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Sex Differences in Lung Cancer

Cancers, 2023-06, Vol.15 (12), p.3111

2023

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Epigenetic Regulation of Cancer in Response to Chemotherapy

1, 2023

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Non-Cell Autonomous Effects of the Senescence-Associated Secretory Phenotype in Cancer Therapy

Frontiers in oncology, 2018-05, Vol.8, p.164-164

2018

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Large-scale integration of DNA methylation and gene expression array platforms identifies both cis and trans relationships

Epigenetics, 2022-12, Vol.17 (12), p.1753-1773

2022

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Autophagy-Dependent Sensitization of Triple-Negative Breast Cancer Models to Topoisomerase II Poisons by Inhibition of the Nucleosome Remodeling Factor

Molecular cancer research, 2021-08, Vol.19 (8), p.1338-1349

2021

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Conversion of a Non-Cancer-Selective Promoter into a Cancer-Selective Promoter

Cancers, 2022-03, Vol.14 (6), p.1497

2022

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Tumor cell escape from therapy-induced senescence

Biochemical pharmacology, 2019-04, Vol.162, p.202-212

2019

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Immunologically programming the tumor microenvironment induces the pattern recognition receptor NLRC4-dependent antitumor immunity

Journal for immunotherapy of cancer, 2021-01, Vol.9 (1), p.e001595

2021

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The potentially conflicting cell autonomous and cell non-autonomous functions of autophagy in mediating tumor response to cancer therapy

Biochemical pharmacology, 2018-07, Vol.153, p.46-50

2018

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The Nucleosome Remodeling Factor

FEBS letters, 2011-10, Vol.585 (20), p.3197-3207

2011

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The Innate Immune System and TRAIL-BCL-XL Axis Mediate a Sex Bias in Lung Cancer and Confer a Therapeutic Vulnerability in Females

Cancer research (Chicago, Ill.), 2024-09

2024

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Pharmacological inhibition of MDA-9/Syntenin blocks breast cancer metastasis through suppression of IL-1β

Proceedings of the National Academy of Sciences - PNAS, 2021-05, Vol.118 (21), p.1

2021

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New Design Rules for Developing Potent Cell-Active Inhibitors of the Nucleosome Remodeling Factor (NURF) via BPTF Bromodomain Inhibition

Journal of medicinal chemistry, 2021-09, Vol.64 (18), p.13902-13917

2021

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ATP-Dependent Chromatin Remodeling Complexes as Novel Targets for Cancer Therapy

Advances in Cancer Research, 2014, Vol.121, p.183-233

2014

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Ino80 is essential for proximal-distal axis asymmetry in part by regulating Bmp4 expression

BMC biology, 2016-03, Vol.14 (18), p.18-18, Article 18

2016

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Chromatin remodeling complex NURF regulates thymocyte maturation

Genes & development, 2011-02, Vol.25 (3), p.275-286

2011

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Functional Interactions between NURF and Ctcf Regulate Gene Expression

Molecular and cellular biology, 2015-01, Vol.35 (1), p.224-237

2015

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BPTF Depletion Enhances T-cell-Mediated Antitumor Immunity

Cancer research (Chicago, Ill.), 2016-11, Vol.76 (21), p.6183-6192

2016

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WITHDRAWN: The chromatin remodeling complex NURF localizes to gene bodies and is required for mRNA processing

The Journal of biological chemistry, 2018-08, p.jbc.RA118.004382

2018

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BPTF inhibits NK cell activity and the abundance of natural cytotoxicity receptor co-ligands

Oncotarget, 2017-09, Vol.8 (38), p.64344-64357

2017

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Leveraging Epigenetics to Enhance the Cellular Response to Chemotherapies and Improve Tumor Immunogenicity

Advances in cancer research, 2018, Vol.138, p.1-39

2018

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Preface

Advances in cancer research, 2023-01, Vol.158, p.xiii-xvi

2023

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Abstract 7553: Dual inhibition of chromatin remodeling and DNA methylation as a novel treatment for triple negative breast cancer

Cancer research (Chicago, Ill.), 2024-03, Vol.84 (6_Supplement), p.7553-7553

2024

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Abstract 670: Sex differences in the innate immune response to lung cancer

Cancer research (Chicago, Ill.), 2023-04, Vol.83 (7_Supplement), p.670-670

2023

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Abstract 901: Elimination of senescent tumor cells by ABT263 interferes with proliferative recovery and provides a two-hit therapeutic approach

Cancer research (Chicago, Ill.), 2019-07, Vol.79 (13_Supplement), p.901-901

2019

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Sex Differences in Lung Cancer

Epigenetic Regulation of Cancer in Response to Chemotherapy

Non-Cell Autonomous Effects of the Senescence-Associated Secretory Phenotype in Cancer Therapy

Large-scale integration of DNA methylation and gene expression array platforms identifies both cis and trans relationships

Autophagy-Dependent Sensitization of Triple-Negative Breast Cancer Models to Topoisomerase II Poisons by Inhibition of the Nucleosome Remodeling Factor

Conversion of a Non-Cancer-Selective Promoter into a Cancer-Selective Promoter

Tumor cell escape from therapy-induced senescence

Immunologically programming the tumor microenvironment induces the pattern recognition receptor NLRC4-dependent antitumor immunity

The potentially conflicting cell autonomous and cell non-autonomous functions of autophagy in mediating tumor response to cancer therapy

The Nucleosome Remodeling Factor

The Innate Immune System and TRAIL-BCL-XL Axis Mediate a Sex Bias in Lung Cancer and Confer a Therapeutic Vulnerability in Females

Pharmacological inhibition of MDA-9/Syntenin blocks breast cancer metastasis through suppression of IL-1β

New Design Rules for Developing Potent Cell-Active Inhibitors of the Nucleosome Remodeling Factor (NURF) via BPTF Bromodomain Inhibition

ATP-Dependent Chromatin Remodeling Complexes as Novel Targets for Cancer Therapy

Ino80 is essential for proximal-distal axis asymmetry in part by regulating Bmp4 expression

Chromatin remodeling complex NURF regulates thymocyte maturation

Functional Interactions between NURF and Ctcf Regulate Gene Expression

BPTF Depletion Enhances T-cell-Mediated Antitumor Immunity

WITHDRAWN: The chromatin remodeling complex NURF localizes to gene bodies and is required for mRNA processing

BPTF inhibits NK cell activity and the abundance of natural cytotoxicity receptor co-ligands

Leveraging Epigenetics to Enhance the Cellular Response to Chemotherapies and Improve Tumor Immunogenicity

Preface

Abstract 7553: Dual inhibition of chromatin remodeling and DNA methylation as a novel treatment for triple negative breast cancer

Abstract 670: Sex differences in the innate immune response to lung cancer

Abstract 901: Elimination of senescent tumor cells by ABT263 interferes with proliferative recovery and provides a two-hit therapeutic approach

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