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Nonalcoholic fatty liver disease: From steatosis to cirrhosis

Hepatology (Baltimore, Md.), 2006-02, Vol.43 (S1), p.S99-S112

2006

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Animal models of NASH: Getting both pathology and metabolic context right

Journal of gastroenterology and hepatology, 2008-11, Vol.23 (11), p.1635-1648

2008

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Hepatic Free Cholesterol Accumulates in Obese, Diabetic Mice and Causes Nonalcoholic Steatohepatitis

Gastroenterology (New York, N.Y. 1943), 2011-10, Vol.141 (4), p.1393-1403.e5

2011

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A fresh look at NASH pathogenesis. Part 1: The metabolic movers

Journal of gastroenterology and hepatology, 2010-04, Vol.25 (4), p.672-690

2010

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Insulin resistance, adiponectin, cytokines in NASH: Which is the best target to treat?

Journal of hepatology, 2006-02, Vol.44 (2), p.253-261

2006

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Strain dependence of diet-induced NASH and liver fibrosis in obese mice is linked to diabetes and inflammatory phenotype

Liver international, 2014-08, Vol.34 (7), p.1084-1093

2014

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Roles of adipose restriction and metabolic factors in progression of steatosis to steatohepatitis in obese, diabetic mice

Journal of gastroenterology and hepatology, 2009-10, Vol.24 (10), p.1658-1668

2009

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Dietary modification dampens liver inflammation and fibrosis in obesity‐related fatty liver disease

Obesity (Silver Spring, Md.), 2013-06, Vol.21 (6), p.1189-1199

2013

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Adaptive failure to high-fat diet characterizes steatohepatitis in Alms1 mutant mice

Biochemical and biophysical research communications, 2006-04, Vol.342 (4), p.1152-1159

2006

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Hepatic free fatty acids accumulate in experimental steatohepatitis: Role of adaptive pathways

Journal of hepatology, 2008-04, Vol.48 (4), p.638-647

2008

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Lipotoxicity: Why do saturated fatty acids cause and monounsaturates protect against it?

Journal of gastroenterology and hepatology, 2009-05, Vol.24 (5), p.703-706

2009

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Pharmacological cholesterol lowering reverses fibrotic NASH in obese, diabetic mice with metabolic syndrome

Journal of hepatology, 2013-07, Vol.59 (1), p.144-152

2013

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MCD-induced steatohepatitis is associated with hepatic adiponectin resistance and adipogenic transformation of hepatocytes

Journal of hepatology, 2008-09, Vol.49 (3), p.407-416

2008

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Peroxisome proliferator-activated receptor-α agonist, Wy 14 643, improves metabolic indices, steatosis and ballooning in diabetic mice with non-alcoholic steatohepatitis

Journal of gastroenterology and hepatology, 2012-02, Vol.27 (2), p.341-350

2012

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A truncating mutation of Alms1 reduces the number of hypothalamic neuronal cilia in obese mice

Developmental neurobiology (Hoboken, N.J.), 2013-01, Vol.73 (1), p.1-13

2013

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Apoptosis in experimental NASH is associated with p53 activation and TRAIL receptor expression

Journal of gastroenterology and hepatology, 2009-03, Vol.24 (3), p.443-452

2009

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Not all models of fatty liver are created equal: Understanding mechanisms of steatosis development is important

Journal of gastroenterology and hepatology, 2007-09, Vol.22 (9), p.1353-1354

Larter, Claire Z

2007

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Activation of peroxisome proliferator-activated receptor α by dietary fish oil attenuates steatosis, but does not prevent experimental steatohepatitis because of hepatic lipoperoxide accumulation

Journal of gastroenterology and hepatology, 2008-02, Vol.23 (2), p.267-275

2008

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Glutathione transferase kappa deficiency causes glomerular nephropathy without overt oxidative stress

Laboratory investigation, 2011-11, Vol.91 (11), p.1572-1583

2011

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Dietary modification dampens liver inflammation and fibrosis in obesity-related fatty liver disease: Dietary Modification Attenuates Fatty Liver

Obesity (Silver Spring, Md.), 2013-06, Vol.21 (6), p.1189-1199

2013

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Hepatic Free Cholesterol Accumulates in Obese, Diabetic Mice and Causes Non-Alcoholic Steatohepatitis

Gastroenterology (New York, N.Y. 1943), 2011-06, Vol.141 (4), p.1393-1403.e5

2011

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Obesity and diabetes accelerate hepatocarcinogenesis via hepatocyte proliferation independent of NF-κB or Akt/mTORC1

Journal of clinical and translational research, 2016-04, Vol.2 (1), p.26-37

2016

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Nonalcoholic fatty liver disease: From steatosis to cirrhosis

Animal models of NASH: Getting both pathology and metabolic context right

Hepatic Free Cholesterol Accumulates in Obese, Diabetic Mice and Causes Nonalcoholic Steatohepatitis

A fresh look at NASH pathogenesis. Part 1: The metabolic movers

Insulin resistance, adiponectin, cytokines in NASH: Which is the best target to treat?

Strain dependence of diet-induced NASH and liver fibrosis in obese mice is linked to diabetes and inflammatory phenotype

Roles of adipose restriction and metabolic factors in progression of steatosis to steatohepatitis in obese, diabetic mice

Dietary modification dampens liver inflammation and fibrosis in obesity‐related fatty liver disease

Adaptive failure to high-fat diet characterizes steatohepatitis in Alms1 mutant mice

Hepatic free fatty acids accumulate in experimental steatohepatitis: Role of adaptive pathways

Lipotoxicity: Why do saturated fatty acids cause and monounsaturates protect against it?

Pharmacological cholesterol lowering reverses fibrotic NASH in obese, diabetic mice with metabolic syndrome

MCD-induced steatohepatitis is associated with hepatic adiponectin resistance and adipogenic transformation of hepatocytes

Peroxisome proliferator-activated receptor-α agonist, Wy 14 643, improves metabolic indices, steatosis and ballooning in diabetic mice with non-alcoholic steatohepatitis

A truncating mutation of Alms1 reduces the number of hypothalamic neuronal cilia in obese mice

Apoptosis in experimental NASH is associated with p53 activation and TRAIL receptor expression

Not all models of fatty liver are created equal: Understanding mechanisms of steatosis development is important

Activation of peroxisome proliferator-activated receptor α by dietary fish oil attenuates steatosis, but does not prevent experimental steatohepatitis because of hepatic lipoperoxide accumulation

Glutathione transferase kappa deficiency causes glomerular nephropathy without overt oxidative stress

Dietary modification dampens liver inflammation and fibrosis in obesity-related fatty liver disease: Dietary Modification Attenuates Fatty Liver

Hepatic Free Cholesterol Accumulates in Obese, Diabetic Mice and Causes Non-Alcoholic Steatohepatitis

Obesity and diabetes accelerate hepatocarcinogenesis via hepatocyte proliferation independent of NF-κB or Akt/mTORC1

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