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The protein product of the c-cbl protooncogene is the 120-kDa tyrosine-phosphorylated protein in Jurkat cells activated via the T cell antigen receptor

The Journal of biological chemistry, 1994-09, Vol.269 (37), p.22921-22924

1994

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Cbl: many adaptations to regulate protein tyrosine kinases

Nature reviews. Molecular cell biology, 2001-04, Vol.2 (4), p.294-307

2001

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Association of a chromosome deletion syndrome with a fragile site within the proto-oncogene CBL2

Nature (London), 1995-07, Vol.376 (6536), p.145-149

1995

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Mutation of the c-Cbl TKB Domain Binding Site on the Met Receptor Tyrosine Kinase Converts It into a Transforming Protein

Molecular cell, 2001-11, Vol.8 (5), p.995-1004

2001

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v-cbl, an Oncogene from a Dual-Recombinant Murine Retrovirus that Induces Early B-Lineage Lymphomas

Proceedings of the National Academy of Sciences - PNAS, 1989-02, Vol.86 (4), p.1168-1172

1989

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c-Cbl/Sli-1 regulates endocytic sorting and ubiquitination of the epidermal growth factor receptor

Genes & development, 1998-12, Vol.12 (23), p.3663-3674

1998

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Phosphotyrosine binding domain-dependent upregulation of the platelet-derived growth factor receptor alpha signaling cascade by transforming mutants of Cbl: implications for Cbl's function and oncogenicity

Molecular and Cellular Biology, 1997-08, Vol.17 (8), p.4597-4610

1997

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Sprouty2 attenuates epidermal growth factor receptor ubiquitylation and endocytosis, and consequently enhances Ras/ERK signalling

The EMBO journal, 2002-09, Vol.21 (18), p.4796-4808

2002

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The truncation that generated the v‐cbl oncogene reveals an ability for nuclear transport, DNA binding and acute transformation

The EMBO journal, 1993-05, Vol.12 (5), p.2017-2026

1993

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EGF receptor binding and transformation by v-cbl is ablated by the introduction of a loss-of-function mutation from the Caenorhabditis elegans sli-1 gene

Oncogene, 1997-05, Vol.14 (18), p.2239-2249

1997

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Tyrosine kinase activity of the EGF receptor is enhanced by the expression of oncogenic 70Z-Cbl

Oncogene, 1997-12, Vol.15 (24), p.2909-2919

1997

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Tissue Hyperplasia and Enhanced T-Cell Signalling via ZAP-70 in c-Cbl-Deficient Mice

Molecular and Cellular Biology, 1998-08, Vol.18 (8), p.4872-4882

1998

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Physical linkage of the fragile site FRA11B and a Jacobsen syndrome chromosome deletion breakpoint in 11q23.3

Human molecular genetics, 1994-12, Vol.3 (12), p.2123-2130

1994

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The E mu-myc transgenic mouse. A model for high-incidence spontaneous lymphoma and leukemia of early B cells

The Journal of experimental medicine, 1988-02, Vol.167 (2), p.353-371

1988

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Fyn, Yes, and Syk Phosphorylation Sites in c-Cbl Map to the Same Tyrosine Residues That Become Phosphorylated in Activated T Cells

The Journal of biological chemistry, 1998-04, Vol.273 (14), p.8323-8331

1998

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The c-cbl proto-oncogene is preferentially expressed in thymus and testis tissue and encodes a nuclear protein

Journal of Virology, 1989-12, Vol.63 (12), p.5420-5424

1989

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Oncogenic forms of Cbl abrogate the anchorage requirement but not the growth factor requirement for proliferation

Oncogene, 1998-06, Vol.16 (24), p.3159-3167

1998

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Tumour induction by activated abl involves tyrosine phosphorylation of the product of the cbl oncogene

The EMBO journal, 1994-10, Vol.13 (19), p.4515-4523

1994

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c-Cbl localizes to actin lamellae and regulates lamellipodia formation and cell morphology

Journal of cell science, 2000-01, Vol.113 Pt 2 (2), p.215-226

2000

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The Adapter Type Protein CMS/CD2AP Binds to the Proto-oncogenic Protein c-Cbl through a Tyrosine Phosphorylation-regulated Src Homology 3 Domain Interaction

The Journal of biological chemistry, 2001-02, Vol.276 (7), p.4957-4963

2001

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Serine mutations that abrogate ligand-induced ubiquitination and internalization of the EGF receptor do not affect c-Cbl association with the receptor

Oncogene, 2003-11, Vol.22 (52), p.8509-8518

2003

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The Cbl Proto-Oncogene Product Negatively Regulates the Src-Family Tyrosine Kinase Fyn by Enhancing Its Degradation

Molecular and Cellular Biology, 2000-02, Vol.20 (3), p.851-867

2000

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A mouse with a loss-of-function mutation in the c-Cbl TKB domain shows perturbed thymocyte signaling without enhancing the activity of the ZAP-70 tyrosine kinase

The Journal of experimental medicine, 2003-02, Vol.197 (4), p.503-513

2003

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Perturbed Regulation of ZAP-70 and Sustained Tyrosine Phosphorylation of LAT and SLP-76 in c-Cbl-Deficient Thymocytes

The Journal of immunology (1950), 1999-06, Vol.162 (12), p.7133-7139

1999

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Stimulation through the T Cell Receptor Induces Cbl Association with Crk Proteins and the Guanine Nucleotide Exchange Protein C3G

The Journal of biological chemistry, 1996-04, Vol.271 (14), p.8435-8442

1996

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The protein product of the c-cbl protooncogene is the 120-kDa tyrosine-phosphorylated protein in Jurkat cells activated via the T cell antigen receptor

Cbl: many adaptations to regulate protein tyrosine kinases

Association of a chromosome deletion syndrome with a fragile site within the proto-oncogene CBL2

Mutation of the c-Cbl TKB Domain Binding Site on the Met Receptor Tyrosine Kinase Converts It into a Transforming Protein

v-cbl, an Oncogene from a Dual-Recombinant Murine Retrovirus that Induces Early B-Lineage Lymphomas

c-Cbl/Sli-1 regulates endocytic sorting and ubiquitination of the epidermal growth factor receptor

Phosphotyrosine binding domain-dependent upregulation of the platelet-derived growth factor receptor alpha signaling cascade by transforming mutants of Cbl: implications for Cbl's function and oncogenicity

Sprouty2 attenuates epidermal growth factor receptor ubiquitylation and endocytosis, and consequently enhances Ras/ERK signalling

The truncation that generated the v‐cbl oncogene reveals an ability for nuclear transport, DNA binding and acute transformation

EGF receptor binding and transformation by v-cbl is ablated by the introduction of a loss-of-function mutation from the Caenorhabditis elegans sli-1 gene

Tyrosine kinase activity of the EGF receptor is enhanced by the expression of oncogenic 70Z-Cbl

Tissue Hyperplasia and Enhanced T-Cell Signalling via ZAP-70 in c-Cbl-Deficient Mice

Physical linkage of the fragile site FRA11B and a Jacobsen syndrome chromosome deletion breakpoint in 11q23.3

The E mu-myc transgenic mouse. A model for high-incidence spontaneous lymphoma and leukemia of early B cells

Fyn, Yes, and Syk Phosphorylation Sites in c-Cbl Map to the Same Tyrosine Residues That Become Phosphorylated in Activated T Cells

The c-cbl proto-oncogene is preferentially expressed in thymus and testis tissue and encodes a nuclear protein

Oncogenic forms of Cbl abrogate the anchorage requirement but not the growth factor requirement for proliferation

Tumour induction by activated abl involves tyrosine phosphorylation of the product of the cbl oncogene

c-Cbl localizes to actin lamellae and regulates lamellipodia formation and cell morphology

The Adapter Type Protein CMS/CD2AP Binds to the Proto-oncogenic Protein c-Cbl through a Tyrosine Phosphorylation-regulated Src Homology 3 Domain Interaction

Serine mutations that abrogate ligand-induced ubiquitination and internalization of the EGF receptor do not affect c-Cbl association with the receptor

The Cbl Proto-Oncogene Product Negatively Regulates the Src-Family Tyrosine Kinase Fyn by Enhancing Its Degradation

A mouse with a loss-of-function mutation in the c-Cbl TKB domain shows perturbed thymocyte signaling without enhancing the activity of the ZAP-70 tyrosine kinase

Perturbed Regulation of ZAP-70 and Sustained Tyrosine Phosphorylation of LAT and SLP-76 in c-Cbl-Deficient Thymocytes

Stimulation through the T Cell Receptor Induces Cbl Association with Crk Proteins and the Guanine Nucleotide Exchange Protein C3G

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