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Contact inhibition and high cell density deactivate the mammalian target of rapamycin pathway, thus suppressing the senescence program

Proceedings of the National Academy of Sciences - PNAS, 2014-06, Vol.111 (24), p.8832-8837

2014

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CDK4/6-inhibiting drug substitutes for p21 and p16 in senescence: Duration of cell cycle arrest and MTOR activity determine geroconversion

Cell cycle (Georgetown, Tex.), 2013-09, Vol.12 (18), p.3063-3069

2013

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Hypoxia suppresses conversion from proliferative arrest to cellular senescence

Proceedings of the National Academy of Sciences - PNAS, 2012-08, Vol.109 (33), p.13314-13318

2012

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Weak p53 permits senescence during cell cycle arrest

Cell cycle (Georgetown, Tex.), 2010-11, Vol.9 (21), p.4323-4327

2010

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M(o)TOR of pseudo-hypoxic state in aging: Rapamycin to the rescue

Cell cycle (Georgetown, Tex.), 2014-02, Vol.13 (4), p.509-515

2014

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The choice between p53-induced senescence and quiescence is determined in part by the mTOR pathway

Aging (Albany, NY.), 2010-06, Vol.2 (6), p.344-352

2010

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Exploring long-term protection of normal human fibroblasts and epithelial cells from chemotherapy in cell culture

Oncotarget, 2011-03, Vol.2 (3), p.222-233

2011

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Suppression of replicative senescence by rapamycin in rodent embryonic cells

Cell cycle (Georgetown, Tex.), 2012-06, Vol.11 (12), p.2402-2407

2012

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Elimination of proliferating cells unmasks the shift from senescence to quiescence caused by rapamycin

PloS one, 2011-10, Vol.6 (10), p.e26126-e26126

2011

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Hypoxia and gerosuppression: The mTOR saga continues

Cell cycle (Georgetown, Tex.), 2012-11, Vol.11 (21), p.3926-3931

2012

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RNA-binding motif protein 35A is a novel tumor suppressor for colorectal cancer

Cell cycle (Georgetown, Tex.), 2009-02, Vol.8 (3), p.490-497

2009

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Gerosuppression by pan-mTOR inhibitors

Aging (Albany, NY.), 2016-12, Vol.8 (12), p.3535-3551

2016

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Weekly administration of rapamycin improves survival and biomarkers in obese male mice on high‐fat diet

Aging cell, 2014-08, Vol.13 (4), p.616-622

2014

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DNA damaging agents and p53 do not cause senescence in quiescent cells, while consecutive re-activation of mTOR is associated with conversion to senescence

Aging (Albany, NY.), 2010-12, Vol.2 (12), p.924-935

2010

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Hyper-mitogenic drive coexists with mitotic incompetence in senescent cells

Cell cycle (Georgetown, Tex.), 2012-12, Vol.11 (24), p.4642-4649

2012

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Yeast-like chronological senescence in mammalian cells: phenomenon, mechanism and pharmacological suppression

Aging (Albany, NY.), 2011-11, Vol.3 (11), p.1078-1091

2011

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Dual mTORC1/C2 inhibitors suppress cellular geroconversion (a senescence program)

Oncotarget, 2015-09, Vol.6 (27), p.23238-23248

2015

Volltextzugriff (PDF)

Immune checkpoint protein VSIG4 as a biomarker of aging in murine adipose tissue

Aging cell, 2020-10, Vol.19 (10), p.e13219-n/a

2020

Volltextzugriff (PDF)

Tumor promoter-induced cellular senescence: cell cycle arrest followed by geroconversion

Oncotarget, 2014-12, Vol.5 (24), p.12715-12727

2014

Volltextzugriff (PDF)

While reinforcing cell cycle arrest, rapamycin and Torins suppress senescence in UVA-irradiated fibroblasts

Oncotarget, 2017-12, Vol.8 (65), p.109848-109856

2017

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Rapamycin extends lifespan and delays tumorigenesis in heterozygous p53+/- mice

Aging (Albany, NY.), 2012-10, Vol.4 (10), p.709-714

2012

Volltextzugriff (PDF)

Involvement of the ERK Signaling Cascade in Protein Kinase C-mediated Cell Cycle Arrest in Intestinal Epithelial Cells

The Journal of biological chemistry, 2004-03, Vol.279 (10), p.9233-9247

2004

Volltextzugriff (PDF)

Comparison of rapamycin schedules in mice on high-fat diet

Cell cycle (Georgetown, Tex.), 2014-11, Vol.13 (21), p.3350-3356

2014

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Fasting levels of hepatic p-S6 are increased in old mice

Cell cycle (Georgetown, Tex.), 2014-09, Vol.13 (17), p.2656-2659

2014

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S6K in geroconversion

Cell cycle (Georgetown, Tex.), 2013-10, Vol.12 (20), p.3249-3252

2013

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Contact inhibition and high cell density deactivate the mammalian target of rapamycin pathway, thus suppressing the senescence program

CDK4/6-inhibiting drug substitutes for p21 and p16 in senescence: Duration of cell cycle arrest and MTOR activity determine geroconversion

Hypoxia suppresses conversion from proliferative arrest to cellular senescence

Weak p53 permits senescence during cell cycle arrest

M(o)TOR of pseudo-hypoxic state in aging: Rapamycin to the rescue

The choice between p53-induced senescence and quiescence is determined in part by the mTOR pathway

Exploring long-term protection of normal human fibroblasts and epithelial cells from chemotherapy in cell culture

Suppression of replicative senescence by rapamycin in rodent embryonic cells

Elimination of proliferating cells unmasks the shift from senescence to quiescence caused by rapamycin

Hypoxia and gerosuppression: The mTOR saga continues

RNA-binding motif protein 35A is a novel tumor suppressor for colorectal cancer

Gerosuppression by pan-mTOR inhibitors

Weekly administration of rapamycin improves survival and biomarkers in obese male mice on high‐fat diet

DNA damaging agents and p53 do not cause senescence in quiescent cells, while consecutive re-activation of mTOR is associated with conversion to senescence

Hyper-mitogenic drive coexists with mitotic incompetence in senescent cells

Yeast-like chronological senescence in mammalian cells: phenomenon, mechanism and pharmacological suppression

Dual mTORC1/C2 inhibitors suppress cellular geroconversion (a senescence program)

Immune checkpoint protein VSIG4 as a biomarker of aging in murine adipose tissue

Tumor promoter-induced cellular senescence: cell cycle arrest followed by geroconversion

While reinforcing cell cycle arrest, rapamycin and Torins suppress senescence in UVA-irradiated fibroblasts

Rapamycin extends lifespan and delays tumorigenesis in heterozygous p53+/- mice

Involvement of the ERK Signaling Cascade in Protein Kinase C-mediated Cell Cycle Arrest in Intestinal Epithelial Cells

Comparison of rapamycin schedules in mice on high-fat diet

Fasting levels of hepatic p-S6 are increased in old mice

S6K in geroconversion

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