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HGF receptor associates with the anti‐apoptotic protein BAG‐1 and prevents cell death

The EMBO journal, 1996-11, Vol.15 (22), p.6205-6212

1996

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3D pancreatic carcinoma spheroids induce a matrix-rich, chemoresistant phenotype offering a better model for drug testing

BMC cancer, 2013-02, Vol.13 (1), p.95-95, Article 95

2013

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Cross-talk between the proto-oncogenes Met and Ron

Oncogene, 2000-06, Vol.19 (27), p.3041-3049

2000

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Phosphorylation of serine 985 negatively regulates the hepatocyte growth factor receptor kinase

The Journal of biological chemistry, 1994-01, Vol.269 (3), p.1815-1820

1994

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A Family of Transmembrane Proteins with Homology to the MET-Hepatocyte Growth Factor Receptor

Proceedings of the National Academy of Sciences - PNAS, 1996-01, Vol.93 (2), p.674-678

1996

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Receptor tyrosine kinases as therapeutic targets: the model of the MET oncogene

Current drug targets, 2001-03, Vol.2 (1), p.41-55

2001

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Identification of the major autophosphorylation site of the Met/hepatocyte growth factor receptor tyrosine kinase

The Journal of biological chemistry, 1991-10, Vol.266 (29), p.19558-19564

1991

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A novel recognition motif for phosphatidylinositol 3-kinase binding mediates its association with the hepatocyte growth factor/scatter factor receptor

Molecular and Cellular Biology, 1993-08, Vol.13 (8), p.4600-4608

1993

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C-terminal truncated forms of Met, the hepatocyte growth factor receptor

Molecular and Cellular Biology, 1991-12, Vol.11 (12), p.5954-5962

1991

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Stabilin-1 and -2 constitute a novel family of fasciclin-like hyaluronan receptor homologues

Biochemical journal, 2002-02, Vol.362 (Pt 1), p.155-164

2002

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The tyrosine kinase encoded by the MET proto-oncogene is activated by autophosphorylation

Molecular and Cellular Biology, 1991-04, Vol.11 (4), p.1793-1803

1991

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Stabilin-1 and stabilin-2 are both directed into the early endocytic pathway in hepatic sinusoidal endothelium via interactions with clathrin/AP-2, independent of ligand binding

Experimental cell research, 2005-02, Vol.303 (1), p.160-173

2005

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Structural and functional domains critical for constitutive activation of the HGF-receptor (Met)

Oncogene, 1994-06, Vol.9 (6), p.1691-1697

1994

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Functional Regulation of Semaphorin Receptors by Proprotein Convertases

The Journal of biological chemistry, 2003-03, Vol.278 (12), p.10094-10101

2003

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Gab1 coupling to the HGF/Met receptor multifunctional docking site requires binding of Grb2 and correlates with the transforming potential

Oncogene, 1997-12, Vol.15 (25), p.3103-3111

1997

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Uncoupling Signal Transducers from Oncogenic MET Mutants Abrogates Cell Transformation and Inhibits Invasive Growth

Proceedings of the National Academy of Sciences - PNAS, 1998-11, Vol.95 (24), p.14379-14383

1998

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Hepatocyte growth factor and its receptor, the tyrosine kinase encoded by the c-MET proto-oncogene

Cellular and molecular biology (Noisy-le-Grand, France), 1994-07, Vol.40 (5), p.597

1994

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Loss of the exon encoding the juxtamembrane domain is essential for the oncogenic activation of TPR-MET

Oncogene, 1999-07, Vol.18 (29), p.4275-4281

1999

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A Peptide Representing the Carboxyl-terminal Tail of the Met Receptor Inhibits Kinase Activity and Invasive Growth

The Journal of biological chemistry, 1999-10, Vol.274 (41), p.29274-29281

1999

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Novel mutation in the ATP‐binding site of the MET oncogene tyrosine kinase in a HPRCC family

International journal of cancer, 1999-08, Vol.82 (5), p.640-643

1999

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The role of hypoxia on the tumor-stromal interaction in pancreatic ductal adenocarcinoma

CANCER RESEARCH, 2012, Vol.72

2012

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MET(PRC) mutations in the Ron receptor result in upregulation of tyrosine kinase activity and acquisition of oncogenic potential

Journal of cellular physiology, 1999-12, Vol.181 (3), p.507

1999

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METPRC mutations in the ron receptor result in upregulation of tyrosine kinase activity and acquisition of oncogenic potential

Journal of cellular physiology, 1999-12, Vol.181 (3), p.507-514

1999

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Cross-talk between the proto-oncogenes Metand Ron

Oncogene, 2000-06, Vol.19 (27), p.3041

2000

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Tyrosines1234-1235 are critical for activation of the tyrosine kinase encoded by the MET proto-oncogene (HGF receptor)

Oncogene, 1994, Vol.9 (1), p.49-57

1994

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HGF receptor associates with the anti‐apoptotic protein BAG‐1 and prevents cell death

3D pancreatic carcinoma spheroids induce a matrix-rich, chemoresistant phenotype offering a better model for drug testing

Cross-talk between the proto-oncogenes Met and Ron

Phosphorylation of serine 985 negatively regulates the hepatocyte growth factor receptor kinase

A Family of Transmembrane Proteins with Homology to the MET-Hepatocyte Growth Factor Receptor

Receptor tyrosine kinases as therapeutic targets: the model of the MET oncogene

Identification of the major autophosphorylation site of the Met/hepatocyte growth factor receptor tyrosine kinase

A novel recognition motif for phosphatidylinositol 3-kinase binding mediates its association with the hepatocyte growth factor/scatter factor receptor

C-terminal truncated forms of Met, the hepatocyte growth factor receptor

Stabilin-1 and -2 constitute a novel family of fasciclin-like hyaluronan receptor homologues

The tyrosine kinase encoded by the MET proto-oncogene is activated by autophosphorylation

Stabilin-1 and stabilin-2 are both directed into the early endocytic pathway in hepatic sinusoidal endothelium via interactions with clathrin/AP-2, independent of ligand binding

Structural and functional domains critical for constitutive activation of the HGF-receptor (Met)

Functional Regulation of Semaphorin Receptors by Proprotein Convertases

Gab1 coupling to the HGF/Met receptor multifunctional docking site requires binding of Grb2 and correlates with the transforming potential

Uncoupling Signal Transducers from Oncogenic MET Mutants Abrogates Cell Transformation and Inhibits Invasive Growth

Hepatocyte growth factor and its receptor, the tyrosine kinase encoded by the c-MET proto-oncogene

Loss of the exon encoding the juxtamembrane domain is essential for the oncogenic activation of TPR-MET

A Peptide Representing the Carboxyl-terminal Tail of the Met Receptor Inhibits Kinase Activity and Invasive Growth

Novel mutation in the ATP‐binding site of the MET oncogene tyrosine kinase in a HPRCC family

The role of hypoxia on the tumor-stromal interaction in pancreatic ductal adenocarcinoma

MET(PRC) mutations in the Ron receptor result in upregulation of tyrosine kinase activity and acquisition of oncogenic potential

METPRC mutations in the ron receptor result in upregulation of tyrosine kinase activity and acquisition of oncogenic potential

Cross-talk between the proto-oncogenes Metand Ron

Tyrosines1234-1235 are critical for activation of the tyrosine kinase encoded by the MET proto-oncogene (HGF receptor)

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