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Notch3 signaling between myeloma cells and osteocytes in the tumor niche promotes tumor growth and bone destruction

Neoplasia (New York, N.Y.), 2022-06, Vol.28, p.100785-100785, Article 100785

2022

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Increased S1P expression in osteoclasts enhances bone formation in an animal model of Paget's disease

Journal of cellular biochemistry, 2021-04, Vol.122 (3-4), p.335-348

2021

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Contributions of the Measles Virus Nucleocapsid Gene and the SQSTM1/p62P392L Mutation to Paget's Disease

Cell metabolism, 2011-01, Vol.13 (1), p.23-34

2011

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Activating transcription factor 4 regulates osteoclast differentiation in mice

The Journal of clinical investigation, 2010-08, Vol.120 (8), p.2755-2766

2010

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Thalidomide derivative CC-4047 inhibits osteoclast formation by down-regulation of PU.1

Blood, 2006-04, Vol.107 (8), p.3098-3105

2006

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A SQSTM1/p62 mutation linked to Paget’s disease increases the osteoclastogenic potential of the bone microenvironment

Human molecular genetics, 2008-12, Vol.17 (23), p.3708-3719

2008

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Osteoclast-derived IGF1 induces RANKL production in osteocytes and contributes to pagetic lesion formation

JCI insight, 2023-07, Vol.8 (14)

2023

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Growth factor independence 1 expression in myeloma cells enhances their growth, survival, and osteoclastogenesis

Journal of hematology and oncology, 2018-10, Vol.11 (1), p.123-123, Article 123

2018

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Increased signaling through p62 in the marrow microenvironment increases myeloma cell growth and osteoclast formation

Blood, 2009-05, Vol.113 (20), p.4894-4902

2009

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Expression of Measles Virus Nucleocapsid Protein in Osteoclasts Induces Paget's Disease‐Like Bone Lesions in Mice

Journal of bone and mineral research, 2006-03, Vol.21 (3), p.446-455

2006

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Role of ATF7‐TAF12 interactions in the vitamin D response hypersensitivity of osteoclast precursors in Paget's disease

Journal of bone and mineral research, 2013-06, Vol.28 (6), p.1489-1500

2013

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ADAM8 enhances osteoclast precursor fusion and osteoclast formation in vitro and in vivo

Journal of bone and mineral research, 2011-01, Vol.26 (1), p.169-181

2011

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The ZZ Domain of Sequestosome-1/p62 Plays An Important Role In Stromal Cell Support of Myeloma Cell Growth and Osteoclast Formation

Blood, 2010-11, Vol.116 (21), p.128-128

2010

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Blocking the ZZ Domain of Sequestosome 1/p62 Suppress the Enhancement of Myeloma Cell Growth and Osteoclast Formation by Marrow Stromal Cells

Blood, 2011-11, Vol.118 (21), p.888-888

2011

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Enhanced RANK ligand expression and responsivity of bone marrow cells in Paget's disease of bone

The Journal of clinical investigation, 2000-06, Vol.105 (12), p.1833-1838

2000

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Annexin II Stimulates RANKL Expression Through MAPK

Journal of bone and mineral research, 2005-07, Vol.20 (7), p.1161-1167

2005

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Osteoclasts Formed by Measles Virus-Infected Osteoclast Precursors from hCD46 Transgenic Mice Express Characteristics of Pagetic Osteoclasts

Endocrinology (Philadelphia), 2001-07, Vol.142 (7), p.2898-2905

2001

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Role of TAFII‐17, a VDR Binding Protein, in the Increased Osteoclast Formation in Paget's Disease

Journal of bone and mineral research, 2004-07, Vol.19 (7), p.1154-1164

2004

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Low Intensity Vibration Treatment Reduces Myeloma Cell-Induced Osteoclast Formation By Inhibiting IL-6 and GFI-1 Production By MM Cells

Blood, 2017-12, Vol.130, p.1775-1775

2017

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Pivotal Role of OCL-Derived IGF1 in Drug Resistance and Bone Destruction in MM

Blood, 2019-11, Vol.134 (Supplement_1), p.4336-4336

2019

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Eosinophil chemotactic factor-L (ECF-L) enhances osteoclast formation by increasing in osteoclast precursors expression of LFA-1 and ICAM-1

Bone (New York, N.Y.), 2007-02, Vol.40 (2), p.316-322

2007

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Experimental Models of Paget's Disease

Journal of bone and mineral research, 2006-12, Vol.21 (S2), p.P55-P57

2006

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Osteoclast-stimulating Factor Interacts with the Spinal Muscular Atrophy Gene Product to Stimulate Osteoclast Formation

The Journal of biological chemistry, 2001-11, Vol.276 (44), p.41035-41039

2001

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Aplidin (plitidepsin) is a novel anti-myeloma agent with potent anti-resorptive activity mediated by direct effects on osteoclasts

Oncotarget, 2019-04, Vol.10 (28), p.2709-2721

2019

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Targeting Notch Inhibitors to the Myeloma Bone Marrow Niche Decreases Tumor Growth and Bone Destruction without Gut Toxicity

Cancer research (Chicago, Ill.), 2021-10, Vol.81 (19), p.5102-5114

2021

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Notch3 signaling between myeloma cells and osteocytes in the tumor niche promotes tumor growth and bone destruction

Increased S1P expression in osteoclasts enhances bone formation in an animal model of Paget's disease

Contributions of the Measles Virus Nucleocapsid Gene and the SQSTM1/p62P392L Mutation to Paget's Disease

Activating transcription factor 4 regulates osteoclast differentiation in mice

Thalidomide derivative CC-4047 inhibits osteoclast formation by down-regulation of PU.1

A SQSTM1/p62 mutation linked to Paget’s disease increases the osteoclastogenic potential of the bone microenvironment

Osteoclast-derived IGF1 induces RANKL production in osteocytes and contributes to pagetic lesion formation

Growth factor independence 1 expression in myeloma cells enhances their growth, survival, and osteoclastogenesis

Increased signaling through p62 in the marrow microenvironment increases myeloma cell growth and osteoclast formation

Expression of Measles Virus Nucleocapsid Protein in Osteoclasts Induces Paget's Disease‐Like Bone Lesions in Mice

Role of ATF7‐TAF12 interactions in the vitamin D response hypersensitivity of osteoclast precursors in Paget's disease

ADAM8 enhances osteoclast precursor fusion and osteoclast formation in vitro and in vivo

The ZZ Domain of Sequestosome-1/p62 Plays An Important Role In Stromal Cell Support of Myeloma Cell Growth and Osteoclast Formation

Blocking the ZZ Domain of Sequestosome 1/p62 Suppress the Enhancement of Myeloma Cell Growth and Osteoclast Formation by Marrow Stromal Cells

Enhanced RANK ligand expression and responsivity of bone marrow cells in Paget's disease of bone

Annexin II Stimulates RANKL Expression Through MAPK

Osteoclasts Formed by Measles Virus-Infected Osteoclast Precursors from hCD46 Transgenic Mice Express Characteristics of Pagetic Osteoclasts

Role of TAFII‐17, a VDR Binding Protein, in the Increased Osteoclast Formation in Paget's Disease

Low Intensity Vibration Treatment Reduces Myeloma Cell-Induced Osteoclast Formation By Inhibiting IL-6 and GFI-1 Production By MM Cells

Pivotal Role of OCL-Derived IGF1 in Drug Resistance and Bone Destruction in MM

Eosinophil chemotactic factor-L (ECF-L) enhances osteoclast formation by increasing in osteoclast precursors expression of LFA-1 and ICAM-1

Experimental Models of Paget's Disease

Osteoclast-stimulating Factor Interacts with the Spinal Muscular Atrophy Gene Product to Stimulate Osteoclast Formation

Aplidin (plitidepsin) is a novel anti-myeloma agent with potent anti-resorptive activity mediated by direct effects on osteoclasts

Targeting Notch Inhibitors to the Myeloma Bone Marrow Niche Decreases Tumor Growth and Bone Destruction without Gut Toxicity

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