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Small Heat Shock Proteins, Amyloid Fibrils, and Nicotine Stimulate a Common Immune Suppressive Pathway with Implications for Future Therapies

Cold Spring Harbor perspectives in medicine, 2019-07, Vol.9 (7), p.a034223

2019

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Self-Assembling Peptides Form Immune Suppressive Amyloid Fibrils Effective in Autoimmune Encephalomyelitis

Current topics in behavioral neurosciences, 2015-01, Vol.26, p.221

2015

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Mechanisms of action of therapeutic amyloidogenic hexapeptides in amelioration of inflammatory brain disease

The Journal of experimental medicine, 2014-08, Vol.211 (9), p.1847-1856

2014

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Janus Faces of Amyloid Proteins in Neuroinflammation

Journal of clinical immunology, 2014-07, Vol.34 (Suppl 1), p.61-63

2014

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Amyloid fibrils composed of hexameric peptides attenuate neuroinflammation

Science translational medicine, 2013-04, Vol.5 (179), p.179ra42

2013

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Piet Mondrian’s trees and the evolution in understanding multiple sclerosis, Charcot Prize Lecture 2011

Multiple sclerosis, 2013-01, Vol.19 (1), p.5-14

2013

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Chaperone Activity of Small Heat Shock Proteins Underlies Therapeutic Efficacy in Experimental Autoimmune Encephalomyelitis

The Journal of biological chemistry, 2012-10, Vol.287 (43), p.36423-36434

2012

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Therapeutic Effects of Systemic Administration of Chaperone αB-Crystallin Associated with Binding Proinflammatory Plasma Proteins

The Journal of biological chemistry, 2012-03, Vol.287 (13), p.9708-9721

2012

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Reductions in motor unit number estimates (MUNE) precede motor neuron loss in the plasma membrane calcium ATPase 2 (PMCA2)-heterozygous mice

Experimental neurology, 2008-12, Vol.214 (2), p.341-346

2008

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Identification of Differentially Expressed Proteins in Experimental Autoimmune Encephalomyelitis (EAE) by Proteomic Analysis of the Spinal Cord

Journal of proteome research, 2007-07, Vol.6 (7), p.2565-2575

2007

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Role of Plasma Membrane Calcium ATPase Isoform 2 in Neuronal Function in the Cerebellum and Spinal Cord

Annals of the New York Academy of Sciences, 2007-03, Vol.1099 (1), p.287-291

2007

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Molecular alterations in the cerebellum of the plasma membrane calcium ATPase 2 (PMCA2)-null mouse indicate abnormalities in Purkinje neurons

Molecular and cellular neuroscience, 2007-02, Vol.34 (2), p.178-188

2007

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Plasma membrane calcium ATPase deficiency causes neuronal pathology in the spinal cord: a potential mechanism for neurodegeneration in multiple sclerosis and spinal cord injury

The FASEB journal, 2005-02, Vol.19 (2), p.298-300

2005

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Calcium responses of chicken trigeminal ganglion neurons to methyl anthranilate and capsaicin

Journal of experimental biology, 2004-02, Vol.207 (Pt 5), p.715-722

2004

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Small Heat Shock Proteins, Amyloid Fibrils, and Nicotine Stimulate a Common Immune Suppressive Pathway with Implications for Future Therapies

Self-Assembling Peptides Form Immune Suppressive Amyloid Fibrils Effective in Autoimmune Encephalomyelitis

Mechanisms of action of therapeutic amyloidogenic hexapeptides in amelioration of inflammatory brain disease

Janus Faces of Amyloid Proteins in Neuroinflammation

Amyloid fibrils composed of hexameric peptides attenuate neuroinflammation

Piet Mondrian’s trees and the evolution in understanding multiple sclerosis, Charcot Prize Lecture 2011

Chaperone Activity of Small Heat Shock Proteins Underlies Therapeutic Efficacy in Experimental Autoimmune Encephalomyelitis

Therapeutic Effects of Systemic Administration of Chaperone αB-Crystallin Associated with Binding Proinflammatory Plasma Proteins

Reductions in motor unit number estimates (MUNE) precede motor neuron loss in the plasma membrane calcium ATPase 2 (PMCA2)-heterozygous mice

Identification of Differentially Expressed Proteins in Experimental Autoimmune Encephalomyelitis (EAE) by Proteomic Analysis of the Spinal Cord

Role of Plasma Membrane Calcium ATPase Isoform 2 in Neuronal Function in the Cerebellum and Spinal Cord

Molecular alterations in the cerebellum of the plasma membrane calcium ATPase 2 (PMCA2)-null mouse indicate abnormalities in Purkinje neurons

Plasma membrane calcium ATPase deficiency causes neuronal pathology in the spinal cord: a potential mechanism for neurodegeneration in multiple sclerosis and spinal cord injury

Calcium responses of chicken trigeminal ganglion neurons to methyl anthranilate and capsaicin

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