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Epilepsy in cardiofaciocutaneous syndrome: Clinical burden and response to anti‐seizure medication

American journal of medical genetics. Part A, 2024-02, Vol.194 (2), p.301-310

2024

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RNA induces unique tau strains and stabilizes Alzheimer’s disease seeds

The Journal of biological chemistry, 2022-08, Vol.298 (8), p.102132-102132, Article 102132

2022

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Endotoxin tolerance: new mechanisms, molecules and clinical significance

Trends in immunology, 2009-10, Vol.30 (10), p.475-487

2009

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Effects of CPAP on oxidative stress and nitrate efficiency in sleep apnoea: a randomised trial

Thorax, 2009-07, Vol.64 (7), p.581-586

2009

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Circulating cell-free mitochondrial DNA: A better early prognostic marker in patients with out-of-hospital cardiac arrest

Resuscitation, 2012-07, Vol.83 (7), p.e162-e163

2012

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Potent phagocytic activity with impaired antigen presentation identifying lipopolysaccharide-tolerant human monocytes: demonstration in isolated monocytes from cystic fibrosis patients

The Journal of immunology (1950), 2009-08, Vol.183 (3), p.2194-2194

2009

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Rapid up-regulation of IRAK-M expression following a second endotoxin challenge in human monocytes and in monocytes isolated from septic patients

Biochemical and biophysical research communications, 2003-11, Vol.311 (2), p.465-472

2003

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Response to "In vivo attenuation and genetic evolution of a ST247-SCCmecI MRSA clone after 13 years of pathogenic bronchopulmonary colonization in a patient with cystic fibrosis: implications of the innate immune response"

Mucosal immunology, 2015-05, Vol.8 (3), p.697-698

2015

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Reduced hepatic expression of farnesoid X receptor in hereditary cholestasis associated to mutation in ATP8B1

Human molecular genetics, 2004-10, Vol.13 (20), p.2451-2460

2004

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Surveillance of Immunological Status after Vaccination by two Serological Assays based on SARS-CoV-2 Spike Protein

International journal of infectious diseases, 2022-03, Vol.116, p.S40-S41

2022

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Apoptosis induced by Rac GTPase correlates with induction of FasL and ceramides production

Molecular biology of the cell, 2000-12, Vol.11 (12), p.4347-4358

2000

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In vivo attenuation and genetic evolution of a ST247-SCCmecI MRSA clone after 13 years of pathogenic bronchopulmonary colonization in a patient with cystic fibrosis: implications of the innate immune response

Mucosal immunology, 2015-03, Vol.8 (2), p.362-371

2015

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Tumor Cells Deactivate Human Monocytes by Up-Regulating IL-1 Receptor Associated Kinase-M Expression via CD44 and TLR4

The Journal of immunology (1950), 2005-03, Vol.174 (5), p.3032-3040

2005

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Contribution of globular death domains and unstructured linkers to MyD88.IRAK-4 heterodimer formation: an explanation for the antagonistic activity of MyD88s

Biochemical and biophysical research communications, 2009-02, Vol.380 (1), p.183-187

2009

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Protective effect of cyclosporin A and FK506 from nitric oxide‐dependent apoptosis in activated macrophages

British journal of pharmacology, 1999-03, Vol.126 (5), p.1139-1146

1999

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Nitric oxide activates the expression of IRAK-M via the release of TNF-α in human monocytes

Nitric oxide, 2004-06, Vol.10 (4), p.213-220

2004

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6-Methylprednisolone down-regulates IRAK-M in human and murine osteoclasts and boosts bone-resorbing activity: a putative mechanism for corticoid-induced osteoporosis

Journal of leukocyte biology, 2007-09, Vol.82 (3), p.700-709

2007

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Triggering of Peritoneal Macrophages with IFN-α/β Attenuates the Expression of Inducible Nitric Oxide Synthase Through a Decrease in NF-κB Activation

The Journal of immunology (1950), 1998-03, Vol.160 (6), p.2889-2895

1998

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Liquid Biopsy Characterization of CTCs and THCs Isolated in Patients with UC

The Journal of Liquid Biopsy, 2023-11, Vol.1, p.100074, Article 100074

2023

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Attenuation of innate immune response due to a host-adapted cystic fibrosis ST-245-SCC mecI-MRSA strain compared with their isogenic ancestor

Journal of cystic fibrosis, 2010-06, Vol.9, p.S47-S47

2010

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Attenuation of innate immune response due to a host-adapted cystic fibrosis ST-245-SCC mec I-MRSA strain compared with their isogenic ancestor

Journal of cystic fibrosis, 2010, Vol.9, p.S47-S47

2010

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Interferon-alpha/beta inhibits the apoptosis induced by lipopolysaccharide and interferon-gamma in murine peritoneal macrophages

Journal of interferon & cytokine research, 1998-07, Vol.18 (7), p.461-467

1998

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Inflammatory responses associated with acute coronary syndrome up-regulate IRAK-M and induce endotoxin tolerance in circulating monocytes

Journal of endotoxin research, 2007-01, Vol.13 (1), p.39

2007

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Triggering of peritoneal macrophages with IFN- alpha / beta attenuates the expression of inducible nitric oxide synthase through a decrease in NF- Kappa B activation

The Journal of immunology (1950), 1998-03, Vol.160 (6), p.2887-2895

1998

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Requirement of nitric oxide and calcium mobilization for the induction of apoptosis in adrenal vascular endothelial cells

FEBS letters, 1997-08, Vol.413 (1), p.124-128

1997

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Epilepsy in cardiofaciocutaneous syndrome: Clinical burden and response to anti‐seizure medication

RNA induces unique tau strains and stabilizes Alzheimer’s disease seeds

Endotoxin tolerance: new mechanisms, molecules and clinical significance

Effects of CPAP on oxidative stress and nitrate efficiency in sleep apnoea: a randomised trial

Circulating cell-free mitochondrial DNA: A better early prognostic marker in patients with out-of-hospital cardiac arrest

Potent phagocytic activity with impaired antigen presentation identifying lipopolysaccharide-tolerant human monocytes: demonstration in isolated monocytes from cystic fibrosis patients

Rapid up-regulation of IRAK-M expression following a second endotoxin challenge in human monocytes and in monocytes isolated from septic patients

Response to "In vivo attenuation and genetic evolution of a ST247-SCCmecI MRSA clone after 13 years of pathogenic bronchopulmonary colonization in a patient with cystic fibrosis: implications of the innate immune response"

Reduced hepatic expression of farnesoid X receptor in hereditary cholestasis associated to mutation in ATP8B1

Surveillance of Immunological Status after Vaccination by two Serological Assays based on SARS-CoV-2 Spike Protein

Apoptosis induced by Rac GTPase correlates with induction of FasL and ceramides production

In vivo attenuation and genetic evolution of a ST247-SCCmecI MRSA clone after 13 years of pathogenic bronchopulmonary colonization in a patient with cystic fibrosis: implications of the innate immune response

Tumor Cells Deactivate Human Monocytes by Up-Regulating IL-1 Receptor Associated Kinase-M Expression via CD44 and TLR4

Contribution of globular death domains and unstructured linkers to MyD88.IRAK-4 heterodimer formation: an explanation for the antagonistic activity of MyD88s

Protective effect of cyclosporin A and FK506 from nitric oxide‐dependent apoptosis in activated macrophages

Nitric oxide activates the expression of IRAK-M via the release of TNF-α in human monocytes

6-Methylprednisolone down-regulates IRAK-M in human and murine osteoclasts and boosts bone-resorbing activity: a putative mechanism for corticoid-induced osteoporosis

Triggering of Peritoneal Macrophages with IFN-α/β Attenuates the Expression of Inducible Nitric Oxide Synthase Through a Decrease in NF-κB Activation

Liquid Biopsy Characterization of CTCs and THCs Isolated in Patients with UC

Attenuation of innate immune response due to a host-adapted cystic fibrosis ST-245-SCC mecI-MRSA strain compared with their isogenic ancestor

Attenuation of innate immune response due to a host-adapted cystic fibrosis ST-245-SCC mec I-MRSA strain compared with their isogenic ancestor

Interferon-alpha/beta inhibits the apoptosis induced by lipopolysaccharide and interferon-gamma in murine peritoneal macrophages

Inflammatory responses associated with acute coronary syndrome up-regulate IRAK-M and induce endotoxin tolerance in circulating monocytes

Triggering of peritoneal macrophages with IFN- alpha / beta attenuates the expression of inducible nitric oxide synthase through a decrease in NF- Kappa B activation

Requirement of nitric oxide and calcium mobilization for the induction of apoptosis in adrenal vascular endothelial cells

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