

2012

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High Mobility Group Box-1 mediates hyperoxia-induced impairment of Pseudomonas aeruginosa clearance and inflammatory lung injury in mice

American journal of respiratory cell and molecular biology, 2013-03, Vol.48 (3), p.280-287

2013

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Ascorbic Acid Attenuates Hyperoxia-Compromised Host Defense against Pulmonary Bacterial Infection

American journal of respiratory cell and molecular biology, 2016-10, Vol.55 (4), p.511-520

2016

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Dietary Antioxidants Significantly Attenuate Hyperoxia-Induced Acute Inflammatory Lung Injury by Enhancing Macrophage Function via Reducing the Accumulation of Airway HMGB1

The α7 nicotinic acetylcholine receptor agonist, GTS-21, attenuates hyperoxia-induced acute inflammatory lung injury by alleviating the accumulation of HMGB1 in the airways and the circulation

A Standardized Botanical Composition Mitigated Acute Inflammatory Lung Injury and Reduced Mortality through Extracellular HMGB1 Reduction

The α7 nicotinic acetylcholine receptor agonist GTS-21 improves bacterial clearance in mice by restoring hyperoxia-compromised macrophage function

Correction to: GTS-21, an α7nAChR agonist, increases pulmonary bacterial clearance in mice by restoring hyperoxia-compromised macrophage function

From nicotine to the cholinergic anti-inflammatory reflex - Can nicotine alleviate the dysregulated inflammation in COVID-19?

2-O, 3-O desulfated heparin (ODSH) increases bacterial clearance and attenuates lung injury in cystic fibrosis by restoring HMGB1-compromised macrophage function

Retraction Note: The α7 nicotinic acetylcholine receptor agonist GTS-21 improves bacterial clearance in mice by restoring hyperoxia-compromised macrophage function

Correction to: 2‑O, 3‑O desulfated heparin (ODSH) increases bacterial clearance and attenuates lung injury in cystic fibrosis by restoring HMGB1‑compromised macrophage function

Hyperoxia sensing: From molecular mechanisms to significance in disease

Inhibition of extracellular HMGB1 attenuates hyperoxia-induced inflammatory acute lung injury

Expression of Concern to: The α7 nicotine acetylcholine receptor agonist GTS-21 improves bacterial clearance in mice by restoring hyperoxia-compromised macrophage function

The α7 nicotinic acetylcholine receptor agonist GTS-21 improves bacterial clearance in mice by restoring hyperoxia-compromised macrophage function

HMGB-1 as a therapeutic target for infectious and inflammatory disorders

The Role of HMGB1, a Nuclear Damage-Associated Molecular Pattern Molecule, in the Pathogenesis of Lung Diseases

Carbon monoxide provides protection against hyperoxic lung injury

Dual effects of supplemental oxygen on pulmonary infection, inflammatory lung injury, and neuromodulation in aging and COVID-19

Carbon Nanomaterials Stimulate HMGB1 Release From Macrophages and Induce Cell Migration and Invasion

Interleukin-6-Induced Protection in Hyperoxic Acute Lung Injury

The nitric oxide donor, (Z)-1-[N-(2-aminoethyl)-N-(2-ammonioethyl)amino]diazen-1-ium-1,2-diolate (DETA-NONOate/D-NO), increases survival by attenuating hyperoxia-compromised innate immunity in bacterial clearance in a mouse model of ventilator-associated pneumonia

Nitric Oxide Regulates Gene Expression in Cancers by Controlling Histone Posttranslational Modifications

GAT107-mediated α7 nicotinic acetylcholine receptor signaling attenuates inflammatory lung injury and mortality in a mouse model of ventilator-associated pneumonia by alleviating macrophage mitochondrial oxidative stress via reducing MnSOD-S-glutathionylation

Inhibition of high-mobility group box 1 protein (HMGB1) enhances bacterial clearance and protects against Pseudomonas Aeruginosa pneumonia in cystic fibrosis

High Mobility Group Box-1 mediates hyperoxia-induced impairment of Pseudomonas aeruginosa clearance and inflammatory lung injury in mice

Ascorbic Acid Attenuates Hyperoxia-Compromised Host Defense against Pulmonary Bacterial Infection

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