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Down-regulation of the potassium-chloride cotransporter KCC2 contributes to spasticity after spinal cord injury

Nature Medicine, 2010-03, Vol.16 (3), p.302-307

2010

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Activation of 5-HT2A receptors upregulates the function of the neuronal K-Cl cotransporter KCC2

Proceedings of the National Academy of Sciences - PNAS, 2013-01, Vol.110 (1), p.348-353

2013

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Activation of 5-HT2A Receptors Restores KCC2 Function and Reduces Neuropathic Pain after Spinal Cord Injury

Neuroscience, 2018-09, Vol.387, p.48-57

2018

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Cleavage of Na(+) channels by calpain increases persistent Na(+) current and promotes spasticity after spinal cord injury

Nature Medicine, 2016-04, Vol.22 (4), p.404-411

2016

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Calpain fosters the hyperexcitability of motoneurons after spinal cord injury and leads to spasticity

eLife, 2019-12, Vol.8

2019

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Riluzole treatment modulates KCC2 and EAAT-2 receptor expression and Ca2+ accumulation following ventral root avulsion injury

European journal of cell biology, 2023-06, Vol.102 (2), p.151317-151317, Article 151317

2023

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Prenatal Hypoxia Induces Cl– Cotransporters KCC2 and NKCC1 Developmental Abnormality and Disturbs the Influence of GABAA and Glycine Receptors on Fictive Breathing in a Newborn Rat

Frontiers in physiology, 2022-02, Vol.13, p.786714-786714

2022

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Contribution of the potassium-chloride co-transporter KCC2 to the modulation of lumbar spinal networks in mice

The European journal of neuroscience, 2011-04, Vol.33 (7), p.1212-1222

2011

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Mild Intrauterine Hypoperfusion Leads to Lumbar and Cortical Hyperexcitability, Spasticity, and Muscle Dysfunctions in Rats: Implications for Prematurity

Frontiers in neurology, 2018-06, Vol.9, p.423-423

2018

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Differential effects of spinal cord transection on glycinergic and GABAergic synaptic signaling in sub-lesional lumbar motoneurons

Journal of chemical neuroanatomy, 2020-04, Vol.113

2020

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Inhibitory Postsynaptic Potentials in Lumbar Motoneurons Remain Depolarizing After Neonatal Spinal Cord Transection in the Rat

Journal of neurophysiology, 2006-11, Vol.96 (5), p.2274-2281

2006

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Differential effects of spinal cord transection on glycinergic and GABAergic synaptic signaling in sub-lesional lumbar motoneurons

Journal of chemical neuroanatomy, 2021-04, Vol.113, p.101847-101847, Article 101847

2021

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Early Hypoexcitability in a Subgroup of Spinal Motoneurons in Superoxide Dismutase 1 Transgenic Mice, a Model of Amyotrophic Lateral Sclerosis

Neuroscience, 2021-05, Vol.463, p.337-353

2021

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Early Hypoexcitability in a Subgroup of Spinal Motoneurons in Superoxide Dismutase 1 Transgenic Mice, a Model of Amyotrophic Lateral Sclerosis

Neuroscience, 2021-05, Vol.463, p.337-353

2021

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Activation of 5-HT 2A Receptors Restores KCC2 Function and Reduces Neuropathic Pain after Spinal Cord Injury

Neuroscience, 2018-09, Vol.387, p.48

2018

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Prenatal Hypoxia Induces Cl - Cotransporters KCC2 and NKCC1 Developmental Abnormality and Disturbs the Influence of GABA A and Glycine Receptors on Fictive Breathing in a Newborn Rat

Frontiers in physiology, 2022-02, Vol.13, p.786714

2022

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Prochlorperazine Increases KCC2 Function and Reduces Spasticity after Spinal Cord Injury

Journal of neurotrauma, 2017-12, Vol.34 (24), p.3397

2017

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Riluzole treatment modulates KCC2 and EAAT-2 receptor expression and Ca 2+ accumulation following ventral root avulsion injury

European journal of cell biology, 2023-06, Vol.102 (2), p.151317

2023

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Activation of 5-HT 2A receptors restores KCC2 function and reduces neuropathic pain after spinal cord injury

Neuroscience, 2017

2017

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Cleavage of Na super(+) channels by calpain increases persistent Na super(+) current and promotes spasticity after spinal cord injury

Nature medicine, 2016-04, Vol.22 (4), p.404-411

2016

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Cleavage of [Na.sup.+] channels by calpain increases persistent [Na.sup.+] current and promotes spasticity after spinal cord injury

Nature medicine, 2016-04, Vol.22 (4), p.404

2016

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Activation of 5-HT2A receptors upregulates the function of the neuronal K-CI cotransporter KCC2

Proceedings of the National Academy of Sciences - PNAS, 2013, Vol.110 (1), p.348-353

2013

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Mild Intrauterine Hypoperfusion Leads to Lumbar and Cortical Hyperexcitability, Spasticity, and Muscle Dysfunctions in Rats: Implications for Prematurity

Frontiers in neurology, 2018-06, Vol.9

2018

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Contribution of the potassiumachloride coatransporter KCC2 to the modulation of lumbar spinal networks in mice

The European journal of neuroscience, 2011-04, Vol.33 (7), p.1212-1222

2011

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Contribution of the potassium-chloride co-transporter KCC2 to the modulation of lumbar spinal networks in mice: KCC2 and modulation of lumbar spinal networks

The European journal of neuroscience, 2011-04, Vol.33 (7), p.1212-1222

2011

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Down-regulation of the potassium-chloride cotransporter KCC2 contributes to spasticity after spinal cord injury

Activation of 5-HT2A receptors upregulates the function of the neuronal K-Cl cotransporter KCC2

Activation of 5-HT2A Receptors Restores KCC2 Function and Reduces Neuropathic Pain after Spinal Cord Injury

Cleavage of Na(+) channels by calpain increases persistent Na(+) current and promotes spasticity after spinal cord injury

Calpain fosters the hyperexcitability of motoneurons after spinal cord injury and leads to spasticity

Riluzole treatment modulates KCC2 and EAAT-2 receptor expression and Ca2+ accumulation following ventral root avulsion injury

Prenatal Hypoxia Induces Cl– Cotransporters KCC2 and NKCC1 Developmental Abnormality and Disturbs the Influence of GABAA and Glycine Receptors on Fictive Breathing in a Newborn Rat

Contribution of the potassium-chloride co-transporter KCC2 to the modulation of lumbar spinal networks in mice

Mild Intrauterine Hypoperfusion Leads to Lumbar and Cortical Hyperexcitability, Spasticity, and Muscle Dysfunctions in Rats: Implications for Prematurity

Differential effects of spinal cord transection on glycinergic and GABAergic synaptic signaling in sub-lesional lumbar motoneurons

Inhibitory Postsynaptic Potentials in Lumbar Motoneurons Remain Depolarizing After Neonatal Spinal Cord Transection in the Rat

Differential effects of spinal cord transection on glycinergic and GABAergic synaptic signaling in sub-lesional lumbar motoneurons

Early Hypoexcitability in a Subgroup of Spinal Motoneurons in Superoxide Dismutase 1 Transgenic Mice, a Model of Amyotrophic Lateral Sclerosis

Early Hypoexcitability in a Subgroup of Spinal Motoneurons in Superoxide Dismutase 1 Transgenic Mice, a Model of Amyotrophic Lateral Sclerosis

Activation of 5-HT 2A Receptors Restores KCC2 Function and Reduces Neuropathic Pain after Spinal Cord Injury

Prenatal Hypoxia Induces Cl - Cotransporters KCC2 and NKCC1 Developmental Abnormality and Disturbs the Influence of GABA A and Glycine Receptors on Fictive Breathing in a Newborn Rat

Prochlorperazine Increases KCC2 Function and Reduces Spasticity after Spinal Cord Injury

Riluzole treatment modulates KCC2 and EAAT-2 receptor expression and Ca 2+ accumulation following ventral root avulsion injury

Activation of 5-HT 2A receptors restores KCC2 function and reduces neuropathic pain after spinal cord injury

Cleavage of Na super(+) channels by calpain increases persistent Na super(+) current and promotes spasticity after spinal cord injury

Cleavage of [Na.sup.+] channels by calpain increases persistent [Na.sup.+] current and promotes spasticity after spinal cord injury

Activation of 5-HT2A receptors upregulates the function of the neuronal K-CI cotransporter KCC2

Mild Intrauterine Hypoperfusion Leads to Lumbar and Cortical Hyperexcitability, Spasticity, and Muscle Dysfunctions in Rats: Implications for Prematurity

Contribution of the potassiumachloride coatransporter KCC2 to the modulation of lumbar spinal networks in mice

Contribution of the potassium-chloride co-transporter KCC2 to the modulation of lumbar spinal networks in mice: KCC2 and modulation of lumbar spinal networks

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