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The Physics of Cellular Decision Making During Epithelial-Mesenchymal Transition

Annual review of biophysics, 2020-05, Vol.49 (1), p.1-18

2020

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A mechanism for epithelial-mesenchymal heterogeneity in a population of cancer cells

PLoS computational biology, 2020-02, Vol.16 (2), p.e1007619-e1007619

2020

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Cluster size distribution of cells disseminating from a primary tumor

PLoS computational biology, 2021-11, Vol.17 (11), p.e1009011-e1009011

2021

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Coupling actin flow, adhesion, and morphology in a computational cell motility model

Proceedings of the National Academy of Sciences - PNAS, 2012-05, Vol.109 (18), p.6851-6856

2012

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Survival Outcomes in Cancer Patients Predicted by a Partial EMT Gene Expression Scoring Metric

Cancer research (Chicago, Ill.), 2017-11, Vol.77 (22), p.6415-6428

2017

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Epithelial–mesenchymal transition, a spectrum of states: Role in lung development, homeostasis, and disease

Developmental dynamics, 2018-03, Vol.247 (3), p.346-358

2018

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Identification of EMT signaling cross-talk and gene regulatory networks by single-cell RNA sequencing

Proceedings of the National Academy of Sciences - PNAS, 2021-05, Vol.118 (19), p.1

2021

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Toward understanding cancer stem cell heterogeneity in the tumor microenvironment

Proceedings of the National Academy of Sciences - PNAS, 2019-01, Vol.116 (1), p.148-157

2019

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MicroRNA-based regulation of epithelial–hybrid–mesenchymal fate determination

Proceedings of the National Academy of Sciences - PNAS, 2013-11, Vol.110 (45), p.18144-18149

2013

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Mechanically-driven phase separation in a growing bacterial colony

Proceedings of the National Academy of Sciences - PNAS, 2015-04, Vol.112 (17), p.E2166-E2173

2015

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EMT and MET: necessary or permissive for metastasis?

Molecular oncology, 2017-07, Vol.11 (7), p.755-769

2017

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Alignment of cellular motility forces with tissue flow as a mechanism for efficient wound healing

Proceedings of the National Academy of Sciences - PNAS, 2013-02, Vol.110 (7), p.2452-2459

2013

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Contact inhibition of locomotion determines cell–cell and cell–substrate forces in tissues

Proceedings of the National Academy of Sciences - PNAS, 2016-03, Vol.113 (10), p.2660-2665

2016

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Physics of Molecular and Cellular Processes

1, 2022

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Modeling the Genetic Regulation of Cancer Metabolism: Interplay between Glycolysis and Oxidative Phosphorylation

Cancer research (Chicago, Ill.), 2017-04, Vol.77 (7), p.1564-1574

2017

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Phenotypic Switching: Implications in Biology and Medicine

1, 2020

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Interrogating the topological robustness of gene regulatory circuits by randomization

PLoS computational biology, 2017-03, Vol.13 (3), p.e1005456-e1005456

2017

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Polarity mechanisms such as contact inhibition of locomotion regulate persistent rotational motion of mammalian cells on micropatterns

Proceedings of the National Academy of Sciences - PNAS, 2014-10, Vol.111 (41), p.14770-14775

2014

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Phenotypic Plasticity, Bet-Hedging, and Androgen Independence in Prostate Cancer: Role of Non-Genetic Heterogeneity

Frontiers in oncology, 2018-03, Vol.8, p.50-50

2018

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Crawling and turning in a minimal reaction-diffusion cell motility model: Coupling cell shape and biochemistry

Physical review. E, 2017-01, Vol.95 (1-1), p.012401-012401, Article 012401

2017

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Stability of the hybrid epithelial/mesenchymal phenotype

Oncotarget, 2016-05, Vol.7 (19), p.27067-27084

2016

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Stress-induced plasticity of dynamic collagen networks

Nature communications, 2017-10, Vol.8 (1), p.842-7, Article 842

2017

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The GRHL2/ZEB Feedback Loop—A Key Axis in the Regulation of EMT in Breast Cancer

Journal of cellular biochemistry, 2017-09, Vol.118 (9), p.2559-2570

2017

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Optimal cancer evasion in a dynamic immune microenvironment generates diverse post-escape tumor antigenicity profiles

eLife, 2023-04, Vol.12

2023

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Large population solution of the stochastic Luria–Delbrück evolution model

Proceedings of the National Academy of Sciences - PNAS, 2013-07, Vol.110 (29), p.11682-11687

2013

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The Physics of Cellular Decision Making During Epithelial-Mesenchymal Transition

A mechanism for epithelial-mesenchymal heterogeneity in a population of cancer cells

Cluster size distribution of cells disseminating from a primary tumor

Coupling actin flow, adhesion, and morphology in a computational cell motility model

Survival Outcomes in Cancer Patients Predicted by a Partial EMT Gene Expression Scoring Metric

Epithelial–mesenchymal transition, a spectrum of states: Role in lung development, homeostasis, and disease

Identification of EMT signaling cross-talk and gene regulatory networks by single-cell RNA sequencing

Toward understanding cancer stem cell heterogeneity in the tumor microenvironment

MicroRNA-based regulation of epithelial–hybrid–mesenchymal fate determination

Mechanically-driven phase separation in a growing bacterial colony

EMT and MET: necessary or permissive for metastasis?

Alignment of cellular motility forces with tissue flow as a mechanism for efficient wound healing

Contact inhibition of locomotion determines cell–cell and cell–substrate forces in tissues

Physics of Molecular and Cellular Processes

Modeling the Genetic Regulation of Cancer Metabolism: Interplay between Glycolysis and Oxidative Phosphorylation

Phenotypic Switching: Implications in Biology and Medicine

Interrogating the topological robustness of gene regulatory circuits by randomization

Polarity mechanisms such as contact inhibition of locomotion regulate persistent rotational motion of mammalian cells on micropatterns

Phenotypic Plasticity, Bet-Hedging, and Androgen Independence in Prostate Cancer: Role of Non-Genetic Heterogeneity

Crawling and turning in a minimal reaction-diffusion cell motility model: Coupling cell shape and biochemistry

Stability of the hybrid epithelial/mesenchymal phenotype

Stress-induced plasticity of dynamic collagen networks

The GRHL2/ZEB Feedback Loop—A Key Axis in the Regulation of EMT in Breast Cancer

Optimal cancer evasion in a dynamic immune microenvironment generates diverse post-escape tumor antigenicity profiles

Large population solution of the stochastic Luria–Delbrück evolution model

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