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Cells Release Prions in Association with Exosomes

Proceedings of the National Academy of Sciences - PNAS, 2004-06, Vol.101 (26), p.9683-9688

2004

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The physical relationship between infectivity and prion protein aggregates is strain-dependent

PLoS pathogens, 2010-04, Vol.6 (4), p.e1000859-e1000859

2010

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Sheep and goat BSE propagate more efficiently than cattle BSE in human PrP transgenic mice

PLoS pathogens, 2011-03, Vol.7 (3), p.e1001319-e1001319

2011

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Quaternary structure of pathological prion protein as a determining factor of strain-specific prion replication dynamics

PLoS pathogens, 2013-10, Vol.9 (10), p.e1003702-e1003702

2013

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Transmission of atypical bovine prions to mice transgenic for human prion protein

Emerging infectious diseases, 2008-12, Vol.14 (12), p.1898-1901

2008

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newly identified type of scrapie agent can naturally infect sheep with resistant PrP genotypes

Proceedings of the National Academy of Sciences - PNAS, 2005-11, Vol.102 (44), p.16031-16036

2005

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Atypical/Nor98 scrapie infectivity in sheep peripheral tissues

PLoS pathogens, 2011-02, Vol.7 (2), p.e1001285

2011

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Facilitated Cross-Species Transmission of Prions in Extraneural Tissue

Science (American Association for the Advancement of Science), 2012-01, Vol.335 (6067), p.472-475

2012

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Divergent prion strain evolution driven by PrPC expression level in transgenic mice

Nature communications, 2017-01, Vol.8 (1), p.14170-14170, Article 14170

2017

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Host prion protein expression levels impact prion tropism for the spleen

PLoS pathogens, 2020-07, Vol.16 (7), p.e1008283-e1008283

2020

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Prions Can Infect Primary Cultured Neurons and Astrocytes and Promote Neuronal Cell Death

Proceedings of the National Academy of Sciences - PNAS, 2004-08, Vol.101 (33), p.12271-12276

2004

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Marked influence of the route of infection on prion strain apparent phenotype in a scrapie transgenic mouse model

Neurobiology of disease, 2011-01, Vol.41 (1), p.219-225

2011

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Prominent and persistent extraneural infection in human PrP transgenic mice infected with variant CJD

PloS one, 2008-01, Vol.3 (1), p.e1419-e1419

2008

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Prion Propagation in Cells Expressing PrP Glycosylation Mutants

Journal of Virology, 2011-04, Vol.85 (7), p.3077-3085

2011

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Endogenous Proteolytic Cleavage of Disease-associated Prion Protein to Produce C2 Fragments Is Strongly Cell- and Tissue-dependent

The Journal of biological chemistry, 2010-04, Vol.285 (14), p.10252-10264

2010

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Highly infectious prions generated by a single round of microplate-based protein misfolding cyclic amplification

mBio, 2013-12, Vol.5 (1), p.e00829-e00813

2013

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Absence of Evidence for a Causal Link between Bovine Spongiform Encephalopathy Strain Variant L-BSE and Known Forms of Sporadic Creutzfeldt-Jakob Disease in Human PrP Transgenic Mice

Journal of virology, 2016-12, Vol.90 (23), p.10867-10874

2016

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Crossing Species Barriers Relies on Structurally Distinct Prion Assemblies and Their Complementation

Molecular neurobiology, 2020-06, Vol.57 (6), p.2572-2587

2020

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Glycoform-independent prion conversion by highly efficient, cell-based, protein misfolding cyclic amplification

Scientific reports, 2016-07, Vol.6 (1), p.29116-29116, Article 29116

2016

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Glycoform-selective prion formation in sporadic and familial forms of prion disease

PloS one, 2013-03, Vol.8 (3), p.e58786-e58786

2013

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Expression of Prion Protein Increases Cellular Copper Binding and Antioxidant Enzyme Activities but Not Copper Delivery

The Journal of biological chemistry, 2003-03, Vol.278 (11), p.9064-9072

2003

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Generating Bona Fide Mammalian Prions with Internal Deletions

Journal of virology, 2016-08, Vol.90 (15), p.6963-6975

2016

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Plasminogen-based capture combined with amplification technology for the detection of PrP(TSE) in the pre-clinical phase of infection

PloS one, 2013, Vol.8 (7), p.e69632-e69632

2013

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Prion Strain- and Species-Dependent Effects of Antiprion Molecules in Primary Neuronal Cultures

Journal of Virology, 2007-12, Vol.81 (24), p.13794-13800

2007

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Isolation from cattle of a prion strain distinct from that causing bovine spongiform encephalopathy

PLoS pathogens, 2006-10, Vol.2 (10), p.e112-e112

2006

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Cells Release Prions in Association with Exosomes

The physical relationship between infectivity and prion protein aggregates is strain-dependent

Sheep and goat BSE propagate more efficiently than cattle BSE in human PrP transgenic mice

Quaternary structure of pathological prion protein as a determining factor of strain-specific prion replication dynamics

Transmission of atypical bovine prions to mice transgenic for human prion protein

newly identified type of scrapie agent can naturally infect sheep with resistant PrP genotypes

Atypical/Nor98 scrapie infectivity in sheep peripheral tissues

Facilitated Cross-Species Transmission of Prions in Extraneural Tissue

Divergent prion strain evolution driven by PrPC expression level in transgenic mice

Host prion protein expression levels impact prion tropism for the spleen

Prions Can Infect Primary Cultured Neurons and Astrocytes and Promote Neuronal Cell Death

Marked influence of the route of infection on prion strain apparent phenotype in a scrapie transgenic mouse model

Prominent and persistent extraneural infection in human PrP transgenic mice infected with variant CJD

Prion Propagation in Cells Expressing PrP Glycosylation Mutants

Endogenous Proteolytic Cleavage of Disease-associated Prion Protein to Produce C2 Fragments Is Strongly Cell- and Tissue-dependent

Highly infectious prions generated by a single round of microplate-based protein misfolding cyclic amplification

Absence of Evidence for a Causal Link between Bovine Spongiform Encephalopathy Strain Variant L-BSE and Known Forms of Sporadic Creutzfeldt-Jakob Disease in Human PrP Transgenic Mice

Crossing Species Barriers Relies on Structurally Distinct Prion Assemblies and Their Complementation

Glycoform-independent prion conversion by highly efficient, cell-based, protein misfolding cyclic amplification

Glycoform-selective prion formation in sporadic and familial forms of prion disease

Expression of Prion Protein Increases Cellular Copper Binding and Antioxidant Enzyme Activities but Not Copper Delivery

Generating Bona Fide Mammalian Prions with Internal Deletions

Plasminogen-based capture combined with amplification technology for the detection of PrP(TSE) in the pre-clinical phase of infection

Prion Strain- and Species-Dependent Effects of Antiprion Molecules in Primary Neuronal Cultures

Isolation from cattle of a prion strain distinct from that causing bovine spongiform encephalopathy

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