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Virus Infection Switches TLR-3-Positive Human Neurons To Become Strong Producers of Beta Interferon

Journal of Virology, 2005-10, Vol.79 (20), p.12893-12904

2005

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Comparative analysis of neuroinvasion by Japanese encephalitis virulent and vaccine viral strains in an in vitro model of human blood-brain barrier

PloS one, 2021-06, Vol.16 (6), p.e0252595-e0252595

2021

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Ambivalent Role of the Innate Immune Response in Rabies Virus Pathogenesis

Journal of Virology, 2011-07, Vol.85 (13), p.6657-6668

2011

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Evasive Strategies in Rabies Virus Infection

Advances in Virus Research, 2011, Vol.79, p.33-53

Lafon, Monique

2011

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Toll-like receptor 3 (TLR3) plays a major role in the formation of rabies virus Negri Bodies

PLoS pathogens, 2009-02, Vol.5 (2), p.e1000315-e1000315

2009

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The innate immune facet of brain: human neurons express TLR-3 and sense viral dsRNA

Journal of molecular neuroscience, 2006, Vol.29 (3), p.185-194

2006

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Protective Role of LGP2 in Influenza Virus Pathogenesis

The Journal of infectious diseases, 2014-07, Vol.210 (2), p.214-223

2014

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Attenuation of rabies virulence: takeover by the cytoplasmic domain of its envelope protein

Science signaling, 2010-01, Vol.3 (105), p.ra5

2010

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The type I interferon response bridles rabies virus infection and reduces pathogenicity

Journal of neurovirology, 2011-08, Vol.17 (4), p.353-367

2011

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Detrimental Contribution of the Immuno-Inhibitor B7-H1 to Rabies Virus Encephalitis

The Journal of immunology (1950), 2008-06, Vol.180 (11), p.7506-7515

2008

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Modulation of HLA-G Expression in Human Neural Cells after Neurotropic Viral Infections

Journal of Virology, 2005-12, Vol.79 (24), p.15226-15237

2005

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Glycoprotein of Nonpathogenic Rabies Viruses Is a Key Determinant of Human Cell Apoptosis

Journal of Virology, 2003-10, Vol.77 (19), p.10537-10547

2003

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Rabies virus receptors

Journal of neurovirology, 2005-02, Vol.11 (1), p.82-87

Lafon, Monique

2005

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Genomic diversity contributes to the neuroinvasiveness of the Yellow fever French neurotropic vaccine

npj vaccines, 2021-04, Vol.6 (1), p.64-64, Article 64

2021

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Expression of Toll-like receptor 3 in the human cerebellar cortex in rabies, herpes simplex encephalitis, and other neurological diseases

Journal of neurovirology, 2006-06, Vol.12 (3), p.229-234

2006

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The neural cell adhesion molecule is a receptor for rabies virus

Journal of Virology, 1998-09, Vol.72 (9), p.7181-7190

1998

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Modulation of HLA-G and HLA-E Expression in Human Neuronal Cells After Rabies Virus or Herpes Virus Simplex Type 1 Infections

Human immunology, 2007-04, Vol.68 (4), p.294-302

2007

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Up-regulation of Fas ligand (FasL) in the central nervous system: A mechanism of immune evasion by rabies virus

Journal of neurovirology, 2004-12, Vol.10 (6), p.372-382

2004

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Investigation of rabies virus glycoprotein carboxyl terminus as an in vitro predictive tool of neurovirulence. A 3R approach

Microbes and infection, 2017-09, Vol.19 (9-10), p.476-484

2017

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Chapter 3 - Evasive Strategies in Rabies Virus Infection

Advances in virus research, 2011-01, Vol.79, p.33-53

Lafon, Monique

2011

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Rabies virus glycoprotein enhances spatial memory via the PDZ binding motif

Journal of neurovirology, 2021-06, Vol.27 (3), p.434-443

2021

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Inhibition of Polyamine Biosynthesis Is a Broad-Spectrum Strategy against RNA Viruses

Journal of virology, 2016-11, Vol.90 (21), p.9683-9692

2016

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Structure-based optimization of a PDZ-binding motif within a viral peptide stimulates neurite outgrowth

The Journal of biological chemistry, 2019-09, Vol.294 (37), p.13755-13768

2019

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Bat rabies—the Achilles heel of a viral killer?

The Lancet (British edition), 2005-09, Vol.366 (9489), p.876-877

Lafon, Monique

2005

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Comparison of G protein sequences of South African street rabies viruses showing distinct progression of the disease in a mouse model of experimental rabies

Microbes and infection, 2017-09, Vol.19 (9-10), p.485-491

2017

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Virus Infection Switches TLR-3-Positive Human Neurons To Become Strong Producers of Beta Interferon

Comparative analysis of neuroinvasion by Japanese encephalitis virulent and vaccine viral strains in an in vitro model of human blood-brain barrier

Ambivalent Role of the Innate Immune Response in Rabies Virus Pathogenesis

Evasive Strategies in Rabies Virus Infection

Toll-like receptor 3 (TLR3) plays a major role in the formation of rabies virus Negri Bodies

The innate immune facet of brain: human neurons express TLR-3 and sense viral dsRNA

Protective Role of LGP2 in Influenza Virus Pathogenesis

Attenuation of rabies virulence: takeover by the cytoplasmic domain of its envelope protein

The type I interferon response bridles rabies virus infection and reduces pathogenicity

Detrimental Contribution of the Immuno-Inhibitor B7-H1 to Rabies Virus Encephalitis

Modulation of HLA-G Expression in Human Neural Cells after Neurotropic Viral Infections

Glycoprotein of Nonpathogenic Rabies Viruses Is a Key Determinant of Human Cell Apoptosis

Rabies virus receptors

Genomic diversity contributes to the neuroinvasiveness of the Yellow fever French neurotropic vaccine

Expression of Toll-like receptor 3 in the human cerebellar cortex in rabies, herpes simplex encephalitis, and other neurological diseases

The neural cell adhesion molecule is a receptor for rabies virus

Modulation of HLA-G and HLA-E Expression in Human Neuronal Cells After Rabies Virus or Herpes Virus Simplex Type 1 Infections

Up-regulation of Fas ligand (FasL) in the central nervous system: A mechanism of immune evasion by rabies virus

Investigation of rabies virus glycoprotein carboxyl terminus as an in vitro predictive tool of neurovirulence. A 3R approach

Chapter 3 - Evasive Strategies in Rabies Virus Infection

Rabies virus glycoprotein enhances spatial memory via the PDZ binding motif

Inhibition of Polyamine Biosynthesis Is a Broad-Spectrum Strategy against RNA Viruses

Structure-based optimization of a PDZ-binding motif within a viral peptide stimulates neurite outgrowth

Bat rabies—the Achilles heel of a viral killer?

Comparison of G protein sequences of South African street rabies viruses showing distinct progression of the disease in a mouse model of experimental rabies

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