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Persistence and toxin production by Clostridium difficile within human intestinal organoids result in disruption of epithelial paracellular barrier function

Infection and immunity, 2015-01, Vol.83 (1), p.138-145

2015

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IL-33 drives group 2 innate lymphoid cell-mediated protection during Clostridium difficile infection

Nature communications, 2019-06, Vol.10 (1), p.2712-13, Article 2712

2019

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Host Protective Mechanisms to Intestinal Amebiasis

Trends in parasitology, 2021-02, Vol.37 (2), p.165-175

2021

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The Gut Bacterial Community Potentiates Clostridioides difficile Infection Severity

mBio, 2022-08, Vol.13 (4), p.e0118322-e0118322

2022

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Investigating the impact of antibiotic-induced dysbiosis on protection from Clostridium difficile colitis by mouse colonic innate lymphoid cells

mBio, 2024-03, Vol.15 (3), p.e0333823

2024

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The IL-33-ILC2 pathway protects from amebic colitis

Mucosal immunology, 2022-01, Vol.15 (1), p.165-175

2022

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Protection from Lethal Clostridioides difficile Infection via Intraspecies Competition for Cogerminant

mBio, 2021-03, Vol.12 (2)

2021

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Optimizing a Multi-Component Intranasal Entamoeba Histolytica Vaccine Formulation Using a Design of Experiments Strategy

Frontiers in immunology, 2021-06, Vol.12, p.683157-683157

2021

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The Gut Microbiota Is Associated with Clearance of Clostridium difficile Infection Independent of Adaptive Immunity

mSphere, 2019-01, Vol.4 (1)

2019

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Novel Drivers of Virulence in Clostridioides difficile Identified via Context-Specific Metabolic Network Analysis

mSystems, 2021-10, Vol.6 (5), p.e0091921

2021

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Overdiagnosis of Clostridium difficile Infection in the Molecular Test Era

JAMA internal medicine, 2015-11, Vol.175 (11), p.1792

2015

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Evaluation of Clostridium difficile Fecal Load and Limit of Detection during a Prospective Comparison of Two Molecular Tests, the illumigene C. difficile and Xpert C. difficile/Epi Tests

Journal of Clinical Microbiology, 2013-01, Vol.51 (1), p.278-280

2013

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Interleukin-22-mediated host glycosylation prevents Clostridioides difficile infection by modulating the metabolic activity of the gut microbiota

Nature medicine, 2020-04, Vol.26 (4), p.608-617

2020

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Colitis-Induced Th17 Cells Increase the Risk for Severe Subsequent Clostridium difficile Infection

Cell host & microbe, 2019-05, Vol.25 (5), p.756-765.e5

2019

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The Microbiome and Endothelial Function: An Investigative Fountain or Analytical Morass

Circulation research, 2018-10, Vol.123 (9), p.1015-1016

2018

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Gut microbiome communication with bone marrow regulates susceptibility to amebiasis

The Journal of clinical investigation, 2020-08, Vol.130 (8), p.4019-4024

2020

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Clostridium difficile Colonizes Alternative Nutrient Niches during Infection across Distinct Murine Gut Microbiomes

mSystems, 2017-07, Vol.2 (4)

2017

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Clostridium difficile Alters the Structure and Metabolism of Distinct Cecal Microbiomes during Initial Infection To Promote Sustained Colonization

mSphere, 2018-06, Vol.3 (3)

2018

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The rest of the story: the microbiome and gastrointestinal infections

Current opinion in microbiology, 2015-02, Vol.23, p.121-125

2015

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Lewis Blood-group Antigens Are Associated With Altered Susceptibility to Shigellosis

Clinical infectious diseases, 2021-06, Vol.72 (11), p.e868-e871

2021

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A whole new ball game: Stem cell-derived epithelia in the study of host–microbe interactions

Anaerobe, 2016-02, Vol.37, p.25-28

2016

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Binary Toxin Expression by Clostridioides difficile Is Associated With Worse Disease

Open forum infectious diseases, 2022-03, Vol.9 (3), p.ofac001-ofac001

2022

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The sympathetic nervous system drives hyperinflammatory responses to Clostridioides difficile infection

Cell reports. Medicine, 2024-10, Vol.5 (10), p.101771, Article 101771

2024

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Outcomes in patients tested for Clostridium difficile toxins

Diagnostic microbiology and infectious disease, 2012-12, Vol.74 (4), p.369-373

2012

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Novel Drivers of Virulence in Clostridioides difficile Identified via Context-Specific Metabolic Network Analysis

mSystems, 2021-10, Vol.6 (5)

2021

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Persistence and toxin production by Clostridium difficile within human intestinal organoids result in disruption of epithelial paracellular barrier function

IL-33 drives group 2 innate lymphoid cell-mediated protection during Clostridium difficile infection

Host Protective Mechanisms to Intestinal Amebiasis

The Gut Bacterial Community Potentiates Clostridioides difficile Infection Severity

Investigating the impact of antibiotic-induced dysbiosis on protection from Clostridium difficile colitis by mouse colonic innate lymphoid cells

The IL-33-ILC2 pathway protects from amebic colitis

Protection from Lethal Clostridioides difficile Infection via Intraspecies Competition for Cogerminant

Optimizing a Multi-Component Intranasal Entamoeba Histolytica Vaccine Formulation Using a Design of Experiments Strategy

The Gut Microbiota Is Associated with Clearance of Clostridium difficile Infection Independent of Adaptive Immunity

Novel Drivers of Virulence in Clostridioides difficile Identified via Context-Specific Metabolic Network Analysis

Overdiagnosis of Clostridium difficile Infection in the Molecular Test Era

Evaluation of Clostridium difficile Fecal Load and Limit of Detection during a Prospective Comparison of Two Molecular Tests, the illumigene C. difficile and Xpert C. difficile/Epi Tests

Interleukin-22-mediated host glycosylation prevents Clostridioides difficile infection by modulating the metabolic activity of the gut microbiota

Colitis-Induced Th17 Cells Increase the Risk for Severe Subsequent Clostridium difficile Infection

The Microbiome and Endothelial Function: An Investigative Fountain or Analytical Morass

Gut microbiome communication with bone marrow regulates susceptibility to amebiasis

Clostridium difficile Colonizes Alternative Nutrient Niches during Infection across Distinct Murine Gut Microbiomes

Clostridium difficile Alters the Structure and Metabolism of Distinct Cecal Microbiomes during Initial Infection To Promote Sustained Colonization

The rest of the story: the microbiome and gastrointestinal infections

Lewis Blood-group Antigens Are Associated With Altered Susceptibility to Shigellosis

A whole new ball game: Stem cell-derived epithelia in the study of host–microbe interactions

Binary Toxin Expression by Clostridioides difficile Is Associated With Worse Disease

The sympathetic nervous system drives hyperinflammatory responses to Clostridioides difficile infection

Outcomes in patients tested for Clostridium difficile toxins

Novel Drivers of Virulence in Clostridioides difficile Identified via Context-Specific Metabolic Network Analysis

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