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Rosiglitazone Rescues Memory Impairment in Alzheimer's Transgenic Mice: Mechanisms Involving a Reduced Amyloid and Tau Pathology

Neuropsychopharmacology (New York, N.Y.), 2010-06, Vol.35 (7), p.1593-1604

2010

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LRRK2 Expression Is Deregulated in Fibroblasts and Neurons from Parkinson Patients with Mutations in PINK1

Molecular neurobiology, 2018-01, Vol.55 (1), p.506-516

2018

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LRRK2 G2019S mutant exacerbates basal autophagy through activation of the MEK/ERK pathway

Cellular and molecular life sciences : CMLS, 2013-01, Vol.70 (1), p.121-136

2013

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Gonadotropin-Releasing Hormone Analog Structural Determinants of Selectivity for Inhibition of Cell Growth: Support for the Concept of Ligand-Induced Selective Signaling

Molecular endocrinology (Baltimore, Md.), 2008-07, Vol.22 (7), p.1711-1722

2008

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iPS Cell Cultures from a Gerstmann-Sträussler-Scheinker Patient with the Y218N PRNP Mutation Recapitulate tau Pathology

Molecular neurobiology, 2018-04, Vol.55 (4), p.3033-3048

2018

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Early changes in hippocampal Eph receptors precede the onset of memory decline in mouse models of Alzheimer's disease

Journal of Alzheimer's disease, 2009-01, Vol.17 (4), p.773

2009

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Overexpression of wild-type human APP in mice causes cognitive deficits and pathological features unrelated to Aβ levels

Neurobiology of disease, 2009-03, Vol.33 (3), p.369-378

2009

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Gonadotropin-releasing hormone (GnRH) antagonists promote proapoptotic signaling in peripheral reproductive tumor cells by activating a Gαi-Coupling state of the type I GnRH receptor

Cancer research (Chicago, Ill.), 2004-10, Vol.64 (20), p.7533-7544

2004

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The Isolated N-terminal Domain of the Glucagon-like Peptide-1 (GLP-1) Receptor Binds Exendin Peptides with Much Higher Affinity than GLP-1

The Journal of biological chemistry, 2003-03, Vol.278 (12), p.10195-10200

2003

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The MAPK1/3 pathway is essential for the deregulation of autophagy observed in G2019S LRRK2 mutant fibroblasts

Autophagy, 2012-10, Vol.8 (10), p.1537-1539

2012

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Regulation of corticostriatal synaptic plasticity by G protein-coupled receptors

CNS & neurological disorders drug targets, 2010-11, Vol.9 (5), p.601

2010

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Selection Based on FOXA2 Expression Is Not Sufficient to Enrich for Dopamine Neurons From Human Pluripotent Stem Cells

Stem cells translational medicine, 2014-09, Vol.3 (9), p.1032-1042

2014

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Functional coupling of Cys-226 and Cys-296 in the glucagon-like peptide-1 (GLP-1) receptor indicates a disulfide bond that is close to the activation pocket

Peptides (New York, N.Y. : 1980), 2010-12, Vol.31 (12), p.2289-2293

2010

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GnRH-mediated DAN production regulates the transcription of the GnRH receptor in gonadotrope cells

Neuromolecular medicine, 2007-09, Vol.9 (3), p.230-248

2007

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Class II G protein-coupled receptors and their ligands in neuronal function and protection

Neuromolecular medicine, 2005, Vol.7 (1-2), p.3-36

2005

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Gonadotropin-releasing Hormone-induced Activation of Diacylglycerol Kinase-ζ and Its Association with Active c-Src

The Journal of biological chemistry, 2004-03, Vol.279 (12), p.11906-11916

2004

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Gonadotropin-Releasing Hormone Functionally Antagonizes Testosterone Activation of the Human Androgen Receptor in Prostate Cells through Focal Adhesion Complexes Involving Hic-5

Neuroendocrinology, 2006-01, Vol.84 (5), p.285-300

2006

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Met-204 and Tyr-205 are together important for binding GLP-1 receptor agonists but not their N-terminally truncated analogues

Protein and peptide letters, 2004-02, Vol.11 (1), p.15

2004

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Sustained Increase of PKA Activity in the Postcommissural Putamen of Dyskinetic Monkeys

Molecular neurobiology, 2014-12, Vol.50 (3), p.1131-1141

2014

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The glucagon-like peptide-1 receptor binding site for the N-terminus of GLP-1 requires polarity at Asp198 rather than negative charge

FEBS letters, 2002-10, Vol.530 (1), p.244-248

2002

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Leucine-rich repeat kinase 2 modulates cyclooxygenase 2 and the inflammatory response in idiopathic and genetic Parkinson's disease

Neurobiology of aging, 2014-05, Vol.35 (5), p.1116-1124

2014

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Erratum to: The glucagon-like peptide-1 receptor binding site for the N-terminus of GLP-1 requires polarity at Asp198 rather than negative charge (FEBS 26658) : [ FEBS Letters 530 (2002) 244–248]

FEBS letters, 2002-11, Vol.531 (3), p.572-572

2002

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Erratum to: The glucagon‐like peptide‐1 receptor binding site for the N‐terminus of GLP‐1 requires polarity at Asp198 rather than negative charge (FEBS 26658): FEBS Letters 530 (2002) 244–248

FEBS letters, 2002-11, Vol.531 (3), p.572-572

2002

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Gonadotropin-releasing hormone (GnRH) antagonists promote proapoptotic signaling in peripheral reproductive tumor cells by activating a Galphai-coupling state of the type I GnRH receptor

Cancer research (Chicago, Ill.), 2004-10, Vol.64 (20), p.7533-7544

2004

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Mutations in LRRK2 impair NF-KappaB pathway in iPSC-derived neurons

Journal of neuroinflammation, 2016-01, Vol.13

2016

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Rosiglitazone Rescues Memory Impairment in Alzheimer's Transgenic Mice: Mechanisms Involving a Reduced Amyloid and Tau Pathology

LRRK2 Expression Is Deregulated in Fibroblasts and Neurons from Parkinson Patients with Mutations in PINK1

LRRK2 G2019S mutant exacerbates basal autophagy through activation of the MEK/ERK pathway

Gonadotropin-Releasing Hormone Analog Structural Determinants of Selectivity for Inhibition of Cell Growth: Support for the Concept of Ligand-Induced Selective Signaling

iPS Cell Cultures from a Gerstmann-Sträussler-Scheinker Patient with the Y218N PRNP Mutation Recapitulate tau Pathology

Early changes in hippocampal Eph receptors precede the onset of memory decline in mouse models of Alzheimer's disease

Overexpression of wild-type human APP in mice causes cognitive deficits and pathological features unrelated to Aβ levels

Gonadotropin-releasing hormone (GnRH) antagonists promote proapoptotic signaling in peripheral reproductive tumor cells by activating a Gαi-Coupling state of the type I GnRH receptor

The Isolated N-terminal Domain of the Glucagon-like Peptide-1 (GLP-1) Receptor Binds Exendin Peptides with Much Higher Affinity than GLP-1

The MAPK1/3 pathway is essential for the deregulation of autophagy observed in G2019S LRRK2 mutant fibroblasts

Regulation of corticostriatal synaptic plasticity by G protein-coupled receptors

Selection Based on FOXA2 Expression Is Not Sufficient to Enrich for Dopamine Neurons From Human Pluripotent Stem Cells

Functional coupling of Cys-226 and Cys-296 in the glucagon-like peptide-1 (GLP-1) receptor indicates a disulfide bond that is close to the activation pocket

GnRH-mediated DAN production regulates the transcription of the GnRH receptor in gonadotrope cells

Class II G protein-coupled receptors and their ligands in neuronal function and protection

Gonadotropin-releasing Hormone-induced Activation of Diacylglycerol Kinase-ζ and Its Association with Active c-Src

Gonadotropin-Releasing Hormone Functionally Antagonizes Testosterone Activation of the Human Androgen Receptor in Prostate Cells through Focal Adhesion Complexes Involving Hic-5

Met-204 and Tyr-205 are together important for binding GLP-1 receptor agonists but not their N-terminally truncated analogues

Sustained Increase of PKA Activity in the Postcommissural Putamen of Dyskinetic Monkeys

The glucagon-like peptide-1 receptor binding site for the N-terminus of GLP-1 requires polarity at Asp198 rather than negative charge

Leucine-rich repeat kinase 2 modulates cyclooxygenase 2 and the inflammatory response in idiopathic and genetic Parkinson's disease

Erratum to: The glucagon-like peptide-1 receptor binding site for the N-terminus of GLP-1 requires polarity at Asp198 rather than negative charge (FEBS 26658) : [ FEBS Letters 530 (2002) 244–248]

Erratum to: The glucagon‐like peptide‐1 receptor binding site for the N‐terminus of GLP‐1 requires polarity at Asp198 rather than negative charge (FEBS 26658): FEBS Letters 530 (2002) 244–248

Gonadotropin-releasing hormone (GnRH) antagonists promote proapoptotic signaling in peripheral reproductive tumor cells by activating a Galphai-coupling state of the type I GnRH receptor

Mutations in LRRK2 impair NF-KappaB pathway in iPSC-derived neurons

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