UB Paderborn / Katalog / Suche / in CDI

Sortiert nach

Einfacher Volltextzugriff von (fast) überall

Installieren Sie die LibKey Nomad-Browsererweiterung und erhalten Sie von vielen Webseiten einfachen Zugriff auf lizenzierte Volltexte oder Open Access Publikationen. Mehr erfahren...

Soluble Aβ oligomer production and toxicity

Journal of neurochemistry, 2012-01, Vol.120 (s1), p.125-139

2012

Volltextzugriff (PDF)

Brain amyloid-β oligomers in ageing and Alzheimer's disease

Brain (London, England : 1878), 2013-05, Vol.136 (Pt 5), p.1383-1398

2013

Volltextzugriff (PDF)

Soluble α-synuclein is a novel modulator of Alzheimer's disease pathophysiology

The Journal of neuroscience, 2012-07, Vol.32 (30), p.10253-10266

2012

Volltextzugriff (PDF)

Soluble endogenous oligomeric α-synuclein species in neurodegenerative diseases: Expression, spreading, and cross-talk

Journal of Parkinson's disease, 2020-01, Vol.10 (3), p.791-818

2020

Volltextzugriff (PDF)

The Complex PrPc-Fyn Couples Human Oligomeric A beta with Pathological Tau Changes in Alzheimer's Disease

The Journal of neuroscience, 2012-11, Vol.32 (47), p.16857-16871

2012

Volltextzugriff (PDF)

Tau is required for progressive synaptic and memory deficits in a transgenic mouse model of α-synucleinopathy

Acta neuropathologica, 2019-10, Vol.138 (4), p.551-574

2019

Volltextzugriff (PDF)

Soluble Conformers of Aβ and Tau Alter Selective Proteins Governing Axonal Transport

The Journal of neuroscience, 2016-09, Vol.36 (37), p.9647-9658

2016

Volltextzugriff (PDF)

The complex PrP(c)-Fyn couples human oligomeric Aβ with pathological tau changes in Alzheimer's disease

The Journal of neuroscience, 2012-11, Vol.32 (47), p.16857

2012

Volltextzugriff (PDF)

Genetic modulation of soluble Aβ rescues cognitive and synaptic impairment in a mouse model of Alzheimer's disease

The Journal of neuroscience, 2014-06, Vol.34 (23), p.7871-7885

2014

Volltextzugriff (PDF)

Comment on "Activation of the Omega-3 Fatty Acid Receptor GPR120 Protects against Focal Cerebral Ischemic Injury by Preventing Inflammation and Apoptosis in Mice"

The Journal of immunology (1950), 2022-10, Vol.209 (7), p.1229-1233

2022

Volltextzugriff (PDF)

Gain-of-function mutations in protein kinase Cα (PKCα) may promote synaptic defects in Alzheimer's disease

Science signaling, 2016-05, Vol.9 (427), p.ra47

2016

Volltextzugriff (PDF)

Toxic oligomer species of amyloid-β in Alzheimer's disease, a timing issue

Swiss medical weekly, 2014-11, Vol.144, p.w14021

Lesne, Sylvain E

2014

Volltextzugriff (PDF)

Selective lowering of synapsins induced by oligomeric α-synuclein exacerbates memory deficits

Proceedings of the National Academy of Sciences - PNAS, 2017-06, Vol.114 (23), p.E4648-E4657

2017

Volltextzugriff (PDF)

Bidirectional modulation of Alzheimer phenotype by alpha-synuclein in mice and primary neurons

Acta neuropathologica, 2018-10, Vol.136 (4), p.589-605

2018

Volltextzugriff (PDF)

A specific amyloid-β protein assembly in the brain impairs memory

Nature, 2006-03, Vol.440 (7082), p.352-357

2006

Volltextzugriff (PDF)

Breaking the Code of Amyloid-β Oligomers

International Journal of Cell Biology, 2013, Vol.2013 (2013), p.659-664

2013

Volltextzugriff (PDF)

SNCA genetic lowering reveals differential cognitive function of alpha-synuclein dependent on sex

Acta neuropathologica communications, 2022-12, Vol.10 (1), p.180-180, Article 180

2022

Volltextzugriff (PDF)

Accelerating Amyloid-β Fibrillization Reduces Oligomer Levels and Functional Deficits in Alzheimer Disease Mouse Models

The Journal of biological chemistry, 2007-08, Vol.282 (33), p.23818-23828

2007

Volltextzugriff (PDF)

Correction: SNCA genetic lowering reveals differential cognitive function of alpha-synuclein dependent on sex

Acta neuropathologica communications, 2024-06, Vol.12 (1), p.92-3

2024

Volltextzugriff (PDF)

Cognitive effects of cell-derived and synthetically derived Aβ oligomers

Neurobiology of aging, 2011-10, Vol.32 (10), p.1784-1794

2011

Volltextzugriff (PDF)

The amyloid-β oligomer Aβ56 induces specific alterations in neuronal signaling that lead to tau phosphorylation and aggregation

Science signaling, 2017-05, Vol.10 (478)

2017

Volltextzugriff (PDF)

Involvement of β-site APP cleaving enzyme 1 (BACE1) in amyloid precursor protein-mediated enhancement of memory and activity-dependent synaptic plasticity

Proceedings of the National Academy of Sciences - PNAS, 2007-05, Vol.104 (19), p.8167-8172

2007

Volltextzugriff (PDF)

NMDA receptor activation inhibits alpha-secretase and promotes neuronal amyloid-beta production

The Journal of neuroscience, 2005-10, Vol.25 (41), p.9367-9377

2005

Volltextzugriff (PDF)

Retraction Note: A specific amyloid-β protein assembly in the brain impairs memory

Nature (London), 2024-07, Vol.631 (8019), p.240-240

2024

Volltextzugriff (PDF)

1 – 25 von 86

Suche als RSS-Feed

Suchergebnisse filtern

Filter anzeigen

Nur zeigen

Geprüft (Peer-Review)
(83)
Mit Volltextzugriff
(77)

Publikationsform

Artikel
(86)

Sprache

Englisch
(84)
Französisch
(2)

Erscheinungsjahr

n.n

Thema

15 weitere anzeigen...

Weniger anzeigen...

Neu hinzugefügt

... seit 90 Tagen
(1)

Menü

Weitere Dienste

Einstellungen

Amyloid Oligomers: A Joint Experimental/Computational Perspective on Alzheimer’s Disease, Parkinson’s Disease, Type II Diabetes, and Amyotrophic Lateral Sclerosis

Soluble Aβ oligomer production and toxicity

Brain amyloid-β oligomers in ageing and Alzheimer's disease

Soluble α-synuclein is a novel modulator of Alzheimer's disease pathophysiology

Soluble endogenous oligomeric α-synuclein species in neurodegenerative diseases: Expression, spreading, and cross-talk

The Complex PrPc-Fyn Couples Human Oligomeric A beta with Pathological Tau Changes in Alzheimer's Disease

Tau is required for progressive synaptic and memory deficits in a transgenic mouse model of α-synucleinopathy

Soluble Conformers of Aβ and Tau Alter Selective Proteins Governing Axonal Transport

The complex PrP(c)-Fyn couples human oligomeric Aβ with pathological tau changes in Alzheimer's disease

Genetic modulation of soluble Aβ rescues cognitive and synaptic impairment in a mouse model of Alzheimer's disease

Comment on "Activation of the Omega-3 Fatty Acid Receptor GPR120 Protects against Focal Cerebral Ischemic Injury by Preventing Inflammation and Apoptosis in Mice"

Gain-of-function mutations in protein kinase Cα (PKCα) may promote synaptic defects in Alzheimer's disease

Toxic oligomer species of amyloid-β in Alzheimer's disease, a timing issue

Selective lowering of synapsins induced by oligomeric α-synuclein exacerbates memory deficits

Bidirectional modulation of Alzheimer phenotype by alpha-synuclein in mice and primary neurons

A specific amyloid-β protein assembly in the brain impairs memory

Breaking the Code of Amyloid-β Oligomers

SNCA genetic lowering reveals differential cognitive function of alpha-synuclein dependent on sex

Accelerating Amyloid-β Fibrillization Reduces Oligomer Levels and Functional Deficits in Alzheimer Disease Mouse Models

Correction: SNCA genetic lowering reveals differential cognitive function of alpha-synuclein dependent on sex

Cognitive effects of cell-derived and synthetically derived Aβ oligomers

The amyloid-β oligomer Aβ56 induces specific alterations in neuronal signaling that lead to tau phosphorylation and aggregation

Involvement of β-site APP cleaving enzyme 1 (BACE1) in amyloid precursor protein-mediated enhancement of memory and activity-dependent synaptic plasticity

NMDA receptor activation inhibits alpha-secretase and promotes neuronal amyloid-beta production

Retraction Note: A specific amyloid-β protein assembly in the brain impairs memory

Suchergebnisse filtern