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Neurotensin stimulates sortilin and mTOR in human microglia inhibitable by methoxyluteolin, a potential therapeutic target for autism

Proceedings of the National Academy of Sciences - PNAS, 2016-11, Vol.113 (45), p.E7049-E7058

2016

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SP and IL-33 together markedly enhance TNF synthesis and secretion from human mast cells mediated by the interaction of their receptors

Proceedings of the National Academy of Sciences - PNAS, 2017-05, Vol.114 (20), p.E4002-E4009

2017

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Neurokinin-1 receptor: functional significance in the immune system in reference to selected infections and inflammation

Annals of the New York Academy of Sciences, 2011-01, Vol.1217 (1), p.83-95

2011

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Substance P and IL-33 administered together stimulate a marked secretion of IL-1β from human mast cells, inhibited by methoxyluteolin

Proceedings of the National Academy of Sciences - PNAS, 2018-10, Vol.115 (40), p.E9381-E9390

2018

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IL-38 inhibits microglial inflammatory mediators and is decreased in amygdala of children with autism spectrum disorder

Proceedings of the National Academy of Sciences - PNAS, 2020-07, Vol.117 (28), p.16475-16480

2020

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IL-33 augments substance P-induced VEGF secretion from human mast cells and is increased in psoriatic skin

Proceedings of the National Academy of Sciences - PNAS, 2010-03, Vol.107 (9), p.4448-4453

2010

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IL-37 is increased in brains of children with autism spectrum disorder and inhibits human microglia stimulated by neurotensin

Proceedings of the National Academy of Sciences - PNAS, 2019-10, Vol.116 (43), p.21659-21665

2019

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Insulin stimulates the cleavage and release of the extracellular domain of Klotho by ADAM10 and ADAM17

Proceedings of the National Academy of Sciences - PNAS, 2007-12, Vol.104 (50), p.19796-19801

2007

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Effect of IL-33 on de novo synthesized mediators from human mast cells

Journal of allergy and clinical immunology, 2019-01, Vol.143 (1), p.451-451

2019

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Human mast cell degranulation and preformed TNF secretion require mitochondrial translocation to exocytosis sites: Relevance to atopic dermatitis

Journal of allergy and clinical immunology, 2011-06, Vol.127 (6), p.1522-1531.e8

2011

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Diet-induced obesity in mice causes changes in immune responses and bone loss manifested by bacterial challenge

Proceedings of the National Academy of Sciences - PNAS, 2007-12, Vol.104 (51), p.20466-20471

2007

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Pivotal role of NOD2 in inflammatory processes affecting atherosclerosis and periodontal bone loss

Proceedings of the National Academy of Sciences - PNAS, 2013-12, Vol.110 (52), p.E5059-E5068

2013

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Is substance P a nasal epithelial neuropeptide?

Journal of allergy and clinical immunology, 2018-11, Vol.142 (5), p.1677-1677

2018

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Signaling mechanisms in the restoration of impaired immune function due to diet-induced obesity

Proceedings of the National Academy of Sciences - PNAS, 2011-02, Vol.108 (7), p.2867-2872

2011

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Signaling Mechanisms Involved in Altered Function of Macrophages from Diet-Induced Obese Mice Affect Immune Responses

Proceedings of the National Academy of Sciences - PNAS, 2009-06, Vol.106 (26), p.10740-10745

2009

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LPS-Induced TNF-α Factor (LITAF)-Deficient Mice Express Reduced LPS-Induced Cytokine: Evidence for LITAF-Dependent LPS Signaling Pathways

Proceedings of the National Academy of Sciences - PNAS, 2006-09, Vol.103 (37), p.13777-13782

2006

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Substance P induces rapid and transient membrane blebbing in U373MG cells in a p21-activated kinase-dependent manner

PloS one, 2011-09, Vol.6 (9), p.e25332-e25332

2011

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BMP9 ameliorates amyloidosis and the cholinergic defect in a mouse model of Alzheimer’s disease

Proceedings of the National Academy of Sciences - PNAS, 2013-11, Vol.110 (48), p.19567-19572

2013

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Whole-body deletion of LPS-induced TNF-α factor (LITAF) markedly improves experimental endotoxic shock and inflammatory arthritis

Proceedings of the National Academy of Sciences - PNAS, 2011-12, Vol.108 (52), p.21247-21252

2011

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REPLY TO FATTORI ET AL: Action of SP and IL-33 on mast cells

Proceedings of the National Academy of Sciences - PNAS, 2017-11, Vol.114 (47), p.E10036-E10036

2017

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LPS Induces the Interaction of a Transcription Factor, LPS-Induced TNF-α Factor, and STAT6(B) with Effects on Multiple Cytokines

Proceedings of the National Academy of Sciences - PNAS, 2005-04, Vol.102 (14), p.5132-5137

2005

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Statins (HMG-CoA reductase inhibitors) decrease postoperative adhesions by increasing peritoneal fibrinolytic activity

Annals of surgery, 2007-02, Vol.245 (2), p.176-184

2007

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Truncated neurokinin-1 receptor is increased in colonic epithelial cells from patients with colitis-associated cancer

Proceedings of the National Academy of Sciences - PNAS, 2011-10, Vol.108 (42), p.17420-17425

2011

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Differences in the length of the carboxyl terminus mediate functional properties of neurokinin-1 receptor

Proceedings of the National Academy of Sciences - PNAS, 2008-08, Vol.105 (34), p.12605-12610

2008

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LITAF mediation of increased TNF-α secretion from inflamed colonic lamina propria macrophages

PloS one, 2011-09, Vol.6 (9), p.e25849

2011

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Neurotensin stimulates sortilin and mTOR in human microglia inhibitable by methoxyluteolin, a potential therapeutic target for autism

SP and IL-33 together markedly enhance TNF synthesis and secretion from human mast cells mediated by the interaction of their receptors

Neurokinin-1 receptor: functional significance in the immune system in reference to selected infections and inflammation

Substance P and IL-33 administered together stimulate a marked secretion of IL-1β from human mast cells, inhibited by methoxyluteolin

IL-38 inhibits microglial inflammatory mediators and is decreased in amygdala of children with autism spectrum disorder

IL-33 augments substance P-induced VEGF secretion from human mast cells and is increased in psoriatic skin

IL-37 is increased in brains of children with autism spectrum disorder and inhibits human microglia stimulated by neurotensin

Insulin stimulates the cleavage and release of the extracellular domain of Klotho by ADAM10 and ADAM17

Effect of IL-33 on de novo synthesized mediators from human mast cells

Human mast cell degranulation and preformed TNF secretion require mitochondrial translocation to exocytosis sites: Relevance to atopic dermatitis

Diet-induced obesity in mice causes changes in immune responses and bone loss manifested by bacterial challenge

Pivotal role of NOD2 in inflammatory processes affecting atherosclerosis and periodontal bone loss

Is substance P a nasal epithelial neuropeptide?

Signaling mechanisms in the restoration of impaired immune function due to diet-induced obesity

Signaling Mechanisms Involved in Altered Function of Macrophages from Diet-Induced Obese Mice Affect Immune Responses

LPS-Induced TNF-α Factor (LITAF)-Deficient Mice Express Reduced LPS-Induced Cytokine: Evidence for LITAF-Dependent LPS Signaling Pathways

Substance P induces rapid and transient membrane blebbing in U373MG cells in a p21-activated kinase-dependent manner

BMP9 ameliorates amyloidosis and the cholinergic defect in a mouse model of Alzheimer’s disease

Whole-body deletion of LPS-induced TNF-α factor (LITAF) markedly improves experimental endotoxic shock and inflammatory arthritis

REPLY TO FATTORI ET AL: Action of SP and IL-33 on mast cells

LPS Induces the Interaction of a Transcription Factor, LPS-Induced TNF-α Factor, and STAT6(B) with Effects on Multiple Cytokines

Statins (HMG-CoA reductase inhibitors) decrease postoperative adhesions by increasing peritoneal fibrinolytic activity

Truncated neurokinin-1 receptor is increased in colonic epithelial cells from patients with colitis-associated cancer

Differences in the length of the carboxyl terminus mediate functional properties of neurokinin-1 receptor

LITAF mediation of increased TNF-α secretion from inflamed colonic lamina propria macrophages

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