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Identifying potential germline variants from sequencing hematopoietic malignancies

Blood, 2020-11, Vol.136 (22), p.2498-2506

2020

Volltextzugriff (PDF)

Sequential loss of tumor surface antigens following chimeric antigen receptor T-cell therapies in diffuse large B-cell lymphoma

Haematologica (Roma), 2018-05, Vol.103 (5), p.e215

2018

Volltextzugriff (PDF)

BCR-ABL1 compound mutations in tyrosine kinase inhibitor–resistant CML: frequency and clonal relationships

Blood, 2013-01, Vol.121 (3), p.489-498

2013

Volltextzugriff (PDF)

shRNA library screening identifies nucleocytoplasmic transport as a mediator of BCR-ABL1 kinase-independent resistance

Blood, 2015-03, Vol.125 (11), p.1772-1781

2015

Volltextzugriff (PDF)

Modulation of Target Antigen Density Improves CAR T-cell Functionality and Persistence

Clinical cancer research, 2019-09, Vol.25 (17), p.5329-5341

2019

Volltextzugriff (PDF)

Identifying potential germline variants from sequencing hematopoietic malignancies

Hematology, 2020-12, Vol.2020 (1), p.219-227

2020

Volltextzugriff (PDF)

Sequential tumor molecular profiling identifies likely germline variants

Genetics in medicine, 2024-03, Vol.26 (3), p.101037, Article 101037

2024

Volltextzugriff (PDF)

Outcomes of Children and Adolescents with Advanced Hereditary Medullary Thyroid Carcinoma Treated with Vandetanib

Clinical cancer research, 2018-02, Vol.24 (4), p.753-765

2018

Volltextzugriff (PDF)

CARDIOPULMONARY PRESENTATION OF A PATIENT WITH XXXY SYNDROME

Journal of the American College of Cardiology, 2018-03, Vol.71 (11), p.A2444-A2444

2018

Volltextzugriff (PDF)

Heterozygous germ line CSF3R variants as risk alleles for development of hematologic malignancies

Blood advances, 2020-10, Vol.4 (20), p.5269-5284

2020

Volltextzugriff (PDF)

KIT Signaling Governs Differential Sensitivity of Mature and Primitive CML Progenitors to Tyrosine Kinase Inhibitors

Cancer research (Chicago, Ill.), 2013-09, Vol.73 (18), p.5775-5786

2013

Volltextzugriff (PDF)

Reversible Orthopalladation of Phosphinimine−Imine Dichloropalladium(II) Complexes

Organometallics, 2009-07, Vol.28 (13), p.3889-3895

2009

Volltextzugriff (PDF)

Electron rich bidentate phosphinimine-imine ligands: synthesis and reactivity of late transition metal complexes

Dalton transactions : an international journal of inorganic chemistry, 2010-01, Vol.39 (2), p.541-547

2010

Volltextzugriff (PDF)

New Germline Syndrome Discovery: Heterozygous CSF3R Mutations May Predispose to Myeloid and Lymphoid Malignancies

Blood, 2019-11, Vol.134 (Supplement_1), p.2543-2543

2019

Volltextzugriff (PDF)

β-Catenin is required for intrinsic but not extrinsic BCR-ABL1 kinase-independent resistance to tyrosine kinase inhibitors in chronic myeloid leukemia

Leukemia, 2015-12, Vol.29 (12), p.2328-2337

2015

Volltextzugriff (PDF)

Abstract 4882: Genomic mechanisms of disease progression in pediatric medullary thyroid cancer (MTC)

Cancer research (Chicago, Ill.), 2017-07, Vol.77 (13_Supplement), p.4882-4882

2017

Volltextzugriff (PDF)

Direct Contact With Bone Marrow Stromal Cells Protects CML Progenitors From Imatinib Through Cytoplasmic Stabilization Of β-Catenin

Blood, 2013-11, Vol.122 (21), p.3982-3982

2013

Volltextzugriff (PDF)

Design, Optimization, and Pre-Clinical Evaluation of Direct, Mechanism-Based STAT3 Inhibitors for Treating Myeloid Disorders

Blood, 2014-12, Vol.124 (21), p.4816-4816

2014

Volltextzugriff (PDF)

Intrinsic and Extrinsic Survival Signals Converge on STAT3 As a Critical Mediator of BCR-ABL-Independent Tyrosine Kinase Inhibitor Resistance

Blood, 2011-11, Vol.118 (21), p.3742-3742

2011

Volltextzugriff (PDF)

Next-Generation STAT3 Inhibitors As Targeted Therapeutics in Chronic Myeloid Leukemia

Blood, 2012-11, Vol.120 (21), p.2445-2445

2012

Volltextzugriff (PDF)

STAT3 Inhibition Synergizes with BCR-ABL1 Inhibition to Overcome Kinase-Independent TKI Resistance in Chronic Myeloid Leukemia (CML)

Blood, 2012-11, Vol.120 (21), p.31-31

2012

Volltextzugriff (PDF)

BP5-087, a Novel STAT3 Inhibitor, Combines With BCR-ABL1 Inhibition To Overcome Kinase-Independent Resistance In Chronic Myeloid Leukemia

Blood, 2013-11, Vol.122 (21), p.854-854

2013

Volltextzugriff (PDF)

An Unbiased shRNA Library Screen Identifies Nucleocytoplasmic Transport As a Potential Target For Treatment Of Chronic Myeloid Leukemia

Blood, 2013-11, Vol.122 (21), p.2707-2707

2013

Volltextzugriff (PDF)

Frequency and Clonality of BCR-ABL Compound Mutations in Chronic Myeloid Leukemia

Blood, 2011-11, Vol.118 (21), p.3744-3744

2011

Volltextzugriff (PDF)

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Identifying potential germline variants from sequencing hematopoietic malignancies

Sequential loss of tumor surface antigens following chimeric antigen receptor T-cell therapies in diffuse large B-cell lymphoma

BCR-ABL1 compound mutations in tyrosine kinase inhibitor–resistant CML: frequency and clonal relationships

shRNA library screening identifies nucleocytoplasmic transport as a mediator of BCR-ABL1 kinase-independent resistance

Modulation of Target Antigen Density Improves CAR T-cell Functionality and Persistence

Identifying potential germline variants from sequencing hematopoietic malignancies

Sequential tumor molecular profiling identifies likely germline variants

Combined STAT3 and BCR-ABL1 inhibition induces synthetic lethality in therapy-resistant chronic myeloid leukemia

Outcomes of Children and Adolescents with Advanced Hereditary Medullary Thyroid Carcinoma Treated with Vandetanib

CARDIOPULMONARY PRESENTATION OF A PATIENT WITH XXXY SYNDROME

Heterozygous germ line CSF3R variants as risk alleles for development of hematologic malignancies

KIT Signaling Governs Differential Sensitivity of Mature and Primitive CML Progenitors to Tyrosine Kinase Inhibitors

Reversible Orthopalladation of Phosphinimine−Imine Dichloropalladium(II) Complexes

Electron rich bidentate phosphinimine-imine ligands: synthesis and reactivity of late transition metal complexes

New Germline Syndrome Discovery: Heterozygous CSF3R Mutations May Predispose to Myeloid and Lymphoid Malignancies

β-Catenin is required for intrinsic but not extrinsic BCR-ABL1 kinase-independent resistance to tyrosine kinase inhibitors in chronic myeloid leukemia

Abstract 4882: Genomic mechanisms of disease progression in pediatric medullary thyroid cancer (MTC)

Direct Contact With Bone Marrow Stromal Cells Protects CML Progenitors From Imatinib Through Cytoplasmic Stabilization Of β-Catenin

Design, Optimization, and Pre-Clinical Evaluation of Direct, Mechanism-Based STAT3 Inhibitors for Treating Myeloid Disorders

Intrinsic and Extrinsic Survival Signals Converge on STAT3 As a Critical Mediator of BCR-ABL-Independent Tyrosine Kinase Inhibitor Resistance

Next-Generation STAT3 Inhibitors As Targeted Therapeutics in Chronic Myeloid Leukemia

STAT3 Inhibition Synergizes with BCR-ABL1 Inhibition to Overcome Kinase-Independent TKI Resistance in Chronic Myeloid Leukemia (CML)

BP5-087, a Novel STAT3 Inhibitor, Combines With BCR-ABL1 Inhibition To Overcome Kinase-Independent Resistance In Chronic Myeloid Leukemia

An Unbiased shRNA Library Screen Identifies Nucleocytoplasmic Transport As a Potential Target For Treatment Of Chronic Myeloid Leukemia

Frequency and Clonality of BCR-ABL Compound Mutations in Chronic Myeloid Leukemia

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