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Bioavailability of dietary isoquercitrin-γ-cyclodextrin molecular inclusion complex in Sprague–Dawley rats and healthy humans

Journal of functional foods, 2021-10, Vol.85, p.104663, Article 104663

2021

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Increased bioavailability of diosmetin-7-glucoside-γ-cyclodextrin inclusion complex compared with diosmin in Sprague-Dawley rats

Bioscience, biotechnology, and biochemistry, 2023-06, Vol.87 (7), p.771-776

2023

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Lack of hepatocarcinogenicity of 2,2’-[1,2-ethanediylbis(oxymethylene)]bis-oxirane, 3-hydroxy-2-naphthoic acid, and acetoacetanilide in a medium-term rat liver bioassay

Journal of Toxicologic Pathology, 2022, Vol.35(4), pp.313-320

2022

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In vivo mutagenicity and initiation following oxidative DNA lesion in the kidneys of rats given potassium bromate

Cancer science, 2006-09, Vol.97 (9), p.829-835

2006

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A Crucial Role of Nrf2 in In Vivo Defense against Oxidative Damage by an Environmental Pollutant, Pentachlorophenol

Toxicological sciences, 2006-03, Vol.90 (1), p.111-119

2006

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Lack of nrf2 results in progression of proliferative lesions to neoplasms induced by long-term exposure to non-genotoxic hepatocarcinogens involving oxidative stress

Experimental and toxicologic pathology : official journal of the Gesellschaft für Toxikologische Pathologie, 2014-01, Vol.66 (1), p.19-26

2014

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Transition of Historical Control Data for High Incidence Tumors in F344 Rats

Journal of Toxicologic Pathology, 2013, Vol.26(2), pp.227-230

2013

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Spontaneous convulsions in Sprague-Dawley rats in carcinogenicity studies

The Journal of Toxicological Sciences, 2012/06/01, Vol.37(3), pp.645-647

2012

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Enhancement of esophageal carcinogenesis in acid reflux model rats treated with ascorbic acid and sodium nitrite in combination with or without initiation

Cancer science, 2008-01, Vol.99 (1), p.7-13

2008

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A Possible Role of Nrf2 in Prevention of Renal Oxidative Damage by Ferric Nitrilotriacetate

Toxicologic pathology, 2008-02, Vol.36 (2), p.353-361

2008

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Antigenotoxic effects of p53 on spontaneous and ultraviolet light B-induced deletions in the epidermis of gpt delta transgenic mice

Environmental and molecular mutagenesis, 2011-04, Vol.52 (3), p.244-252

2011

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Combined treatment with green tea catechins and sodium nitrite selectively promotes rat forestomach carcinogenesis after initiation with N‐methyl‐N′‐ nitro‐N‐nitrosoguanidine

Cancer science, 2007-07, Vol.98 (7), p.949-957

2007

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Detection of oxidative DNA damage, cell proliferation and in vivo mutagenicity induced by dicyclanil, a non-genotoxic carcinogen, using gpt delta mice

Mutation research, 2007-09, Vol.633 (1), p.46-54

2007

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Lung as a new target in rats of 2‐amino‐3‐methylimidazo[4,5‐f]quinoline carcinogenesis: Results of a two‐stage model initiated with N‐bis(2‐hydroxypropyl)nitrosamine

Cancer science, 2006-05, Vol.97 (5), p.368-373

2006

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Combined Ascorbic Acid and Sodium Nitrite Treatment Induces Oxidative DNA Damage-Associated Mutagenicity In Vitro, but Lacks Initiation Activity in Rat Forestomach Epithelium

Toxicological sciences, 2008-08, Vol.104 (2), p.274-282

2008

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Lack of in vivo mutagenicity and oxidative DNA damage by flumequine in the livers of gpt delta mice

Archives of toxicology, 2007-01, Vol.81 (1), p.63-69

2007

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Bioavailability of Flavonoids in Ginkgo biloba Extract-γ-Cyclodextrin Complex

Natural product communications, 2023-05, Vol.18 (5)

2023

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Nine-week detection of six genotoxic lung carcinogens using the rasH2/BHT mouse model

Cancer letters, 2006-01, Vol.231 (2), p.314-318

2006

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Lack of hepatocarcinogenicity of 2,2'-[1,2-ethanediylbis(oxymethylene)]bis-oxirane, 3-hydroxy-2-naphthoic acid, and acetoacetanilide in a medium-term rat liver bioassay

Journal of Toxicologic Pathology, 2022, pp.2022-0010

2022

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Protective effects of benzyl isothiocyanate and sulforaphane but not resveratrol against initiation of pancreatic carcinogenesis in hamsters

Cancer letters, 2006-09, Vol.241 (2), p.275-280

2006

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Oxidative DNA damage and in vivo mutagenicity caused by reactive oxygen species generated in the livers of p53-proficient or -deficient gpt delta mice treated with non-genotoxic hepatocarcinogens

Journal of applied toxicology, 2013-12, Vol.33 (12), p.1433-1441

2013

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Possible involvement of oxidative stress in piperonyl butoxide induced hepatocarcinogenesis in rats

Toxicology (Amsterdam), 2007-07, Vol.236 (1), p.61-75

2007

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Effect of Fenofibrate on Oxidative DNA Damage and on Gene Expression Related to Cell Proliferation and Apoptosis in Rats

Toxicological sciences, 2007-05, Vol.97 (1), p.44-54

2007

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Induction of characteristic hepatocyte proliferative lesion with dietary exposure of Wistar Hannover rats to tocotrienol for 1 year

Toxicology (Amsterdam), 2008-09, Vol.250 (2), p.143-150

2008

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Simultaneous induction of non-neoplastic and neoplastic lesions with highly proliferative hepatocytes following dietary exposure of rats to tocotrienol for 2 years

Archives of toxicology, 2009-11, Vol.83 (11), p.1021-1030

2009

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Bioavailability of dietary isoquercitrin-γ-cyclodextrin molecular inclusion complex in Sprague–Dawley rats and healthy humans

Increased bioavailability of diosmetin-7-glucoside-γ-cyclodextrin inclusion complex compared with diosmin in Sprague-Dawley rats

Lack of hepatocarcinogenicity of 2,2’-[1,2-ethanediylbis(oxymethylene)]bis-oxirane, 3-hydroxy-2-naphthoic acid, and acetoacetanilide in a medium-term rat liver bioassay

In vivo mutagenicity and initiation following oxidative DNA lesion in the kidneys of rats given potassium bromate

A Crucial Role of Nrf2 in In Vivo Defense against Oxidative Damage by an Environmental Pollutant, Pentachlorophenol

Lack of nrf2 results in progression of proliferative lesions to neoplasms induced by long-term exposure to non-genotoxic hepatocarcinogens involving oxidative stress

Transition of Historical Control Data for High Incidence Tumors in F344 Rats

Spontaneous convulsions in Sprague-Dawley rats in carcinogenicity studies

Enhancement of esophageal carcinogenesis in acid reflux model rats treated with ascorbic acid and sodium nitrite in combination with or without initiation

A Possible Role of Nrf2 in Prevention of Renal Oxidative Damage by Ferric Nitrilotriacetate

Antigenotoxic effects of p53 on spontaneous and ultraviolet light B-induced deletions in the epidermis of gpt delta transgenic mice

Combined treatment with green tea catechins and sodium nitrite selectively promotes rat forestomach carcinogenesis after initiation with N‐methyl‐N′‐ nitro‐N‐nitrosoguanidine

Detection of oxidative DNA damage, cell proliferation and in vivo mutagenicity induced by dicyclanil, a non-genotoxic carcinogen, using gpt delta mice

Lung as a new target in rats of 2‐amino‐3‐methylimidazo[4,5‐f]quinoline carcinogenesis: Results of a two‐stage model initiated with N‐bis(2‐hydroxypropyl)nitrosamine

Combined Ascorbic Acid and Sodium Nitrite Treatment Induces Oxidative DNA Damage-Associated Mutagenicity In Vitro, but Lacks Initiation Activity in Rat Forestomach Epithelium

Lack of in vivo mutagenicity and oxidative DNA damage by flumequine in the livers of gpt delta mice

Bioavailability of Flavonoids in Ginkgo biloba Extract-γ-Cyclodextrin Complex

Nine-week detection of six genotoxic lung carcinogens using the rasH2/BHT mouse model

Lack of hepatocarcinogenicity of 2,2'-[1,2-ethanediylbis(oxymethylene)]bis-oxirane, 3-hydroxy-2-naphthoic acid, and acetoacetanilide in a medium-term rat liver bioassay

Protective effects of benzyl isothiocyanate and sulforaphane but not resveratrol against initiation of pancreatic carcinogenesis in hamsters

Oxidative DNA damage and in vivo mutagenicity caused by reactive oxygen species generated in the livers of p53-proficient or -deficient gpt delta mice treated with non-genotoxic hepatocarcinogens

Possible involvement of oxidative stress in piperonyl butoxide induced hepatocarcinogenesis in rats

Effect of Fenofibrate on Oxidative DNA Damage and on Gene Expression Related to Cell Proliferation and Apoptosis in Rats

Induction of characteristic hepatocyte proliferative lesion with dietary exposure of Wistar Hannover rats to tocotrienol for 1 year

Simultaneous induction of non-neoplastic and neoplastic lesions with highly proliferative hepatocytes following dietary exposure of rats to tocotrienol for 2 years

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