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Brain ischemia and reperfusion: molecular mechanisms of neuronal injury

Journal of the Neurological Sciences, 2000-10, Vol.179 (1), p.1-33

2000

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Molecular Pathways of Protein Synthesis Inhibition During Brain Reperfusion: Implications for Neuronal Survival or Death

Journal of Cerebral Blood Flow & Metabolism, 2002-02, Vol.22 (2), p.127-141

2002

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Insulin blocks cytochrome c release in the reperfused brain through PI3-K signaling and by promoting Bax/Bcl-XL binding

Journal of neurochemistry, 2008-08, Vol.106 (3), p.1248-1258

2008

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Dysfunction of the Unfolded Protein Response During Global Brain Ischemia and Reperfusion

Journal of cerebral blood flow and metabolism, 2003-04, Vol.23 (4), p.462-471

2003

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Insulin activates the PI3K-Akt survival pathway in vulnerable neurons following global brain ischemia

Neurological research (New York), 2009-11, Vol.31 (9), p.947-958

2009

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Suppression of Protein Synthesis in Brain during Hibernation Involves Inhibition of Protein Initiation and Elongation

Proceedings of the National Academy of Sciences - PNAS, 1998-11, Vol.95 (24), p.14511-14516

1998

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PERK is responsible for the increased phosphorylation of eIF2α and the severe inhibition of protein synthesis after transient global brain ischemia

Journal of neurochemistry, 2005-09, Vol.94 (5), p.1235-1242

2005

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Dysfunction of the unfolded protein response during global brain ischemia and reperfusion

Journal of cerebral blood flow and metabolism, 2003-04, Vol.23 (4), p.462-471

2003

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Insulin blocks cytochrome c release in the reperfused brain through PI3-K signaling and by promoting Bax-Bcl-XLbinding

Journal of neurochemistry, 2008-08, Vol.106 (3), p.1248-1258

2008

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Gene expression analysis of the development of congenital hydrocephalus in the H-Tx rat

Brain research, 2006-02, Vol.1075 (1), p.36-47

2006

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Calpain mediates eukaryotic initiation factor 4G degradation during global brain ischemia

Journal of cerebral blood flow and metabolism, 1998-08, Vol.18 (8), p.876-881

1998

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Postischemic tissue injury by iron-mediated free radical lipid peroxidation

Annals of emergency medicine, 1985-08, Vol.14 (8), p.804-809

1985

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Status report: Development of emergency medicine research since the macy report

Annals of emergency medicine, 2003-07, Vol.42 (1), p.66-80

2003

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Brain iron delocalization and lipid peroxidation following cardiac arrest

Annals of emergency medicine, 1986-04, Vol.15 (4), p.384-389

1986

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Insulin blocks cytochrome c release in the reperfused brain through PI3‐K signaling and by promoting Bax/Bcl‐X L binding

Journal of neurochemistry, 2008-08, Vol.106 (3), p.1248-1258

2008

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Natural course of iron delocalization and lipid peroxidation during the first eight hours following a 15-minute cardiac arrest in dogs

Annals of emergency medicine, 1987-11, Vol.16 (11), p.1200-1205

1987

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Incidence of cocaine-Associated rhabdomyolysis

Annals of emergency medicine, 1991-02, Vol.20 (2), p.154-157

1991

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Persistent eIf2α(p) is colocalized with cytoplasmic cytochrome c in vulnerable hippocampal neurons after 4 hours of reperfusion following 10-minute complete brain ischemia

Acta neuropathologica, 2003-07, Vol.106 (1), p.8-16

2003

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Brain cell death following ischemia and reperfusion: A proposed biochemical sequence

Critical care medicine, 1988-07, Vol.16 (7), p.714-726

1988

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Brain ischemia and reperfusion activates the eukaryotic initiation factor 2α kinase, PERK

Journal of neurochemistry, 2001-06, Vol.77 (5), p.1418-1421

2001

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Brain injury and repair mechanisms: The potential for pharmacologic therapy in closed-head trauma

Annals of emergency medicine, 1993-06, Vol.22 (6), p.970-979

1993

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Nuclear-Envelope Nucleoside Triphosphatase Kinetics and mRNA Transport Following Brain Ischemia and Reperfusion

Annals of emergency medicine, 1995-06, Vol.25 (6), p.809-817

1995

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Insulin blocks cytochrome c release in the reperfused brain through P13-K signaling and by promoting Bax/Bcl-XL binding

Journal of neurochemistry, 2008, Vol.106 (3), p.1248-1258

2008

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To The Editor

The Journal of emergency medicine, 2002-04, Vol.22 (3), p.301

Krause, Gary S.

2002

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Re: Henderson, Academic productivity in emergency medicine, The Journal of Emergency Medicine, July 2001

The Journal of emergency medicine, 2002-04, Vol.22 (3), p.301; author reply 301-301; author reply 302

Krause, Gary S

2002

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Brain ischemia and reperfusion: molecular mechanisms of neuronal injury

Molecular Pathways of Protein Synthesis Inhibition During Brain Reperfusion: Implications for Neuronal Survival or Death

Insulin blocks cytochrome c release in the reperfused brain through PI3-K signaling and by promoting Bax/Bcl-XL binding

Dysfunction of the Unfolded Protein Response During Global Brain Ischemia and Reperfusion

Insulin activates the PI3K-Akt survival pathway in vulnerable neurons following global brain ischemia

Suppression of Protein Synthesis in Brain during Hibernation Involves Inhibition of Protein Initiation and Elongation

PERK is responsible for the increased phosphorylation of eIF2α and the severe inhibition of protein synthesis after transient global brain ischemia

Dysfunction of the unfolded protein response during global brain ischemia and reperfusion

Insulin blocks cytochrome c release in the reperfused brain through PI3-K signaling and by promoting Bax-Bcl-XLbinding

Gene expression analysis of the development of congenital hydrocephalus in the H-Tx rat

Calpain mediates eukaryotic initiation factor 4G degradation during global brain ischemia

Postischemic tissue injury by iron-mediated free radical lipid peroxidation

Status report: Development of emergency medicine research since the macy report

Brain iron delocalization and lipid peroxidation following cardiac arrest

Insulin blocks cytochrome c release in the reperfused brain through PI3‐K signaling and by promoting Bax/Bcl‐X L binding

Natural course of iron delocalization and lipid peroxidation during the first eight hours following a 15-minute cardiac arrest in dogs

Incidence of cocaine-Associated rhabdomyolysis

Persistent eIf2α(p) is colocalized with cytoplasmic cytochrome c in vulnerable hippocampal neurons after 4 hours of reperfusion following 10-minute complete brain ischemia

Brain cell death following ischemia and reperfusion: A proposed biochemical sequence

Brain ischemia and reperfusion activates the eukaryotic initiation factor 2α kinase, PERK

Brain injury and repair mechanisms: The potential for pharmacologic therapy in closed-head trauma

Nuclear-Envelope Nucleoside Triphosphatase Kinetics and mRNA Transport Following Brain Ischemia and Reperfusion

Insulin blocks cytochrome c release in the reperfused brain through P13-K signaling and by promoting Bax/Bcl-XL binding

To The Editor

Re: Henderson, Academic productivity in emergency medicine, The Journal of Emergency Medicine, July 2001

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