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Structural alterations in the peptide backbone of beta-amyloid core protein may account for its deposition and stability in Alzheimer's disease

The Journal of biological chemistry, 1993-02, Vol.268 (5), p.3072-3083

1993

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Recognition of D-aspartyl residues in polypeptides by the erythrocyte L-isoaspartyl/D-aspartyl protein methyltransferase. Implications for the repair hypothesis

The Journal of biological chemistry, 1992-03, Vol.267 (9), p.5985-5995

1992

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Protein repair L-isoaspartyl methyltransferase in plants. Phylogenetic distribution and the accumulation of substrate proteins in aged barley seeds

Plant physiology (Bethesda), 1997-12, Vol.115 (4), p.1481-1489

1997

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Deficiency of a Protein-Repair Enzyme Results in the Accumulation of Altered Proteins, Retardation of Growth, and Fatal Seizures in Mice

Proceedings of the National Academy of Sciences - PNAS, 1997-06, Vol.94 (12), p.6132-6137

1997

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Structural elements affecting the recognition of L-isoaspartyl residues by the L-isoaspartyl/D-aspartyl protein methyltransferase. Implications for the repair hypothesis

The Journal of biological chemistry, 1991-10, Vol.266 (29), p.19396-19406

1991

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Limited accumulation of damaged proteins in l-isoaspartyl (D-aspartyl) O-methyltransferase-deficient mice

The Journal of biological chemistry, 2001-06, Vol.276 (23), p.20695-20702

2001

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D-aspartate content of erythrocyte membrane proteins is decreased in uremia : Implications for the repair of damaged proteins

Journal of the American Society of Nephrology, 1997, Vol.8 (1), p.95-104

1997

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Phenotypic Analysis of Seizure-prone Mice Lacking l-Isoaspartate (d-Aspartate)O-Methyltransferase

The Journal of biological chemistry, 1999-07, Vol.274 (29), p.20671-20678

1999

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Protein Repair L-Isoaspartyl Methyltransferase in Plants (Phylogenetic Distribution and the Accumulation of Substrate Proteins in Aged Barley Seeds)

Plant physiology (Bethesda), 1997-12, Vol.115 (4), p.1481-1489

1997

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An Arabidopsis ATP-Dependent, DEAD-Box RNA Helicase Loses Activity upon IsoAsp Formation but Is Restored by PROTEIN ISOASPARTYL METHYLTRANSFERASE

The Plant cell, 2013-07, Vol.25 (7), p.2573-2586

2013

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Spontaneous degradation and enzymatic repair of aspartyl and asparaginyl residues in aging red cell proteins analyzed by computer simulation

Gerontology (Basel), 1991-01, Vol.37 (1-3), p.128-151

1991

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Brain Proteomics Supports the Role of Glutamate Metabolism and Suggests Other Metabolic Alterations in Protein l‑Isoaspartyl Methyltransferase (PIMT)-Knockout Mice

Journal of proteome research, 2013-10, Vol.12 (10), p.4566-4576

2013

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Identification of isoaspartyl-containing sequences in peptides and proteins that are unusually poor substrates for the class II protein carboxyl methyltransferase

The Journal of biological chemistry, 1990-02, Vol.265 (6), p.3106-3110

1990

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[7] Chemical modifications of deposited amyloid-β peptides

Methods in Enzymology, 1999, Vol.309, p.89-105

1999

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Synapsin I Is a Major Endogenous Substrate for Protein L-Isoaspartyl Methyltransferase in Mammalian Brain

The Journal of biological chemistry, 2006-03, Vol.281 (13), p.8389-8398

2006

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Identification of isoaspartyl-containing sequences in peptides and proteins that are usually poor substrates for the class II protein carboxyl methyltransferase

The Journal of biological chemistry, 1990-02, Vol.265 (6), p.3106-3110

1990

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Intracellular protein modification associated with altered T cell functions in autoimmunity

The Journal of immunology (1950), 2006-12, Vol.177 (12), p.8878-8878

2006

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A new type of protein methylation activated by tyrphostin A25 and vanadate

FEBS letters, 2004-11, Vol.577 (1), p.181-186

2004

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Protein aging: Extracellular amyloid formation and intracellular repair

Trends in Cardiovascular Medicine, 1994, Vol.4 (1), p.3-8

1994

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Protein repair L-isoaspartyl methyltransferase in plants. Phylogenetic distribution and the accumulation of substrate proteins in aged barley seeds

Plant physiology (Bethesda), 1997-12, Vol.115 (4), p.1491-1498

1997

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Structural elements affecting the recognition of L-isopartyl residues by the L-isoaspartyl/D-aspartyl protein methyltransferase : implications for the repair hypothesis

The Journal of biological chemistry, 1991, Vol.266 (29), p.19396-19406

1991

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Human ARMT1 structure and substrate specificity indicates that it is a DUF89 family damage-control phosphatase

Journal of structural biology, 2020-10, Vol.212 (1), p.107576-107576, Article 107576

2020

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Chemo-Enzymatic Detection of Protein Isoaspartate Using Protein Isoaspartate Methyltransferase and Hydrazine Trapping

Analytical chemistry (Washington), 2008-05, Vol.80 (10), p.3882-3889

2008

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Intracellular Protein Modification Associated with Altered T Cell Functions in Autoimmunity

Journal of Immunology, 2006-10, Vol.177 (7), p.4541-4549

2006

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Replacement of a labile aspartyl residue increases the stability of human epidermal growth factor

Biochemistry (Easton), 1990-10, Vol.29 (41), p.9584-9591

1990

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Structural alterations in the peptide backbone of beta-amyloid core protein may account for its deposition and stability in Alzheimer's disease

Recognition of D-aspartyl residues in polypeptides by the erythrocyte L-isoaspartyl/D-aspartyl protein methyltransferase. Implications for the repair hypothesis

Protein repair L-isoaspartyl methyltransferase in plants. Phylogenetic distribution and the accumulation of substrate proteins in aged barley seeds

Deficiency of a Protein-Repair Enzyme Results in the Accumulation of Altered Proteins, Retardation of Growth, and Fatal Seizures in Mice

Structural elements affecting the recognition of L-isoaspartyl residues by the L-isoaspartyl/D-aspartyl protein methyltransferase. Implications for the repair hypothesis

Limited accumulation of damaged proteins in l-isoaspartyl (D-aspartyl) O-methyltransferase-deficient mice

D-aspartate content of erythrocyte membrane proteins is decreased in uremia : Implications for the repair of damaged proteins

Phenotypic Analysis of Seizure-prone Mice Lacking l-Isoaspartate (d-Aspartate)O-Methyltransferase

Protein Repair L-Isoaspartyl Methyltransferase in Plants (Phylogenetic Distribution and the Accumulation of Substrate Proteins in Aged Barley Seeds)

An Arabidopsis ATP-Dependent, DEAD-Box RNA Helicase Loses Activity upon IsoAsp Formation but Is Restored by PROTEIN ISOASPARTYL METHYLTRANSFERASE

Spontaneous degradation and enzymatic repair of aspartyl and asparaginyl residues in aging red cell proteins analyzed by computer simulation

Brain Proteomics Supports the Role of Glutamate Metabolism and Suggests Other Metabolic Alterations in Protein l‑Isoaspartyl Methyltransferase (PIMT)-Knockout Mice

Identification of isoaspartyl-containing sequences in peptides and proteins that are unusually poor substrates for the class II protein carboxyl methyltransferase

[7] Chemical modifications of deposited amyloid-β peptides

Synapsin I Is a Major Endogenous Substrate for Protein L-Isoaspartyl Methyltransferase in Mammalian Brain

Identification of isoaspartyl-containing sequences in peptides and proteins that are usually poor substrates for the class II protein carboxyl methyltransferase

Intracellular protein modification associated with altered T cell functions in autoimmunity

A new type of protein methylation activated by tyrphostin A25 and vanadate

Protein aging: Extracellular amyloid formation and intracellular repair

Protein repair L-isoaspartyl methyltransferase in plants. Phylogenetic distribution and the accumulation of substrate proteins in aged barley seeds

Structural elements affecting the recognition of L-isopartyl residues by the L-isoaspartyl/D-aspartyl protein methyltransferase : implications for the repair hypothesis

Human ARMT1 structure and substrate specificity indicates that it is a DUF89 family damage-control phosphatase

Chemo-Enzymatic Detection of Protein Isoaspartate Using Protein Isoaspartate Methyltransferase and Hydrazine Trapping

Intracellular Protein Modification Associated with Altered T Cell Functions in Autoimmunity

Replacement of a labile aspartyl residue increases the stability of human epidermal growth factor

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