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Pharmacological Characterization of RS-1259, an Orally Active Dual Inhibitor of Acetylcholinesterase and Serotonin Transporter, in Rodents: Possible Treatment of Alzheimer’s Disease

Journal of Pharmacological Sciences, 2003, Vol.93(1), pp.95-105

2003

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In vivo conversion of racemized β-amyloid ([D-Ser26]Aβ1-40) to truncated and toxic fragments ([D-Ser26]Aβ25-35/40) and fragment presence in the brains of Alzheimer's patients

Journal of neuroscience research, 2002-11, Vol.70 (3), p.474-483

2002

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Obesity-Induced Diabetes in Mouse Strains Treated With Gold Thioglucose: A Novel Animal Model for Studying β-Cell Dysfunction

Obesity (Silver Spring, Md.), 2011-03, Vol.19 (3), p.514-521

2011

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A novel model of type 2 diabetes mellitus based on obesity induced by high-fat diet in BDF1 mice

Metabolism, clinical and experimental, 2009-03, Vol.58 (3), p.296-303

2009

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Obesity-Induced Diabetes in Mouse Strains Treated With Gold Thioglucose: A Novel Animal Model for Studying [beta]-Cell Dysfunction

Obesity (Silver Spring, Md.), 2011-03, Vol.19 (3), p.514

2011

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Monoamine oxidase inhibitors prevent striatal neuronal necrosis induced by transient forebrain ischemia

Neuroscience letters, 1991-05, Vol.126 (2), p.175-178

1991

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beta-amyloid racemized at the Ser(26) residue in the brains of patients with Alzheimer disease: Implications in the pathogenesis of Alzheimer disease

Journal of neuropathology and experimental neurology, 2003-03, Vol.62 (3), p.248

2003

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Pharmacological Characterization of RS-1259, an Orally Active Dual Inhibitor of Acetylcholinesterase and Serotonin Transporter, in Rodents: Possible Treatment of Alzheimer’s Disease

Journal of pharmacological sciences, 2003, Vol.93 (4), p.95-105

2003

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β-Amyloid Racemized at the Ser 26 Residue in the Brains of Patients with Alzheimer Disease: Implications in the Pathogenesis of Alzheimer Disease

Journal of neuropathology and experimental neurology, 2003-03, Vol.62 (3), p.248-259

2003

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β-Amyloid Racemized at the Ser26 Residue in the Brains of Patients with Alzheimer Disease: Implications in the Pathogenesis of Alzheimer Disease

Journal of neuropathology and experimental neurology, 2003-03, Vol.62 (3), p.248-259

2003

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Output, Tissue Levels, and Synthesis of Acetylcholine During and After Transient Forebrain Ischemia in the Rat

Journal of neurochemistry, 1991-04, Vol.56 (4), p.1169-1173

1991

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Participation of Type A Monoamine Oxidase in the Activated Deamination of Brain Monoamines Shortly after Reperfusion in Rats

The Japanese Journal of Pharmacology, 1990, Vol.54(4), pp.407-413

1990

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Deamination of Norepinephrine, Dopamine, and Serotonin by Type A Monoamine Oxidase in Discrete Regions of the Rat Brain and Inhibition by RS-8359

The Japanese Journal of Pharmacology, 1991, Vol.55(1), pp.121-128

1991

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Deamination of Norepinephrine, Dopamine, and Serotonin by Type A Monoamine Oxidase in Discrete Regions of the Rat Brain and Inhibition by RS-8359

Japanese Journal of Pharmacology, 1991, Vol.55 (1), p.121-128

1991

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In vitro and in vivo activities of RS-8359, an MAO-A inhibitor, and its enantiomers

Japanese Journal of Pharmacology, 1998, Vol.76 (suppl.1), p.119-119

1998

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Human c-Jun N-terminal kinase expression and activation in the nervous system

Brain research. Molecular brain research., 1999-04, Vol.67 (1), p.10-17

1999

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Changes in brain acetylcholine contents and release in vivo during and after transient ischemia in rats

Japanese Journal of Pharmacology, 1990, Vol.52 (suppl-1.1), p.294-294

1990

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Assessment of pharmacological effects and concentrations of RS-8359 on same rats by microdialysis

Japanese Journal of Pharmacology, 1994, Vol.64 (suppl.1), p.224-224

1994

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Modulation by Bovine Serum Albumin of Stimulated Formation of Cyclic AMP in Guinea Pig Hippocampus

The Japanese Journal of Pharmacology, 1984, Vol.36(2), pp.272-274

1984

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Analysis of the processing of Alzheimer amyloid precursor protein in culture cells by cDNA transfection

Japanese Journal of Pharmacology, 1993, Vol.61 (suppl.1), p.69-69

1993

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The toxic effect of Alzheimer amyloid protein precursor overexpressed in the neuroblastoma cell line NB-1 on neurite outgrowth

Gerontology (Basel), 1994-01, Vol.40 Suppl 2 (2), p.57-64

1994

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Up-regulation of JNK-3, human SAPKβ, after schemia in cultured neuroblastoma cells

Japanese Journal of Pharmacology, 1998, Vol.76 (suppl.2), p.271-271

1998

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Up-regulation of JNK-3, human SAPKβ, after ischemia in cultured neuroblastoma cells

Japanese journal of pharmacology, 1998, Vol.76, p.271-271

1998

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Inhibition of ischemia-induced striatal neuronal death by monoamine oxidase (MAO) inhibitors in the rat

Japanese journal of pharmacology, 1991, Vol.55, p.108

1991

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O-130 - Inhibition of ischemia-induced striatal neuronal death by monoamine oxidase (MAO) inhibitors in the rat

Japanese journal of pharmacology, 1991, Vol.55, p.108-108

1991

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Pharmacological Characterization of RS-1259, an Orally Active Dual Inhibitor of Acetylcholinesterase and Serotonin Transporter, in Rodents: Possible Treatment of Alzheimer’s Disease

In vivo conversion of racemized β-amyloid ([D-Ser26]Aβ1-40) to truncated and toxic fragments ([D-Ser26]Aβ25-35/40) and fragment presence in the brains of Alzheimer's patients

Obesity-Induced Diabetes in Mouse Strains Treated With Gold Thioglucose: A Novel Animal Model for Studying β-Cell Dysfunction

A novel model of type 2 diabetes mellitus based on obesity induced by high-fat diet in BDF1 mice

Obesity-Induced Diabetes in Mouse Strains Treated With Gold Thioglucose: A Novel Animal Model for Studying [beta]-Cell Dysfunction

Monoamine oxidase inhibitors prevent striatal neuronal necrosis induced by transient forebrain ischemia

beta-amyloid racemized at the Ser(26) residue in the brains of patients with Alzheimer disease: Implications in the pathogenesis of Alzheimer disease

Pharmacological Characterization of RS-1259, an Orally Active Dual Inhibitor of Acetylcholinesterase and Serotonin Transporter, in Rodents: Possible Treatment of Alzheimer’s Disease

β-Amyloid Racemized at the Ser 26 Residue in the Brains of Patients with Alzheimer Disease: Implications in the Pathogenesis of Alzheimer Disease

β-Amyloid Racemized at the Ser26 Residue in the Brains of Patients with Alzheimer Disease: Implications in the Pathogenesis of Alzheimer Disease

Output, Tissue Levels, and Synthesis of Acetylcholine During and After Transient Forebrain Ischemia in the Rat

Participation of Type A Monoamine Oxidase in the Activated Deamination of Brain Monoamines Shortly after Reperfusion in Rats

Deamination of Norepinephrine, Dopamine, and Serotonin by Type A Monoamine Oxidase in Discrete Regions of the Rat Brain and Inhibition by RS-8359

Deamination of Norepinephrine, Dopamine, and Serotonin by Type A Monoamine Oxidase in Discrete Regions of the Rat Brain and Inhibition by RS-8359

In vitro and in vivo activities of RS-8359, an MAO-A inhibitor, and its enantiomers

Human c-Jun N-terminal kinase expression and activation in the nervous system

Changes in brain acetylcholine contents and release in vivo during and after transient ischemia in rats

Assessment of pharmacological effects and concentrations of RS-8359 on same rats by microdialysis

Modulation by Bovine Serum Albumin of Stimulated Formation of Cyclic AMP in Guinea Pig Hippocampus

Analysis of the processing of Alzheimer amyloid precursor protein in culture cells by cDNA transfection

The toxic effect of Alzheimer amyloid protein precursor overexpressed in the neuroblastoma cell line NB-1 on neurite outgrowth

Up-regulation of JNK-3, human SAPKβ, after schemia in cultured neuroblastoma cells

Up-regulation of JNK-3, human SAPKβ, after ischemia in cultured neuroblastoma cells

Inhibition of ischemia-induced striatal neuronal death by monoamine oxidase (MAO) inhibitors in the rat

O-130 - Inhibition of ischemia-induced striatal neuronal death by monoamine oxidase (MAO) inhibitors in the rat

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