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Motor neuron disease-associated loss of nuclear TDP-43 is linked to DNA double-strand break repair defects

Proceedings of the National Academy of Sciences - PNAS, 2019-03, Vol.116 (10), p.4696-4705

2019

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The tau tubulin kinases TTBK1/2 promote accumulation of pathological TDP-43

PLoS genetics, 2014-12, Vol.10 (12), p.e1004803-e1004803

2014

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Protein Kinase CK‑1 Inhibitors As New Potential Drugs for Amyotrophic Lateral Sclerosis

Journal of medicinal chemistry, 2014-03, Vol.57 (6), p.2755-2772

2014

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Constitutive XBP-1s-mediated activation of the endoplasmic reticulum unfolded protein response protects against pathological tau

Nature communications, 2019-09, Vol.10 (1), p.4443-12, Article 4443

2019

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TDP-43/FUS in motor neuron disease: Complexity and challenges

Progress in neurobiology, 2016-10, Vol.145-146, p.78-97

2016

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Phosphorylation promotes neurotoxicity in a Caenorhabditis elegans model of TDP-43 proteinopathy

The Journal of neuroscience, 2010-12, Vol.30 (48), p.16208-16219

2010

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Regulation of TDP-43 phosphorylation in aging and disease

GeroScience, 2021-08, Vol.43 (4), p.1605-1614

2021

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Genome wide analysis reveals heparan sulfate epimerase modulates TDP-43 proteinopathy

PLoS genetics, 2019-12, Vol.15 (12), p.e1008526

2019

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CDC7 inhibition blocks pathological TDP-43 phosphorylation and neurodegeneration

Annals of neurology, 2013-07, Vol.74 (1), p.39-52

2013

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Transgenic Dendra2::tau expression allows in vivo monitoring of tau proteostasis in Caenorhabditis elegans

Disease models & mechanisms, 2024-03, Vol.17 (3)

2024

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Targeting nuclear protein TDP-43 by cell division cycle kinase 7 inhibitors: A new therapeutic approach for amyotrophic lateral sclerosis

European journal of medicinal chemistry, 2021-01, Vol.210, p.112968, Article 112968

2021

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Simple models to understand complex disease: 10 years of progress from Caenorhabditis elegans models of amyotrophic lateral sclerosis and frontotemporal lobar degeneration

Frontiers in neuroscience, 2024-01, Vol.17, p.1300705

2024

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Transcriptomic evaluation of tau and TDP-43 synergism shows tauopathy predominance and reveals potential modulating targets

Neurobiology of disease, 2024-04, Vol.193, p.106441-106441, Article 106441

2024

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Pathological phosphorylation of tau and TDP-43 by TTBK1 and TTBK2 drives neurodegeneration

Molecular neurodegeneration, 2018-02, Vol.13 (1), p.7-7, Article 7

2018

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Tau–RNA complexes inhibit microtubule polymerization and drive disease-relevant conformation change

Brain (London, England : 1878), 2023-08, Vol.146 (8), p.3206-3220

2023

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SPOP loss of function protects against tauopathy

Proceedings of the National Academy of Sciences - PNAS, 2023-01, Vol.120 (1), p.e2207250120

2023

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Tau tubulin kinases in proteinopathy

The FEBS journal, 2019-07, Vol.286 (13), p.2434-2446

2019

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Evaluation of Motor Impairment in C. elegans Models of Amyotrophic Lateral Sclerosis

Journal of visualized experiments, 2021-09 (175)

2021

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Resistance and resilience to Alzheimer's disease pathology are associated with reduced cortical pTau and absence of limbic-predominant age-related TDP-43 encephalopathy in a community-based cohort

Acta neuropathologica communications, 2019-06, Vol.7 (1), p.91-91, Article 91

2019

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Sertraline, Paroxetine, and Chlorpromazine Are Rapidly Acting Anthelmintic Drugs Capable of Clinical Repurposing

Scientific reports, 2018-01, Vol.8 (1), p.975-17, Article 975

2018

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Tau and TDP-43 synergy: a novel therapeutic target for sporadic late-onset Alzheimer’s disease

GeroScience, 2021-08, Vol.43 (4), p.1627-1634

2021

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TDP‐43 promotes pathological tau phosphorylation and selective neurotoxicity in C. elegans

Alzheimer's & dementia, 2021-12, Vol.17 (S2), p.e058137-n/a

2021

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Activity of the poly(A) binding protein MSUT2 determines susceptibility to pathological tau in the mammalian brain

Science translational medicine, 2019-12, Vol.11 (523)

2019

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Hereditary spastic paraplegia with thin corpus callosum and SPG11 mutation: A neuropathological evaluation

Neuropathology, 2024-10

2024

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TDP-43 promotes tau accumulation and selective neurotoxicity in bigenic Caenorhabditis elegans

Disease models & mechanisms, 2022-04, Vol.15 (4)

2022

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Motor neuron disease-associated loss of nuclear TDP-43 is linked to DNA double-strand break repair defects

The tau tubulin kinases TTBK1/2 promote accumulation of pathological TDP-43

Protein Kinase CK‑1 Inhibitors As New Potential Drugs for Amyotrophic Lateral Sclerosis

Constitutive XBP-1s-mediated activation of the endoplasmic reticulum unfolded protein response protects against pathological tau

TDP-43/FUS in motor neuron disease: Complexity and challenges

Phosphorylation promotes neurotoxicity in a Caenorhabditis elegans model of TDP-43 proteinopathy

Regulation of TDP-43 phosphorylation in aging and disease

Genome wide analysis reveals heparan sulfate epimerase modulates TDP-43 proteinopathy

CDC7 inhibition blocks pathological TDP-43 phosphorylation and neurodegeneration

Transgenic Dendra2::tau expression allows in vivo monitoring of tau proteostasis in Caenorhabditis elegans

Targeting nuclear protein TDP-43 by cell division cycle kinase 7 inhibitors: A new therapeutic approach for amyotrophic lateral sclerosis

Simple models to understand complex disease: 10 years of progress from Caenorhabditis elegans models of amyotrophic lateral sclerosis and frontotemporal lobar degeneration

Transcriptomic evaluation of tau and TDP-43 synergism shows tauopathy predominance and reveals potential modulating targets

Pathological phosphorylation of tau and TDP-43 by TTBK1 and TTBK2 drives neurodegeneration

Tau–RNA complexes inhibit microtubule polymerization and drive disease-relevant conformation change

SPOP loss of function protects against tauopathy

Tau tubulin kinases in proteinopathy

Evaluation of Motor Impairment in C. elegans Models of Amyotrophic Lateral Sclerosis

Resistance and resilience to Alzheimer's disease pathology are associated with reduced cortical pTau and absence of limbic-predominant age-related TDP-43 encephalopathy in a community-based cohort

Sertraline, Paroxetine, and Chlorpromazine Are Rapidly Acting Anthelmintic Drugs Capable of Clinical Repurposing

Tau and TDP-43 synergy: a novel therapeutic target for sporadic late-onset Alzheimer’s disease

TDP‐43 promotes pathological tau phosphorylation and selective neurotoxicity in C. elegans

Activity of the poly(A) binding protein MSUT2 determines susceptibility to pathological tau in the mammalian brain

Hereditary spastic paraplegia with thin corpus callosum and SPG11 mutation: A neuropathological evaluation

TDP-43 promotes tau accumulation and selective neurotoxicity in bigenic Caenorhabditis elegans

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