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Caractérisation des miRNAs de GNAS pour essayer de mieux comprendre la physiopathologie de la pseudohypoparathyroïdie 1B (PHP1B)

Annales d'endocrinologie, 2016-09, Vol.77 (4), p.283-283

2016

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Caractérisation fonctionnelle de mutations de PDE4D responsables d’acrodysostose sans résistance hormonale

Annales d'endocrinologie, 2014-10, Vol.75 (5), p.298-298

2014

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Comparaison fonctionnelle de mutants PRKAR1A responsables de complexe de Carney et d’acrodysostose

Annales d'endocrinologie, 2013-09, Vol.74 (4), p.278-279

2013

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A common promoter variant of the leptin gene is associated with changes in the relationship between serum leptin and fat mass in obese girls

Diabetes (New York, N.Y.), 2000-12, Vol.49 (12), p.2196-2200

2000

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Absence of renal resistance to PTH in PDE4D-mutated acrodysostosis patients: Regulation of cAMP/PKA pathway by PDE4 may be tissue and agonist-specific – example of renal PTH signaling pathway

Annales d'endocrinologie, 2013-09, Vol.74 (4), p.294-294

2013

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Early changes in postprandial insulin secretion, not in insulin sensitivity, characterize juvenile obesity

Diabetes (New York, N.Y.), 1994-05, Vol.43 (5), p.696-702

1994

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The insulin gene VNTR is associated with fasting insulin levels and development of juvenile obesity

Nature genetics, 2000-12, Vol.26 (4), p.444-446

2000

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Glycerol production and utilization during the early phase of human obesity

Diabetes (New York, N.Y.), 1992-04, Vol.41 (4), p.444-450

1992

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Paternal transmission of the very common class I INS VNTR alleles predisposes to childhood obesity

Nature genetics, 2001-09, Vol.29 (1), p.96-99

2001

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The Common −866 G/A Polymorphism in the Promoter of Uncoupling Protein 2 Is Associated With Increased Carbohydrate and Decreased Lipid Oxidation in Juvenile Obesity

Diabetes (New York, N.Y.), 2004-01, Vol.53 (1), p.235-239

2004

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Molecular Genetics of Human Obesity-Associated MC4R Mutations

Annals of the New York Academy of Sciences, 2003-06, Vol.994 (1), p.49-57

2003

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Increased Insulin Resistance in Obese Children Who Have Both 972 IRS-1 and 1057 IRS-2 Polymorphisms

Diabetes (New York, N.Y.), 2002-12, Vol.51 (suppl 3), p.S304-S307

2002

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Time course of increased lipid and decreased glucose oxidation during early phase of childhood obesity

Diabetes (New York, N.Y.), 1993-07, Vol.42 (7), p.1010-1016

1993

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Heterogeneity of class I INS VNTR allele association with insulin secretion in obese children

Physiological genomics, 2006-05, Vol.25 (3), p.480-484

2006

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Time course of increased lipid and decreased glucose oxidation during early phase of childhood obesity

Diabetes (New York, N.Y.), 1993-07, Vol.42 (7), p.1010-1016

1993

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INS VNTR is a QTL for the insulin response to oral glucose in obese children

Physiological genomics, 2004-02, Vol.16 (3), p.309-313

2004

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Glycerol production and utilization during the early phase of human obesity

Diabetes (New York, N.Y.), 1992-04, Vol.41 (4), p.444-450

1992

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Growth hormone stimulates interferon regulatory factor-1 gene expression in the liver

Endocrinology (Philadelphia), 1998-03, Vol.139 (3), p.859-866

1998

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Acute nuclear actions of growth hormone (GH): cycloheximide inhibits inducible activator protein-1 activity, but does not block GH-regulated signal transducer and activator of transcription activation or gene expression

Endocrinology (Philadelphia), 1996-01, Vol.137 (1), p.55-64

1996

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Glycemic anomalies observed in obese adolescents

Journees annuelles de diabetologie de l'Hotel-Dieu, 2001, p.159

2001

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Precocious metabolic defects in juvenile obesity

Journees annuelles de diabetologie de l'Hotel-Dieu, 1995, p.147

1995

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Acrodysostosis syndromes

BoneKEy reports, 2012-11, Vol.1, p.225-225

2012

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Rapid activation of rat insulin-like growth factor-I gene transcription by growth hormone reveals no changes in deoxyribonucleic acid-protein interactions within the second promoter

Endocrinology (Philadelphia), 1995-05, Vol.136 (5), p.2230-2237

1995

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Contrasting acute in vivo nuclear actions of growth hormone and prolactin

Molecular and cellular endocrinology, 1996-08, Vol.121 (2), p.109-117

1996

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Early changes in postprandial insulin secretion, not in insulin sensitivity, characterize juvenile obesity

Diabetes (New York, N.Y.), 1994-05, Vol.43 (5), p.696-702

1994

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Caractérisation des miRNAs de GNAS pour essayer de mieux comprendre la physiopathologie de la pseudohypoparathyroïdie 1B (PHP1B)

Caractérisation fonctionnelle de mutations de PDE4D responsables d’acrodysostose sans résistance hormonale

Comparaison fonctionnelle de mutants PRKAR1A responsables de complexe de Carney et d’acrodysostose

A common promoter variant of the leptin gene is associated with changes in the relationship between serum leptin and fat mass in obese girls

Absence of renal resistance to PTH in PDE4D-mutated acrodysostosis patients: Regulation of cAMP/PKA pathway by PDE4 may be tissue and agonist-specific – example of renal PTH signaling pathway

Early changes in postprandial insulin secretion, not in insulin sensitivity, characterize juvenile obesity

The insulin gene VNTR is associated with fasting insulin levels and development of juvenile obesity

Glycerol production and utilization during the early phase of human obesity

Paternal transmission of the very common class I INS VNTR alleles predisposes to childhood obesity

The Common −866 G/A Polymorphism in the Promoter of Uncoupling Protein 2 Is Associated With Increased Carbohydrate and Decreased Lipid Oxidation in Juvenile Obesity

Molecular Genetics of Human Obesity-Associated MC4R Mutations

Increased Insulin Resistance in Obese Children Who Have Both 972 IRS-1 and 1057 IRS-2 Polymorphisms

Time course of increased lipid and decreased glucose oxidation during early phase of childhood obesity

Heterogeneity of class I INS VNTR allele association with insulin secretion in obese children

Time course of increased lipid and decreased glucose oxidation during early phase of childhood obesity

INS VNTR is a QTL for the insulin response to oral glucose in obese children

Glycerol production and utilization during the early phase of human obesity

Growth hormone stimulates interferon regulatory factor-1 gene expression in the liver

Acute nuclear actions of growth hormone (GH): cycloheximide inhibits inducible activator protein-1 activity, but does not block GH-regulated signal transducer and activator of transcription activation or gene expression

Glycemic anomalies observed in obese adolescents

Precocious metabolic defects in juvenile obesity

Acrodysostosis syndromes

Rapid activation of rat insulin-like growth factor-I gene transcription by growth hormone reveals no changes in deoxyribonucleic acid-protein interactions within the second promoter

Contrasting acute in vivo nuclear actions of growth hormone and prolactin

Early changes in postprandial insulin secretion, not in insulin sensitivity, characterize juvenile obesity

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