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A causal role for hyperinsulinemia in obesity

Journal of endocrinology, 2017-03, Vol.232 (3), p.R173-R183

2017

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Hyperinsulinemia Drives Diet-Induced Obesity Independently of Brain Insulin Production

Cell metabolism, 2012-12, Vol.16 (6), p.723-737

2012

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Reduced Circulating Insulin Enhances Insulin Sensitivity in Old Mice and Extends Lifespan

Cell reports (Cambridge), 2017-07, Vol.20 (2), p.451-463

2017

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Is dynamic autocrine insulin signaling possible? A mathematical model predicts picomolar concentrations of extracellular monomeric insulin within human pancreatic islets

PloS one, 2013-06, Vol.8 (6), p.e64860-e64860

2013

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Metabolic Contributions of Wnt Signaling: More Than Controlling Flight

Frontiers in cell and developmental biology, 2021-09, Vol.9, p.709823-709823

2021

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Insulin Regulates Glucagon-Like Peptide-1 Secretion from the Enteroendocrine L Cell

Endocrinology (Philadelphia), 2009-02, Vol.150 (2), p.580-591

2009

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Cross Talk between the Insulin and Wnt Signaling Pathways: Evidence from Intestinal Endocrine L Cells

Endocrinology (Philadelphia), 2008-05, Vol.149 (5), p.2341-2351

2008

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14-3-3ζ coordinates adipogenesis of visceral fat

Nature communications, 2015-07, Vol.6 (1), p.7671, Article 7671

2015

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Corrigendum to “Inter-domain tagging implicates caveolin-1 in insulin receptor trafficking and Erk signaling bias in pancreatic beta-cells” [Mol Metab 2016 May; 5 (5): 366–378]

Molecular metabolism (Germany), 2024-05, Vol.83, p.101935-101935

2024

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Scaffold Proteins: From Coordinating Signaling Pathways to Metabolic Regulation

Endocrinology (Philadelphia), 2018-11, Vol.159 (11), p.3615-3630

2018

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Inter-domain tagging implicates caveolin-1 in insulin receptor trafficking and Erk signaling bias in pancreatic beta-cells

Molecular metabolism (Germany), 2016-05, Vol.5 (5), p.366-378

2016

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14-3-3ζ Constrains insulin secretion by regulating mitochondrial function in pancreatic β cells

JCI insight, 2022-04, Vol.7 (8)

2022

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Prenatal Exposure to Nicotine Causes Postnatal Obesity and Altered Perivascular Adipose Tissue Function

Obesity (Silver Spring, Md.), 2005-04, Vol.13 (4), p.687-692

2005

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14-3-3ζ mediates an alternative, non-thermogenic mechanism in male mice to reduce heat loss and improve cold tolerance

Molecular metabolism (Germany), 2020-11, Vol.41, p.101052-101052, Article 101052

2020

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The Rho Guanosine 5′-Triphosphatase, Cell Division Cycle 42, Is Required for Insulin-Induced Actin Remodeling and Glucagon-Like Peptide-1 Secretion in the Intestinal Endocrine L Cell

Endocrinology (Philadelphia), 2009-12, Vol.150 (12), p.5249-5261

2009

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Is 14-3-3 the Combination to Unlock New Pathways to Improve Metabolic Homeostasis and β-Cell Function?

Diabetes (New York, N.Y.), 2023-08, Vol.72 (8), p.1045-1054

2023

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2015-P: 14-3-3zeta Is Required for PKA-Dependent Lipolysis

Diabetes (New York, N.Y.), 2019-06, Vol.68 (Supplement_1)

2019

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1775-P: Liraglutide Corrects Impaired Insulin Secretion in 14-3-3-ζ Overexpressing Mice

Diabetes (New York, N.Y.), 2023-06, Vol.72 (Supplement_1), p.1

2023

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Can 14-3-3 proteins serve as therapeutic targets for the treatment of metabolic diseases?

Pharmacological research, 2019-01, Vol.139, p.199-206

2019

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Glucagon-like peptide 1 secretion by the L-cell : The view from within

Diabetes (New York, N.Y.), 2006-12, Vol.55 (12), p.S70

2006

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1396-P: Critical Cellular Functions That Mediate the Early Stages of Adipogenesis Are Directly Influenced by 14-3-3zeta and Its Interactome

Diabetes (New York, N.Y.), 2022-06, Vol.71 (Supplement_1)

2022

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The Application of High‐Throughput Approaches in Identifying Novel Therapeutic Targets and Agents to Treat Diabetes

Advanced biology, 2023-02, Vol.7 (2), p.e2200151-n/a

2023

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321-OR: Deletion of 14-3-3ζ in Pancreatic ß-Cells Increase Mitochondrial Activity and Insulin Secretion

Diabetes (New York, N.Y.), 2020-06, Vol.69 (Supplement_1)

2020

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Reducing 14-3-3ζ expression influences adipocyte maturity and impairs function

American journal of physiology: endocrinology and metabolism, 2020-07, Vol.319 (1), p.E117-E132

2020

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Adipose depot-specific upregulation of Ucp1 or mitochondrial oxidative complex proteins are early consequences of genetic insulin reduction in mice

American journal of physiology: endocrinology and metabolism, 2020-09, Vol.319 (3), p.E529-E539

2020

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A causal role for hyperinsulinemia in obesity

Hyperinsulinemia Drives Diet-Induced Obesity Independently of Brain Insulin Production

Reduced Circulating Insulin Enhances Insulin Sensitivity in Old Mice and Extends Lifespan

Is dynamic autocrine insulin signaling possible? A mathematical model predicts picomolar concentrations of extracellular monomeric insulin within human pancreatic islets

Metabolic Contributions of Wnt Signaling: More Than Controlling Flight

Insulin Regulates Glucagon-Like Peptide-1 Secretion from the Enteroendocrine L Cell

Cross Talk between the Insulin and Wnt Signaling Pathways: Evidence from Intestinal Endocrine L Cells

14-3-3ζ coordinates adipogenesis of visceral fat

Corrigendum to “Inter-domain tagging implicates caveolin-1 in insulin receptor trafficking and Erk signaling bias in pancreatic beta-cells” [Mol Metab 2016 May; 5 (5): 366–378]

Scaffold Proteins: From Coordinating Signaling Pathways to Metabolic Regulation

Inter-domain tagging implicates caveolin-1 in insulin receptor trafficking and Erk signaling bias in pancreatic beta-cells

14-3-3ζ Constrains insulin secretion by regulating mitochondrial function in pancreatic β cells

Prenatal Exposure to Nicotine Causes Postnatal Obesity and Altered Perivascular Adipose Tissue Function

14-3-3ζ mediates an alternative, non-thermogenic mechanism in male mice to reduce heat loss and improve cold tolerance

The Rho Guanosine 5′-Triphosphatase, Cell Division Cycle 42, Is Required for Insulin-Induced Actin Remodeling and Glucagon-Like Peptide-1 Secretion in the Intestinal Endocrine L Cell

Is 14-3-3 the Combination to Unlock New Pathways to Improve Metabolic Homeostasis and β-Cell Function?

2015-P: 14-3-3zeta Is Required for PKA-Dependent Lipolysis

1775-P: Liraglutide Corrects Impaired Insulin Secretion in 14-3-3-ζ Overexpressing Mice

Can 14-3-3 proteins serve as therapeutic targets for the treatment of metabolic diseases?

Glucagon-like peptide 1 secretion by the L-cell : The view from within

1396-P: Critical Cellular Functions That Mediate the Early Stages of Adipogenesis Are Directly Influenced by 14-3-3zeta and Its Interactome

The Application of High‐Throughput Approaches in Identifying Novel Therapeutic Targets and Agents to Treat Diabetes

321-OR: Deletion of 14-3-3ζ in Pancreatic ß-Cells Increase Mitochondrial Activity and Insulin Secretion

Reducing 14-3-3ζ expression influences adipocyte maturity and impairs function

Adipose depot-specific upregulation of Ucp1 or mitochondrial oxidative complex proteins are early consequences of genetic insulin reduction in mice

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