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Evidence that resistance to nilotinib may be due to BCR-ABL, Pgp, or Src kinase overexpression

Cancer research (Chicago, Ill.), 2008-12, Vol.68 (23), p.9809-9816

2008

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Quantitative phosphoproteomics revealed interplay between Syk and Lyn in the resistance to nilotinib in chronic myeloid leukemia cells

Blood, 2011-08, Vol.118 (8), p.2211-2221

2011

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MDR1 gene overexpression confers resistance to imatinib mesylate in leukemia cell line models

Blood, 2003-03, Vol.101 (6), p.2368-2373

2003

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A tyrosine kinase-STAT5-miR21-PDCD4 regulatory axis in chronic and acute myeloid leukemia cells

Oncotarget, 2017-09, Vol.8 (44), p.76174-76188

2017

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CBL controls a tyrosine kinase network involving AXL, SYK and LYN in nilotinib-resistant chronic myeloid leukaemia

The Journal of pathology, 2015-09, Vol.237 (1), p.14-24

2015

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Variable behavior of iPSCs derived from CML patients for response to TKI and hematopoietic differentiation

PloS one, 2013-08, Vol.8 (8), p.e71596-e71596

2013

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Cbl Controls the Expression of Axl and Lyn Tyrosine Kinases Mediating Resistance to Nilotinib in Chronic Myeloid Leukemia Cells

Blood, 2012-11, Vol.120 (21), p.3734-3734

2012

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Proteomic analysis of an imatinib-resistant K562 cell line highlights opposing roles of heat shock cognate 70 and heat shock 70 proteins in resistance

Proteomics (Weinheim), 2008-06, Vol.8 (12), p.2394-2406

2008

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Induced Pluripotent Stem Cells (iPSC) From Chronic Myeloid Leukemia: Study of BCR-ABL Addiction and Effect of Tyrosine Kinase Inhibitors

Blood, 2011-11, Vol.118 (21), p.3754-3754

2011

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Quantitative Phosphoproteomics Identified a New Syk-Lyn-Axl Signalling Pathway Involved In Resistance to Nilotinib In Chronic Myeloid Leukemia Cells

Blood, 2010-11, Vol.116 (21), p.3376-3376

2010

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Les effets du programme de soin sur le processus thérapeutique

L'Information psychiatrique, 2017-05, Vol.93 (5), p.381-386

2017

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α-defensin 1-3 and α-defensin 4 as predictive markers of imatinib resistance and relapse in CML patients

Disease markers, 2011, Vol.30 (5), p.221-227

2011

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Mutation in the ATP‐binding site of BCR‐ABL in a patient with chronic myeloid leukaemia with increasing resistance to STI571

British journal of haematology, 2002-10, Vol.119 (1), p.109-111

2002

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Heat Shock Protein 70 over Expression Is Associated to Imatinib Resistance in Chronic Myelogenous Leukemia

Blood, 2005-11, Vol.106 (11), p.2005-2005

2005

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Mycophenolic Acid Overcomes Imatinib and Nilotinib Resistance of Chronic Myeloid Leukemia Cells by Apoptosis or a Senescent-Like Cell Cycle Arrest

Leukemia Research and Treatment, 2012-01, Vol.2012, p.861301-9

2012

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The Proteome Analysis of an Imatinib-Resistant Cell Line Identifies Changes in Molecular Chaperones Such as Heat Shock Proteins (Hsp): A Possible New Mechanism of Imatinib Resistance

Blood, 2004-11, Vol.104 (11), p.1981-1981

2004

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CBL controls a tyrosine kinase network involving AXL, SYK and LYN in nilotinib-resistant chronic myeloid leukaemia: CBL controls kinase expression

The Journal of pathology, 2015-09, Vol.237 (1), p.14-24

2015

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Ex vivo cytokine expansion of peripheral blood 5‐fluorouracil‐treated CD34‐positive chronic myeloid leukaemia cells increases the selection of Ph‐negative cells

British journal of haematology, 1997-08, Vol.98 (2), p.467-473

1997

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α-defensin 1-3 and α-defensin 4 as predictive markers of imatinib resistance and relapse in CML patients

Disease markers, 2011, Vol.30 (5), p.221-227

2011

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Overproduction of BCR‐ABL induces apoptosis in imatinib mesylate‐resistant cell lines

Cancer, 2005-01, Vol.103 (1), p.102-110

2005

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Rapid detection of phosphotyrosine proteins by flow cytometric analysis in Bcr‐Abl–positive cells

Cytometry. Part A, 2004-11, Vol.62A (1), p.35-45

2004

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Tyrosine Kinase Proteins profiling of Nilotinib Resistant Chronic Myelogenous Leukemia Cells Unravels a Tyrosine Kinase-Mediated Bypass

Blood, 2009-11, Vol.114 (22), p.2175-2175

2009

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Restoration of sensitivity to STI571 in STI571-resistant chronic myeloid leukemia cells

Blood, 2001-12, Vol.98 (13), p.3864-3867

2001

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Retroviral coexpression of IFN-alpha and IFN-gamma genes and inhibitory effects in chronic myeloid leukemia cells

Journal of interferon & cytokine research, 2000-06, Vol.20 (6), p.577-587

2000

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Evidence that resistance to nilotinib may be due to BCR-ABL, Pgp, or Src kinase overexpression

Quantitative phosphoproteomics revealed interplay between Syk and Lyn in the resistance to nilotinib in chronic myeloid leukemia cells

MDR1 gene overexpression confers resistance to imatinib mesylate in leukemia cell line models

A tyrosine kinase-STAT5-miR21-PDCD4 regulatory axis in chronic and acute myeloid leukemia cells

CBL controls a tyrosine kinase network involving AXL, SYK and LYN in nilotinib-resistant chronic myeloid leukaemia

Variable behavior of iPSCs derived from CML patients for response to TKI and hematopoietic differentiation

Cbl Controls the Expression of Axl and Lyn Tyrosine Kinases Mediating Resistance to Nilotinib in Chronic Myeloid Leukemia Cells

Proteomic analysis of an imatinib-resistant K562 cell line highlights opposing roles of heat shock cognate 70 and heat shock 70 proteins in resistance

Induced Pluripotent Stem Cells (iPSC) From Chronic Myeloid Leukemia: Study of BCR-ABL Addiction and Effect of Tyrosine Kinase Inhibitors

Quantitative Phosphoproteomics Identified a New Syk-Lyn-Axl Signalling Pathway Involved In Resistance to Nilotinib In Chronic Myeloid Leukemia Cells

The Expression of Myeloproliferative Neoplasm-Associated Calreticulin Variants Depends on the Functionality of ER-Associated Degradation

Les effets du programme de soin sur le processus thérapeutique

α-defensin 1-3 and α-defensin 4 as predictive markers of imatinib resistance and relapse in CML patients

Mutation in the ATP‐binding site of BCR‐ABL in a patient with chronic myeloid leukaemia with increasing resistance to STI571

Heat Shock Protein 70 over Expression Is Associated to Imatinib Resistance in Chronic Myelogenous Leukemia

Mycophenolic Acid Overcomes Imatinib and Nilotinib Resistance of Chronic Myeloid Leukemia Cells by Apoptosis or a Senescent-Like Cell Cycle Arrest

The Proteome Analysis of an Imatinib-Resistant Cell Line Identifies Changes in Molecular Chaperones Such as Heat Shock Proteins (Hsp): A Possible New Mechanism of Imatinib Resistance

CBL controls a tyrosine kinase network involving AXL, SYK and LYN in nilotinib-resistant chronic myeloid leukaemia: CBL controls kinase expression

Ex vivo cytokine expansion of peripheral blood 5‐fluorouracil‐treated CD34‐positive chronic myeloid leukaemia cells increases the selection of Ph‐negative cells

α-defensin 1-3 and α-defensin 4 as predictive markers of imatinib resistance and relapse in CML patients

Overproduction of BCR‐ABL induces apoptosis in imatinib mesylate‐resistant cell lines

Rapid detection of phosphotyrosine proteins by flow cytometric analysis in Bcr‐Abl–positive cells

Tyrosine Kinase Proteins profiling of Nilotinib Resistant Chronic Myelogenous Leukemia Cells Unravels a Tyrosine Kinase-Mediated Bypass

Restoration of sensitivity to STI571 in STI571-resistant chronic myeloid leukemia cells

Retroviral coexpression of IFN-alpha and IFN-gamma genes and inhibitory effects in chronic myeloid leukemia cells

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