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Cellular mechanisms of cardiomyopathy

The Journal of cell biology, 2011-08, Vol.194 (3), p.355-365

2011

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The Importance of Biological Sex and Estrogen in Rodent Models of Cardiovascular Health and Disease

Circulation research, 2016-04, Vol.118 (8), p.1294-1312

2016

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Cancer Causes Cardiac Atrophy and Autophagy in a Sexually Dimorphic Manner

Cancer research (Chicago, Ill.), 2011-03, Vol.71 (5), p.1710-1720

2011

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Biology of the cardiac myocyte in heart disease

Molecular biology of the cell, 2016-07, Vol.27 (14), p.2149-2160

2016

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A small-molecule inhibitor of sarcomere contractility suppresses hypertrophic cardiomyopathy in mice

Science (American Association for the Advancement of Science), 2016-02, Vol.351 (6273), p.617-621

2016

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PEG–Anthracene Hydrogels as an On‐Demand Stiffening Matrix To Study Mechanobiology

Angewandte Chemie (International ed.), 2019-07, Vol.58 (29), p.9912-9916

International ed. in English, 2019

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The TEL patch of telomere protein TPP1 mediates telomerase recruitment and processivity

Nature (London), 2012-12, Vol.492 (7428), p.285-289

2012

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Hydrogels preserve native phenotypes of valvular fibroblasts through an elasticity-regulated PI3K/AKT pathway

Proceedings of the National Academy of Sciences - PNAS, 2013-11, Vol.110 (48), p.19336-19341

2013

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miR-30 family microRNAs regulate myogenic differentiation and provide negative feedback on the microRNA pathway

PloS one, 2015-02, Vol.10 (2), p.e0118229-e0118229

2015

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Redirecting valvular myofibroblasts into dormant fibroblasts through light-mediated reduction in substrate modulus

PloS one, 2012-07, Vol.7 (7), p.e39969

2012

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Molecular consequences of the R453C hypertrophic cardiomyopathy mutation on human β-cardiac myosin motor function

Proceedings of the National Academy of Sciences - PNAS, 2013-07, Vol.110 (31), p.12607-12612

2013

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Uncoupling of Expression of an Intronic MicroRNA and Its Myosin Host Gene by Exon Skipping

Molecular and Cellular Biology, 2010-04, Vol.30 (8), p.1937-1945

2010

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Sex-based cardiac physiology

Annual review of physiology, 2009-01, Vol.71 (1), p.1-18

2009

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Roles of transforming growth factor‐β1 and OB‐cadherin in porcine cardiac valve myofibroblast differentiation

The FASEB journal, 2014-10, Vol.28 (10), p.4551-4562

2014

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Distinct cardiac transcriptional profiles defining pregnancy and exercise

PloS one, 2012-07, Vol.7 (7), p.e42297-e42297

2012

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Fatty Acids Identified in the Burmese Python Promote Beneficial Cardiac Growth

Science (American Association for the Advancement of Science), 2011-10, Vol.334 (6055), p.528-531

2011

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Characterization of cell subpopulations expressing progenitor cell markers in porcine cardiac valves

PloS one, 2013-07, Vol.8 (7), p.e69667-e69667

2013

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Ablation of lysophosphatidic acid receptor 1 attenuates hypertrophic cardiomyopathy in a mouse model

Proceedings of the National Academy of Sciences - PNAS, 2022-07, Vol.119 (28), p.e2204174119-e2204174119

2022

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Defining the Cardiac Fibroblast Secretome in a Fibrotic Microenvironment

Journal of the American Heart Association, 2020-10, Vol.9 (19), p.e017025-e017025

2020

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Identification of sequence changes in myosin II that adjust muscle contraction velocity

PLoS biology, 2021-06, Vol.19 (6), p.e3001248-e3001248

2021

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Drug specificity and affinity are encoded in the probability of cryptic pocket opening in myosin motor domains

eLife, 2023-01, Vol.12

2023

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Skip residues modulate the structural properties of the myosin rod and guide thick filament assembly

Proceedings of the National Academy of Sciences - PNAS, 2015-07, Vol.112 (29), p.E3806-E3815

2015

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Estrogens Mediate Cardiac Hypertrophy in a Stimulus-Dependent Manner

Endocrinology (Philadelphia), 2012-09, Vol.153 (9), p.4480-4490

2012

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Modulating fast skeletal muscle contraction protects skeletal muscle in animal models of Duchenne muscular dystrophy

The Journal of clinical investigation, 2023-05, Vol.133 (10), p.1-14

2023

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Cardiac Fibroblasts Mediate a Sexually Dimorphic Fibrotic Response to β-Adrenergic Stimulation

Journal of the American Heart Association, 2021-06, Vol.10 (11), p.e018876-e018876

2021

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Cellular mechanisms of cardiomyopathy

The Importance of Biological Sex and Estrogen in Rodent Models of Cardiovascular Health and Disease

Cancer Causes Cardiac Atrophy and Autophagy in a Sexually Dimorphic Manner

Biology of the cardiac myocyte in heart disease

A small-molecule inhibitor of sarcomere contractility suppresses hypertrophic cardiomyopathy in mice

PEG–Anthracene Hydrogels as an On‐Demand Stiffening Matrix To Study Mechanobiology

The TEL patch of telomere protein TPP1 mediates telomerase recruitment and processivity

Hydrogels preserve native phenotypes of valvular fibroblasts through an elasticity-regulated PI3K/AKT pathway

miR-30 family microRNAs regulate myogenic differentiation and provide negative feedback on the microRNA pathway

Redirecting valvular myofibroblasts into dormant fibroblasts through light-mediated reduction in substrate modulus

Molecular consequences of the R453C hypertrophic cardiomyopathy mutation on human β-cardiac myosin motor function

Uncoupling of Expression of an Intronic MicroRNA and Its Myosin Host Gene by Exon Skipping

Sex-based cardiac physiology

Roles of transforming growth factor‐β1 and OB‐cadherin in porcine cardiac valve myofibroblast differentiation

Distinct cardiac transcriptional profiles defining pregnancy and exercise

Fatty Acids Identified in the Burmese Python Promote Beneficial Cardiac Growth

Characterization of cell subpopulations expressing progenitor cell markers in porcine cardiac valves

Ablation of lysophosphatidic acid receptor 1 attenuates hypertrophic cardiomyopathy in a mouse model

Defining the Cardiac Fibroblast Secretome in a Fibrotic Microenvironment

Identification of sequence changes in myosin II that adjust muscle contraction velocity

Drug specificity and affinity are encoded in the probability of cryptic pocket opening in myosin motor domains

Skip residues modulate the structural properties of the myosin rod and guide thick filament assembly

Estrogens Mediate Cardiac Hypertrophy in a Stimulus-Dependent Manner

Modulating fast skeletal muscle contraction protects skeletal muscle in animal models of Duchenne muscular dystrophy

Cardiac Fibroblasts Mediate a Sexually Dimorphic Fibrotic Response to β-Adrenergic Stimulation

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