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Bruton's tyrosine kinase inhibition increases BCL-2 dependence and enhances sensitivity to venetoclax in chronic lymphocytic leukemia

Leukemia, 2017-10, Vol.31 (10), p.2075-2084

2017

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Mimicking the BH3 domain to kill cancer cells

Oncogene, 2008-12, Vol.27 (S1), p.S149-S157

2008

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Pharmacological manipulation of Bcl-2 family members to control cell death

The Journal of clinical investigation, 2005-10, Vol.115 (10), p.2648-2655

Letai, Anthony

2005

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BH3 profiling identifies heterogeneous dependency on Bcl-2 family members in multiple myeloma and predicts sensitivity to BH3 mimetics

Leukemia, 2016-03, Vol.30 (3), p.761-764

2016

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p53 regulates a non-apoptotic death induced by ROS

Cell death and differentiation, 2013-11, Vol.20 (11), p.1465-1474

2013

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Displacement of Bim by Bmf and Puma rather than increase in Bim level mediates paclitaxel-induced apoptosis in breast cancer cells

Cell death and differentiation, 2010-10, Vol.17 (10), p.1624-1635

2010

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Decreased mitochondrial priming determines chemoresistance of colon cancer stem cells

Cell death and differentiation, 2014-07, Vol.21 (7), p.1170-1177

2014

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A novel orally active proteasome inhibitor induces apoptosis in multiple myeloma cells with mechanisms distinct from Bortezomib

Cancer cell, 2005-11, Vol.8 (5), p.407-419

2005

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Activity of a selective inhibitor of nuclear export, selinexor (KPT-330), against AML-initiating cells engrafted into immunosuppressed NSG mice

Leukemia, 2016-01, Vol.30 (1), p.190-199

2016

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Parkin selectively alters the intrinsic threshold for mitochondrial cytochrome c release

Human molecular genetics, 2009-11, Vol.18 (22), p.4317-4328

2009

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BH3 domains define selective inhibitory interactions with BHRF-1 and KSHV BCL-2

Cell death and differentiation, 2008-03, Vol.15 (3), p.580-588

2008

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Bcl-xL controls a switch between cell death modes during mitotic arrest

Cell death & disease, 2014-06, Vol.5 (6), p.e1291-e1291

2014

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Mitochondria—Judges and Executioners of Cell Death Sentences

Molecular cell, 2016-03, Vol.61 (5), p.695-704

2016

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BIM and other BCL-2 family proteins exhibit cross-species conservation of function between zebrafish and mammals

Cell death and differentiation, 2008-06, Vol.15 (6), p.1063-1072

2008

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The Bcl-2 repertoire of mesothelioma spheroids underlies acquired apoptotic multicellular resistance

Cell death & disease, 2011-06, Vol.2 (6), p.e174-e174

2011

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715: BCL-2 dependence in T-cell leukemia is defined by the maturation stage of the clone of origin

European journal of cancer (1990), 2014-07, Vol.50, p.S172-S172

2014

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P310: BH3 PROFILING IDENTIFIES SELECTIVE BCL‐2 DEPENDENCE OF ADULT EARLY T‐CCELL PROGENITOR AND TYPICAL ACUTE LYMPHOBLASTIC LEUKEMIA PATIENTS

HemaSphere, 2022-06, Vol.6, p.211-212

2022

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427 The mitochondrial basis of resistance to BCL-2 antagonists and conventional chemotherapy

European journal of cancer (1990), 2012-11, Vol.48, p.133-133

2012

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Bcl-xL controls a switch between cell death modes during mitotic arrest

Cell death & disease, 2014-10, Vol.5 (10), p.e1429-e1429

2014

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BH3 peptidomimetics potently activate apoptosis and demonstrate single agent efficacy in neuroblastoma

Oncogene, 2006-08, Vol.25 (33), p.4525-4533

2006

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BCL-2: found bound and drugged

Trends in molecular medicine, 2005-10, Vol.11 (10), p.442-444

Letai, Anthony

2005

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Distinct BH3 domains either sensitize or activate mitochondrial apoptosis, serving as prototype cancer therapeutics

Cancer cell, 2002-09, Vol.2 (3), p.183-192

2002

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Harnessing apoptosis to kill breast cancer cells

Cancer research (Chicago, Ill.), 2009-01, Vol.69 (2_Supplement), p.ES-2

Letai, A

2009

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Growth Factor Withdrawal and Apoptosis: The Middle Game

Molecular cell, 2006-03, Vol.21 (6), p.728-730

Letai, Anthony

2006

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Features of poorly primed apoptotic subpopulations identified using functional measurements of apoptotic priming and multiplexed immunofluorescence on single cells

European journal of cancer (1990), 2022-10, Vol.174, p.S51-S52

2022

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Bruton's tyrosine kinase inhibition increases BCL-2 dependence and enhances sensitivity to venetoclax in chronic lymphocytic leukemia

Mimicking the BH3 domain to kill cancer cells

Pharmacological manipulation of Bcl-2 family members to control cell death

BH3 profiling identifies heterogeneous dependency on Bcl-2 family members in multiple myeloma and predicts sensitivity to BH3 mimetics

p53 regulates a non-apoptotic death induced by ROS

Displacement of Bim by Bmf and Puma rather than increase in Bim level mediates paclitaxel-induced apoptosis in breast cancer cells

Decreased mitochondrial priming determines chemoresistance of colon cancer stem cells

A novel orally active proteasome inhibitor induces apoptosis in multiple myeloma cells with mechanisms distinct from Bortezomib

Activity of a selective inhibitor of nuclear export, selinexor (KPT-330), against AML-initiating cells engrafted into immunosuppressed NSG mice

Parkin selectively alters the intrinsic threshold for mitochondrial cytochrome c release

BH3 domains define selective inhibitory interactions with BHRF-1 and KSHV BCL-2

Bcl-xL controls a switch between cell death modes during mitotic arrest

Mitochondria—Judges and Executioners of Cell Death Sentences

BIM and other BCL-2 family proteins exhibit cross-species conservation of function between zebrafish and mammals

The Bcl-2 repertoire of mesothelioma spheroids underlies acquired apoptotic multicellular resistance

715: BCL-2 dependence in T-cell leukemia is defined by the maturation stage of the clone of origin

P310: BH3 PROFILING IDENTIFIES SELECTIVE BCL‐2 DEPENDENCE OF ADULT EARLY T‐CCELL PROGENITOR AND TYPICAL ACUTE LYMPHOBLASTIC LEUKEMIA PATIENTS

427 The mitochondrial basis of resistance to BCL-2 antagonists and conventional chemotherapy

Bcl-xL controls a switch between cell death modes during mitotic arrest

BH3 peptidomimetics potently activate apoptosis and demonstrate single agent efficacy in neuroblastoma

BCL-2: found bound and drugged

Distinct BH3 domains either sensitize or activate mitochondrial apoptosis, serving as prototype cancer therapeutics

Harnessing apoptosis to kill breast cancer cells

Growth Factor Withdrawal and Apoptosis: The Middle Game

Features of poorly primed apoptotic subpopulations identified using functional measurements of apoptotic priming and multiplexed immunofluorescence on single cells

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