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WWOX, the common chromosomal fragile site, FRA16D, cancer gene

Cytogenetic and Genome Research, 2003-01, Vol.100 (1-4), p.101-110

2003

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WWOX binds the specific proline-rich ligand PPXY: identification of candidate interacting proteins

Oncogene, 2004-06, Vol.23 (29), p.5049-5055

2004

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'Gain of function' phenotype of tumor-derived mutant p53 requires the oligomerization/nonsequence-specific nucleic acid-binding domain

Oncogene, 1998-06, Vol.16 (24), p.3169-3176

1998

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Forkhead Homologue in Rhabdomyosarcoma Functions as a Bifunctional Nuclear Receptor-interacting Protein with Both Coactivator and Corepressor Functions

The Journal of biological chemistry, 2001-07, Vol.276 (30), p.27907-27912

2001

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Expression of common chromosomal fragile site genes, WWOX/FRA16D and FHIT/FRA3B is downregulated by exposure to environmental carcinogens, UV, and BPDE but not by IR

Molecular carcinogenesis, 2005-11, Vol.44 (3), p.174-182

2005

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Transcriptional activation of the human epidermal growth factor receptor promoter by human p53

Molecular and Cellular Biology, 1996-11, Vol.16 (11), p.6009-6019

1996

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AP-1 blockade inhibits the growth of normal and malignant breast cells

Oncogene, 2001-05, Vol.20 (22), p.2771-2780

2001

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WWOX protein expression varies among ovarian carcinoma histotypes and correlates with less favorable outcome

BMC cancer, 2005-06, Vol.5 (1), p.64-64, Article 64

2005

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Isolation of an amino-terminal region of bovine papillomavirus type 1 E1 protein that retains origin binding and E2 interaction capacity

Journal of Virology, 1997-01, Vol.71 (1), p.848-852

1997

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Simple Procedure for Creation of In-Frame Deletion Mutations Throughout an Open Reading Frame

BioTechniques, 1996-03, Vol.20 (3), p.433-438

1996

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CtIP, a BRCA1 binding protein, is a transcriptional corepressor of estrogen receptor α

Cancer research (Chicago, Ill.), 2009-01, Vol.69 (2_Supplement), p.606

2009

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WWOX, the common chromosomal fragile site, FRA16D, cancer gene: Nucleotide and protein expansions and human disease

Cytogenetic and genome research, 2003, Vol.100 (1-4), p.101-110

2003

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WWOX hypomorphic mice display a higher incidence of B-cell lymphomas and develop testicular atrophy

Genes chromosomes & cancer, 2007-12, Vol.46 (12), p.1129-1136

2007

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Inhibition of AP-1 transcription factor causes blockade of multiple signal transduction pathways and inhibits breast cancer growth

Oncogene, 2002-10, Vol.21 (50), p.7680-7689

2002

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Frequent loss of WWOX expression in breast cancer: correlation with estrogen receptor status

Breast cancer research and treatment, 2005, Vol.89 (2), p.99-105

2005

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WWOX, the common chromosomal fragile site, FRA16D, cancer gene

WWOX binds the specific proline-rich ligand PPXY: identification of candidate interacting proteins

'Gain of function' phenotype of tumor-derived mutant p53 requires the oligomerization/nonsequence-specific nucleic acid-binding domain

Forkhead Homologue in Rhabdomyosarcoma Functions as a Bifunctional Nuclear Receptor-interacting Protein with Both Coactivator and Corepressor Functions

Expression of common chromosomal fragile site genes, WWOX/FRA16D and FHIT/FRA3B is downregulated by exposure to environmental carcinogens, UV, and BPDE but not by IR

Transcriptional activation of the human epidermal growth factor receptor promoter by human p53

AP-1 blockade inhibits the growth of normal and malignant breast cells

WWOX protein expression varies among ovarian carcinoma histotypes and correlates with less favorable outcome

Isolation of an amino-terminal region of bovine papillomavirus type 1 E1 protein that retains origin binding and E2 interaction capacity

Simple Procedure for Creation of In-Frame Deletion Mutations Throughout an Open Reading Frame

CtIP, a BRCA1 binding protein, is a transcriptional corepressor of estrogen receptor α

WWOX, the common chromosomal fragile site, FRA16D, cancer gene: Nucleotide and protein expansions and human disease

WWOX hypomorphic mice display a higher incidence of B-cell lymphomas and develop testicular atrophy

Inhibition of AP-1 transcription factor causes blockade of multiple signal transduction pathways and inhibits breast cancer growth

Frequent loss of WWOX expression in breast cancer: correlation with estrogen receptor status

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