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PINK1 and Parkin Target Miro for Phosphorylation and Degradation to Arrest Mitochondrial Motility

Cell, 2011-11, Vol.147 (4), p.893-906

2011

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Is there a special relationship between complex I activity and nigral neuronal loss in Parkinson’s disease? A critical reappraisal

Brain research, 2021-09, Vol.1767, p.147434-147434, Article 147434

2021

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Mitophagy of damaged mitochondria occurs locally in distal neuronal axons and requires PINK1 and Parkin

The Journal of cell biology, 2014-09, Vol.206 (5), p.655-670

2014

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Membrane recruitment of endogenous LRRK2 precedes its potent regulation of autophagy

Human molecular genetics, 2014-08, Vol.23 (16), p.4201-4214

2014

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Miro phosphorylation sites regulate Parkin recruitment and mitochondrial motility

Proceedings of the National Academy of Sciences - PNAS, 2016-10, Vol.113 (41), p.E6097-E6106

2016

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Membrane recruitment of endogenous LRRK2 precedes its potent regulation of autophagy

Human molecular genetics, 2014-08, Vol.23 (16), p.4201-4214

2014

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Lysosome and Inflammatory Defects in GBA1‐Mutant Astrocytes Are Normalized by LRRK2 Inhibition

Movement disorders, 2020-05, Vol.35 (5), p.760-773

2020

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Cell Type-Specific Transcriptomics Reveals that Mutant Huntingtin Leads to Mitochondrial RNA Release and Neuronal Innate Immune Activation

Neuron (Cambridge, Mass.), 2020-09, Vol.107 (5), p.891-908.e8

2020

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Regulation of a distinct activated RIPK1 intermediate bridging complex I and complex II in TNFα-mediated apoptosis

Proceedings of the National Academy of Sciences - PNAS, 2018-06, Vol.115 (26), p.E5944-E5953

2018

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Membrane Localization of LRRK2 Is Associated with Increased Formation of the Highly Active LRRK2 Dimer and Changes in Its Phosphorylation

Biochemistry (Easton), 2010-07, Vol.49 (26), p.5511-5523

2010

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Familial knockin mutation of LRRK2 causes lysosomal dysfunction and accumulation of endogenous insoluble α-synuclein in neurons

Neurobiology of disease, 2018-03, Vol.111, p.26-35

2018

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The ubiquitin E3 ligase parkin regulates the proapoptotic function of Bax

Proceedings of the National Academy of Sciences - PNAS, 2012-04, Vol.109 (16), p.6283-6288

2012

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LRRK2 Kinase Inhibition Rescues Deficits in Lysosome Function Due to Heterozygous GBA1 Expression in Human iPSC-Derived Neurons

Frontiers in neuroscience, 2020-05, Vol.14, p.442-442

2020

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Pathologic and therapeutic implications for the cell biology of parkin

Molecular and cellular neuroscience, 2015-05, Vol.66 (Pt A), p.62-71

2015

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Career building as a neuroscientist at a research hospital

Annals of neurology, 2015-03, Vol.77 (3), p.367-370

LaVoie, Matthew J.

2015

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Parkinson’s disease-linked LRRK2 is expressed in circulating and tissue immune cells and upregulated following recognition of microbial structures

Journal of Neural Transmission, 2011-05, Vol.118 (5), p.795-808

2011

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Polymerization of recombinant tau core fragments in vitro and seeding studies in cultured cells

Frontiers in neuroscience, 2023, Vol.17, p.1268360-1268360

2023

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A Unique Case of COVID-19-related Acute Coronary Thrombosis Complicated by Severe Hypokalemia

Military medicine, 2021-09, Vol.186 (9-10), p.e1053-e1057

2021

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Dopamine covalently modifies and functionally inactivates parkin

Nature medicine, 2005-11, Vol.11 (11), p.1214-1221

2005

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Correction: Mutations in LRRK2 linked to Parkinson disease sequester Rab8a to damaged lysosomes and regulate transferrin-mediated iron uptake in microglia

PLoS biology, 2022-05, Vol.20 (5), p.e3001621-e3001621

2022

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γ-Secretase Is a Membrane Protein Complex Comprised of Presenilin, Nicastrin, Aph-1, and Pen-2

Proceedings of the National Academy of Sciences - PNAS, 2003-05, Vol.100 (11), p.6382-6387

2003

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The Notch Ligands, Jagged and Delta, Are Sequentially Processed by α-Secretase and Presenilin/γ-Secretase and Release Signaling Fragments

The Journal of biological chemistry, 2003-09, Vol.278 (36), p.34427-34437

2003

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The mitochondrial disease associated protein Ndufaf2 is dispensable for Complex-1 assembly but critical for the regulation of oxidative stress

Neurobiology of disease, 2013-10, Vol.58, p.57-67

2013

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Microglial activation precedes dopamine terminal pathology in methamphetamine-induced neurotoxicity

Experimental neurology, 2004-05, Vol.187 (1), p.47-57

2004

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PINK1 and Parkin Target Miro for Phosphorylation and Degradation to Arrest Mitochondrial Motility

Is there a special relationship between complex I activity and nigral neuronal loss in Parkinson’s disease? A critical reappraisal

Mitophagy of damaged mitochondria occurs locally in distal neuronal axons and requires PINK1 and Parkin

Membrane recruitment of endogenous LRRK2 precedes its potent regulation of autophagy

Miro phosphorylation sites regulate Parkin recruitment and mitochondrial motility

Membrane recruitment of endogenous LRRK2 precedes its potent regulation of autophagy

Lysosome and Inflammatory Defects in GBA1‐Mutant Astrocytes Are Normalized by LRRK2 Inhibition

Cell Type-Specific Transcriptomics Reveals that Mutant Huntingtin Leads to Mitochondrial RNA Release and Neuronal Innate Immune Activation

Regulation of a distinct activated RIPK1 intermediate bridging complex I and complex II in TNFα-mediated apoptosis

Membrane Localization of LRRK2 Is Associated with Increased Formation of the Highly Active LRRK2 Dimer and Changes in Its Phosphorylation

Familial knockin mutation of LRRK2 causes lysosomal dysfunction and accumulation of endogenous insoluble α-synuclein in neurons

The ubiquitin E3 ligase parkin regulates the proapoptotic function of Bax

Mutations in LRRK2 linked to Parkinson disease sequester Rab8a to damaged lysosomes and regulate transferrin-mediated iron uptake in microglia

LRRK2 Kinase Inhibition Rescues Deficits in Lysosome Function Due to Heterozygous GBA1 Expression in Human iPSC-Derived Neurons

Pathologic and therapeutic implications for the cell biology of parkin

Career building as a neuroscientist at a research hospital

Parkinson’s disease-linked LRRK2 is expressed in circulating and tissue immune cells and upregulated following recognition of microbial structures

Polymerization of recombinant tau core fragments in vitro and seeding studies in cultured cells

A Unique Case of COVID-19-related Acute Coronary Thrombosis Complicated by Severe Hypokalemia

Dopamine covalently modifies and functionally inactivates parkin

Correction: Mutations in LRRK2 linked to Parkinson disease sequester Rab8a to damaged lysosomes and regulate transferrin-mediated iron uptake in microglia

γ-Secretase Is a Membrane Protein Complex Comprised of Presenilin, Nicastrin, Aph-1, and Pen-2

The Notch Ligands, Jagged and Delta, Are Sequentially Processed by α-Secretase and Presenilin/γ-Secretase and Release Signaling Fragments

The mitochondrial disease associated protein Ndufaf2 is dispensable for Complex-1 assembly but critical for the regulation of oxidative stress

Microglial activation precedes dopamine terminal pathology in methamphetamine-induced neurotoxicity

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