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Role of the Helicobacter pylori-Induced inflammatory response in the development of gastric cancer

Journal of cellular biochemistry, 2013-03, Vol.114 (3), p.491-497

2013

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Fibroblast-derived 3D matrix differentially regulates the growth and drug-responsiveness of human cancer cells

Matrix biology, 2008-07, Vol.27 (6), p.573-585

2008

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The many roads traveled by Helicobacter pylori to NF-κB activation

Gut microbes, 2010-03, Vol.1 (2), p.109-113

2010

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Methylation, a new epigenetic mark for protein stability

Epigenetics, 2009-10, Vol.4 (7), p.429-433

2009

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Posttranslational modifications of NF-κB: Another layer of regulation for NF-κB signaling pathway

Cellular signalling, 2010-09, Vol.22 (9), p.1282-1290

2010

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A Curriculum Development and Education Activity: Improving Pediatric Provider Comfort in Caring for LGBTQ Patients

Pediatrics (Evanston), 2019-08, Vol.144 (2_MeetingAbstract), p.579-579

2019

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Negative regulation of NF-κB action by Set9-mediated lysine methylation of the RelA subunit

The EMBO journal, 2009-04, Vol.28 (8), p.1055-1066

2009

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Helicobacter pylori CagA activates NF-κB by targeting TAK1 for TRAF6-mediated Lys 63 ubiquitination

EMBO reports, 2009-11, Vol.10 (11), p.1242-1249

2009

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Helicobacter pylori activates NF-κB by inducing Ubc13-mediated ubiquitination of lysine 158 of TAK1

Journal of cellular biochemistry, 2013-10, Vol.114 (10), p.2284-2292

2013

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Helicobacter pylori activates NF-[kappa]B by inducing Ubc13-mediated ubiquitination of lysine 158 of TAK1

Journal of cellular biochemistry, 2013-10, Vol.114 (10), p.2284

2013

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Helicobacter pylori activates NF-κB by inducing Ubc13-mediated ubiquitination of lysine 158 of TAK1: Activation of TAK1 by H. pylori CagA

Journal of cellular biochemistry, 2013-10, Vol.114 (10), p.2284-2292

2013

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Posttranslational modifications of NF-[kappa]B: Another layer of regulation for NF-[kappa]B signaling pathway

Cellular signalling, 2010-09, Vol.22 (9), p.1282-1290

2010

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New insights into the inactivation of gastric tumor suppressor RUNX3: The role of H. pylori infection

Journal of cellular biochemistry, 2011-02, Vol.112 (2), p.381-386

2011

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Negative regulation of NF-[kappa]B action by Set9-mediated lysine methylation of the RelA subunit

The EMBO journal, 2009-04, Vol.28 (8), p.1055

2009

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Response: strain-specific activation of NF-κB by Helicobacter pylori CagA

EMBO reports, 2010-01, Vol.11 (1), p.11-12

2010

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Response: strain-specific activation of NF-[kappa]B by Helicobacter pylori CagA

EMBO reports, 2010-01, Vol.11 (1), p.11

2010

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Helicobacter pylori CagA activates NF-[kappa]B by targeting TAK1 for TRAF6-mediated Lys 63 ubiquitination

EMBO reports, 2009-11, Vol.10 (11), p.1242-1249

2009

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New insights into a complex host-pathogen interaction: Mechanisms of Helicobacter pylori activation of NF-κB

Lamb, Acacia Christine

2012

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Posttranslational modifications of NF-kappaB: another layer of regulation for NF-kappaB signaling pathway

Cellular signalling, 2010-09, Vol.22 (9), p.1282-1290

2010

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Negative regulation of NF-kappaB action by Set9-mediated lysine methylation of the RelA subunit

The EMBO journal, 2009-04, Vol.28 (8), p.1055-1066

2009

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Helicobacter pylori CagA activates NF-kappaB by targeting TAK1 for TRAF6-mediated Lys 63 ubiquitination

EMBO reports, 2009-11, Vol.10 (11), p.1242-1249

2009

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A rapid procedure to assess shifts in discriminative control over drinking during recovery-like behavior

Alcohol (Fayetteville, N.Y.), 2024-12, Vol.121, p.87-93

2024

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Conditioned stimulus effects on paired or alternative reinforcement depend on presentation duration: Implications for conceptualizations of craving

Frontiers in behavioral neuroscience, 2022-08, Vol.16, p.958643-958643

2022

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Design of stimulus-responsive two-state hinge proteins

Science (American Association for the Advancement of Science), 2023-08, Vol.381 (6659), p.754-760

2023

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Methylation
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Role of the Helicobacter pylori-Induced inflammatory response in the development of gastric cancer

Fibroblast-derived 3D matrix differentially regulates the growth and drug-responsiveness of human cancer cells

The many roads traveled by Helicobacter pylori to NF-κB activation

Methylation, a new epigenetic mark for protein stability

Posttranslational modifications of NF-κB: Another layer of regulation for NF-κB signaling pathway

A Curriculum Development and Education Activity: Improving Pediatric Provider Comfort in Caring for LGBTQ Patients

Negative regulation of NF-κB action by Set9-mediated lysine methylation of the RelA subunit

Helicobacter pylori CagA activates NF-κB by targeting TAK1 for TRAF6-mediated Lys 63 ubiquitination

Helicobacter pylori activates NF-κB by inducing Ubc13-mediated ubiquitination of lysine 158 of TAK1

Helicobacter pylori activates NF-[kappa]B by inducing Ubc13-mediated ubiquitination of lysine 158 of TAK1

Helicobacter pylori activates NF-κB by inducing Ubc13-mediated ubiquitination of lysine 158 of TAK1: Activation of TAK1 by H. pylori CagA

Posttranslational modifications of NF-[kappa]B: Another layer of regulation for NF-[kappa]B signaling pathway

New insights into the inactivation of gastric tumor suppressor RUNX3: The role of H. pylori infection

Negative regulation of NF-[kappa]B action by Set9-mediated lysine methylation of the RelA subunit

Response: strain-specific activation of NF-κB by Helicobacter pylori CagA

Response: strain-specific activation of NF-[kappa]B by Helicobacter pylori CagA

Helicobacter pylori CagA activates NF-[kappa]B by targeting TAK1 for TRAF6-mediated Lys 63 ubiquitination

New insights into a complex host-pathogen interaction: Mechanisms of Helicobacter pylori activation of NF-κB

Posttranslational modifications of NF-kappaB: another layer of regulation for NF-kappaB signaling pathway

Negative regulation of NF-kappaB action by Set9-mediated lysine methylation of the RelA subunit

Helicobacter pylori CagA activates NF-kappaB by targeting TAK1 for TRAF6-mediated Lys 63 ubiquitination

A rapid procedure to assess shifts in discriminative control over drinking during recovery-like behavior

Conditioned stimulus effects on paired or alternative reinforcement depend on presentation duration: Implications for conceptualizations of craving

Design of stimulus-responsive two-state hinge proteins

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