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Akap1 deficiency exacerbates diabetic cardiomyopathy in mice by NDUFS1-mediated mitochondrial dysfunction and apoptosis

Diabetologia, 2020-05, Vol.63 (5), p.1072-1087

2020

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Sirt3 deficiency exacerbates diabetic cardiac dysfunction: Role of Foxo3A-Parkin-mediated mitophagy

Biochimica et biophysica acta. Molecular basis of disease, 2017-08, Vol.1863 (8), p.1973-1983

2017

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Luteolin Attenuates Doxorubicin-Induced Cardiotoxicity Through Promoting Mitochondrial Autophagy

Frontiers in physiology, 2020-02, Vol.11, p.113-113

2020

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Luteolin attenuates sepsis‑induced myocardial injury by enhancing autophagy in mice

International journal of molecular medicine, 2020-05, Vol.45 (5), p.1477-1487

2020

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Melatonin Ameliorates the Progression of Atherosclerosis via Mitophagy Activation and NLRP3 Inflammasome Inhibition

Oxidative medicine and cellular longevity, 2018-01, Vol.2018 (1), p.9286458-9286458

2018

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Defective branched chain amino acid catabolism contributes to cardiac dysfunction and remodeling following myocardial infarction

American journal of physiology. Heart and circulatory physiology, 2016-11, Vol.311 (5), p.H1160-H1169

2016

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TBC1D15/RAB7-regulated mitochondria-lysosome interaction confers cardioprotection against acute myocardial infarction-induced cardiac injury

Theranostics, 2020-01, Vol.10 (24), p.11244-11263

2020

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SIRT1 Activation by Resveratrol Alleviates Cardiac Dysfunction via Mitochondrial Regulation in Diabetic Cardiomyopathy Mice

Oxidative medicine and cellular longevity, 2017-01, Vol.2017 (1), p.4602715-4602715

2017

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MST1 coordinately regulates autophagy and apoptosis in diabetic cardiomyopathy in mice

Diabetologia, 2016-11, Vol.59 (11), p.2435-2447

2016

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Pretreatment of Diabetic Adipose-derived Stem Cells with mitoTEMPO Reverses their Defective Proangiogenic Function in Diabetic Mice with Critical Limb Ischemia

Cell transplantation, 2019-12, Vol.28 (12), p.1652-1663

2019

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Activation of Cannabinoid Receptor Type II by AM1241 Ameliorates Myocardial Fibrosis via Nrf2-Mediated Inhibition of TGF-β1/Smad3 Pathway in Myocardial Infarction Mice

Cellular physiology and biochemistry, 2016, Vol.39 (4), p.1521-1536

2016

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Small extracellular vesicles derived from Nrf2-overexpressing human amniotic mesenchymal stem cells protect against lipopolysaccharide-induced acute lung injury by inhibiting NLRP3

Biology direct, 2022-11, Vol.17 (1), p.35-13, Article 35

2022

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Adipsin alleviates cardiac microvascular injury in diabetic cardiomyopathy through Csk-dependent signaling mechanism

BMC medicine, 2023-05, Vol.21 (1), p.197-197, Article 197

2023

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Luteolin alleviates post‐infarction cardiac dysfunction by up‐regulating autophagy through Mst1 inhibition

Journal of cellular and molecular medicine, 2016-01, Vol.20 (1), p.147-156

2016

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Rosuvastatin enhances the therapeutic efficacy of adipose-derived mesenchymal stem cells for myocardial infarction via PI3K/Akt and MEK/ERK pathways

Basic research in cardiology, 2013-03, Vol.108 (2), p.333-333, Article 333

2013

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Tailorable Hydrogel Improves Retention and Cardioprotection of Intramyocardial Transplanted Mesenchymal Stem Cells for the Treatment of Acute Myocardial Infarction in Mice

Journal of the American Heart Association, 2020-01, Vol.9 (2), p.e013784-e013784

2020

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18F-FDG PET imaging-monitored anti-inflammatory therapy for acute myocardial infarction: Exploring the role of MCC950 in murine model

Journal of nuclear cardiology, 2021-10, Vol.28 (5), p.2346-2357

2021

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Mst1 inhibits CMECs autophagy and participates in the development of diabetic coronary microvascular dysfunction

Scientific reports, 2016-09, Vol.6 (1), p.34199-34199, Article 34199

2016

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Ghrelin Improves Functional Survival of Engrafted Adipose-Derived Mesenchymal Stem Cells in Ischemic Heart through PI3K/Akt Signaling Pathway

BioMed research international, 2015-01, Vol.2015, p.858349-12

2015

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OSM mitigates post-infarction cardiac remodeling and dysfunction by up-regulating autophagy through Mst1 suppression

Biochimica et biophysica acta. Molecular basis of disease, 2017-08, Vol.1863 (8), p.1951-1961

2017

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Oncostatin M-induced cardiomyocyte dedifferentiation regulates the progression of diabetic cardiomyopathy through B-Raf/Mek/Erk signaling pathway

Acta biochimica et biophysica Sinica, 2016-03, Vol.48 (3), p.257-265

2016

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Lin28a protects against cardiac ischaemia/reperfusion injury in diabetic mice through the insulin‐PI3K‐mTOR pathway

Journal of cellular and molecular medicine, 2015-06, Vol.19 (6), p.1174-1182

2015

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Lin28a protects against diabetic cardiomyopathy via the PKA/ROCK2 pathway

Biochemical and biophysical research communications, 2016-01, Vol.469 (1), p.29-36

2016

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Irisin Promotes Cardiac Homing of Intravenously Delivered MSCs and Protects against Ischemic Heart Injury

Advanced science, 2022-03, Vol.9 (7), p.e2103697-n/a

2022

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Suppressing Mesenchymal Stromal Cell Ferroptosis Via Targeting a Metabolism‐Epigenetics Axis Corrects their Poor Retention and Insufficient Healing Benefits in the Injured Liver Milieu

Advanced science, 2023-05, Vol.10 (13), p.e2206439-n/a

2023

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Akap1 deficiency exacerbates diabetic cardiomyopathy in mice by NDUFS1-mediated mitochondrial dysfunction and apoptosis

Sirt3 deficiency exacerbates diabetic cardiac dysfunction: Role of Foxo3A-Parkin-mediated mitophagy

Luteolin Attenuates Doxorubicin-Induced Cardiotoxicity Through Promoting Mitochondrial Autophagy

Luteolin attenuates sepsis‑induced myocardial injury by enhancing autophagy in mice

Melatonin Ameliorates the Progression of Atherosclerosis via Mitophagy Activation and NLRP3 Inflammasome Inhibition

Defective branched chain amino acid catabolism contributes to cardiac dysfunction and remodeling following myocardial infarction

TBC1D15/RAB7-regulated mitochondria-lysosome interaction confers cardioprotection against acute myocardial infarction-induced cardiac injury

SIRT1 Activation by Resveratrol Alleviates Cardiac Dysfunction via Mitochondrial Regulation in Diabetic Cardiomyopathy Mice

MST1 coordinately regulates autophagy and apoptosis in diabetic cardiomyopathy in mice

Pretreatment of Diabetic Adipose-derived Stem Cells with mitoTEMPO Reverses their Defective Proangiogenic Function in Diabetic Mice with Critical Limb Ischemia

Activation of Cannabinoid Receptor Type II by AM1241 Ameliorates Myocardial Fibrosis via Nrf2-Mediated Inhibition of TGF-β1/Smad3 Pathway in Myocardial Infarction Mice

Small extracellular vesicles derived from Nrf2-overexpressing human amniotic mesenchymal stem cells protect against lipopolysaccharide-induced acute lung injury by inhibiting NLRP3

Adipsin alleviates cardiac microvascular injury in diabetic cardiomyopathy through Csk-dependent signaling mechanism

Luteolin alleviates post‐infarction cardiac dysfunction by up‐regulating autophagy through Mst1 inhibition

Rosuvastatin enhances the therapeutic efficacy of adipose-derived mesenchymal stem cells for myocardial infarction via PI3K/Akt and MEK/ERK pathways

Tailorable Hydrogel Improves Retention and Cardioprotection of Intramyocardial Transplanted Mesenchymal Stem Cells for the Treatment of Acute Myocardial Infarction in Mice

18F-FDG PET imaging-monitored anti-inflammatory therapy for acute myocardial infarction: Exploring the role of MCC950 in murine model

Mst1 inhibits CMECs autophagy and participates in the development of diabetic coronary microvascular dysfunction

Ghrelin Improves Functional Survival of Engrafted Adipose-Derived Mesenchymal Stem Cells in Ischemic Heart through PI3K/Akt Signaling Pathway

OSM mitigates post-infarction cardiac remodeling and dysfunction by up-regulating autophagy through Mst1 suppression

Oncostatin M-induced cardiomyocyte dedifferentiation regulates the progression of diabetic cardiomyopathy through B-Raf/Mek/Erk signaling pathway

Lin28a protects against cardiac ischaemia/reperfusion injury in diabetic mice through the insulin‐PI3K‐mTOR pathway

Lin28a protects against diabetic cardiomyopathy via the PKA/ROCK2 pathway

Irisin Promotes Cardiac Homing of Intravenously Delivered MSCs and Protects against Ischemic Heart Injury

Suppressing Mesenchymal Stromal Cell Ferroptosis Via Targeting a Metabolism‐Epigenetics Axis Corrects their Poor Retention and Insufficient Healing Benefits in the Injured Liver Milieu

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